Tricarboxylic acid cycle-dependent synthesis of Staphylococcus aureus Type 5 and 8 capsular polysaccharides. - Wikidata - awgldk - TriplyDB

Tricarboxylic acid cycle-dependent synthesis of Staphylococcus aureus Type 5 and 8 capsular polysaccharides.

Tricarboxylic acid cycle-dependent synthesis of Staphylococcus aureus Type 5 and 8 capsular polysaccharides.

http://www.wikidata.org/entity/Q30434928

about

Crystal structure of the enzyme CapF of Staphylococcus aureus reveals a unique architecture composed of two functional domains Evolution of multidrug resistance during Staphylococcus aureus infection involves mutation of the essential two component regulator WalKR Exposure of clinical MRSA heterogeneous strains to β-lactams redirects metabolism to optimize energy production through the TCA cycle The DUF59 Containing Protein SufT Is Involved in the Maturation of Iron-Sulfur (FeS) Proteins during Conditions of High FeS Cofactor Demand in Staphylococcus aureus Intermediate-type vancomycin resistance (VISA) in genetically-distinct Staphylococcus aureus isolates is linked to specific, reversible metabolic alterations.Construction and verification of the transcriptional regulatory response network of Streptococcus mutans upon treatment with the biofilm inhibitor carolacton The catabolite control protein E (CcpE) affects virulence determinant production and pathogenesis of Staphylococcus aureus Staphylococcus aureus ClpC divergently regulates capsule via sae and codY in strain newman but activates capsule via codY in strain UAMS-1 and in strain Newman with repaired saeS Metabolic sensor governing bacterial virulence in Staphylococcus aureus RpiR homologues may link Staphylococcus aureus RNAIII synthesis and pentose phosphate pathway regulation Nfu facilitates the maturation of iron-sulfur proteins and participates in virulence in Staphylococcus aureus.Phenotypic Variation Is Almost Entirely Independent of the Host-Pathogen Relationship in Clinical Isolates of S. aureus The SAV1322 gene from Staphylococcus aureus: genomic and proteomic approaches to identification and characterization of gene function RbsR Activates Capsule but Represses the rbsUDK Operon in Staphylococcus aureus The Staphylococcus aureus SrrAB Regulatory System Modulates Hydrogen Peroxide Resistance Factors, Which Imparts Protection to Aconitase during Aerobic Growth TCA cycle inactivation in Staphylococcus aureus alters nitric oxide production in RAW 264.7 cells.RpiRc Is a Pleiotropic Effector of Virulence Determinant Synthesis and Attenuates Pathogenicity in Staphylococcus aureus.Comprehensive identification of mutations responsible for heterogeneous vancomycin-intermediate Staphylococcus aureus (hVISA)-to-VISA conversion in laboratory-generated VISA strains derived from hVISA clinical strain Mu3 Catabolite control protein E (CcpE) is a LysR-type transcriptional regulator of tricarboxylic acid cycle activity in Staphylococcus aureus.Staphylococcus aureus adaptation to the host and persistence: role of loss of capsular polysaccharide expression.The Suf Iron-Sulfur Cluster Biosynthetic System Is Essential in Staphylococcus aureus, and Decreased Suf Function Results in Global Metabolic Defects and Reduced Survival in Human Neutrophils.The Active Component of Aspirin, Salicylic Acid, Promotes Staphylococcus aureus Biofilm Formation in a PIA-dependent Manner.Effect of 4-methoxy 1-methyl 2-oxopyridine 3-carbamide on Staphylococcus aureus by inhibiting UDP-MurNAc-pentapeptide, peptidyl deformylase and uridine monophosphate kinase.Staphylococcus aureus SufT: an essential iron-sulphur cluster assembly factor in cells experiencing a high-demand for lipoic acid.Phosphorylation of Staphylococcus aureus Protein-Tyrosine Kinase Affects the Function of Glucokinase and Biofilm Formation.A spectrum of CodY activities drives metabolic reorganization and virulence gene expression in Staphylococcus aureus.Phenotypic heterogeneity and temporal expression of the capsular polysaccharide in Staphylococcus aureus.A metabolomics and proteomics study of the adaptation of Staphylococcus aureus to glucose starvation Modulation of poly-N-acetylglucosamine accumulation within mature Staphylococcus epidermidis biofilms grown in excess glucose

P2860

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P2860

Tricarboxylic acid cycle-dependent synthesis of Staphylococcus aureus Type 5 and 8 capsular polysaccharides.

http://www.wikidata.org/entity/Q30434928

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունվարին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Tricarboxylic acid cycle-depen ...... nd 8 capsular polysaccharides.

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Tricarboxylic acid cycle-depen ...... nd 8 capsular polysaccharides.

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type

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Tricarboxylic acid cycle-depen ...... nd 8 capsular polysaccharides.

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Tricarboxylic acid cycle-depen ...... nd 8 capsular polysaccharides.

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Tricarboxylic acid cycle-depen ...... nd 8 capsular polysaccharides.

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P1433

Journal of Bacteriology

P1476

Tricarboxylic acid cycle-depen ...... nd 8 capsular polysaccharides.

@en

P2093

Chia Y Lee

http://www.w3.org/2001/XMLSchema#string

Greg A Somerville

http://www.w3.org/2001/XMLSchema#string

Marat R Sadykov

http://www.w3.org/2001/XMLSchema#string

Shandra R Day

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Thanh T Luong

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Theodoric A Mattes

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Yefei Zhu

http://www.w3.org/2001/XMLSchema#string

P2860

Two glyceraldehyde-3-phosphate dehydrogenases with opposite physiological roles in a nonphotosynthetic bacterium

Staphylococcal biofilm exopolysaccharide protects against Caenorhabditis elegans immune defenses.

Staphylococcus aureus virulence factors identified by using a high-throughput Caenorhabditis elegans-killing model

Staphylococcus epidermidis polysaccharide intercellular adhesin production significantly increases during tricarboxylic acid cycle stress

The intercellular adhesion (ica) locus is present in Staphylococcus aureus and is required for biofilm formation.

Regulation of Staphylococcus aureus capsular polysaccharide expression by agr and sarA.

Immunochemical properties of the staphylococcal poly-N-acetylglucosamine surface polysaccharide.

Staphylococcus aureus aconitase inactivation unexpectedly inhibits post-exponential-phase growth and enhances stationary-phase survival.

In vitro serial passage of Staphylococcus aureus: changes in physiology, virulence factor production, and agr nucleotide sequence.

Staphylococcus and biofilms.

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1459-1462

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scholarly article

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10.1128/JB.01377-09

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5

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192

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Costi David Sifri

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2010-01-08T00:00:00Z

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20061474

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P921

polysaccharides

P932

2820852

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