Infusing lipid raises plasma free fatty acids and induces insulin resistance in muscle microvasculature - Wikidata - awgldk - TriplyDB

Infusing lipid raises plasma free fatty acids and induces insulin resistance in muscle microvasculature

Infusing lipid raises plasma free fatty acids and induces insulin resistance in muscle microvasculature

http://www.wikidata.org/entity/Q30434930

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Thyroid Hormones, Oxidative Stress, and Inflammation The vascular endothelium in diabetes and its potential as a therapeutic target.Long-term high-fat diet induces hippocampal microvascular insulin resistance and cognitive dysfunction.Liraglutide prevents microvascular insulin resistance and preserves muscle capillary density in high-fat diet-fed rats Exercise resistance across the prediabetes phenotypes: Impact on insulin sensitivity and substrate metabolism.Muscle microvasculature's structural and functional specializations facilitate muscle metabolism.Inflammation-induced microvascular insulin resistance is an early event in diet-induced obesity Globular adiponectin ameliorates metabolic insulin resistance via AMPK-mediated restoration of microvascular insulin responses.Vascular function, insulin action, and exercise: an intricate interplay Adiponectin and insulin cross talk: the microvascular connection.GLP-1 at physiological concentrations recruits skeletal and cardiac muscle microvasculature in healthy humans.Losartan increases muscle insulin delivery and rescues insulin's metabolic action during lipid infusion via microvascular recruitment.Early microvascular recruitment modulates subsequent insulin-mediated skeletal muscle glucose metabolism during lipid infusion.Candesartan acutely recruits skeletal and cardiac muscle microvasculature in healthy humans.GLP-1 increases microvascular recruitment but not glucose uptake in human and rat skeletal muscle.Glucagon-like peptide 1 recruits microvasculature and increases glucose use in muscle via a nitric oxide-dependent mechanism Muscle microvascular recruitment predicts insulin sensitivity in middle-aged patients with type 1 diabetes mellitus Angiotensin II receptors modulate muscle microvascular and metabolic responses to insulin in vivo Insulin regulates its own delivery to skeletal muscle by feed-forward actions on the vasculature.Salsalate attenuates free fatty acid-induced microvascular and metabolic insulin resistance in humans Regulation of muscle microcirculation in health and diabetes.Free fatty acids induce insulin resistance in both cardiac and skeletal muscle microvasculature in humans.Resveratrol recruits rat muscle microvasculature via a nitric oxide-dependent mechanism that is blocked by TNFα.Angiotensin II type 1 and type 2 receptors regulate basal skeletal muscle microvascular volume and glucose use.Hypertension management and microvascular insulin resistance in diabetes Ranolazine recruits muscle microvasculature and enhances insulin action in rats.Meal-related increases in microvascular vasomotion are impaired in obese individuals: a potential mechanism in the pathogenesis of obesity-related insulin resistance.Alpha-receptor blockade improves muscle perfusion and glucose uptake in heart failure.Effects of oral and intravenous fat load on blood pressure, endothelial function, sympathetic activity, and oxidative stress in obese healthy subjects.Progressive hyperglycemia across the glucose tolerance continuum in older obese adults is related to skeletal muscle capillarization and nitric oxide bioavailability.Postmaximal contraction blood volume responses are blunted in obese and type 2 diabetic subjects in a muscle-specific manner.Seven days of aerobic exercise training improves conduit artery blood flow following glucose ingestion in patients with type 2 diabetes.Type 2 diabetes mellitus and exercise impairment Vascular effects of intravenous intralipid and dextrose infusions in obese subjects A sustained increase in plasma NEFA upregulates the Toll-like receptor network in human muscle.Inpatient enteral and parenteral [corrected] nutrition for patients with diabetes.Current understanding of increased insulin sensitivity after exercise - emerging candidates.Microvascular dysfunction: a potential mechanism in the pathogenesis of obesity-associated insulin resistance and hypertension.Microvascular dysfunction: an emerging pathway in the pathogenesis of obesity-related insulin resistance.Microbiota-derived tryptophan indoles increase after gastric bypass surgery and reduce intestinal permeability in vitro and in vivo.

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P2860

Infusing lipid raises plasma free fatty acids and induces insulin resistance in muscle microvasculature

http://www.wikidata.org/entity/Q30434930

description

2009 nî lūn-bûn

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2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հունիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Infusing lipid raises plasma f ...... nce in muscle microvasculature

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Infusing lipid raises plasma f ...... nce in muscle microvasculature

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type

label

Infusing lipid raises plasma f ...... nce in muscle microvasculature

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Infusing lipid raises plasma f ...... nce in muscle microvasculature

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Infusing lipid raises plasma f ...... nce in muscle microvasculature

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Infusing lipid raises plasma f ...... nce in muscle microvasculature

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P1433

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P1433

The Journal of Clinical Endocrinology and Metabolism

P1476

Infusing lipid raises plasma f ...... nce in muscle microvasculature

@en

P2093

Dale E Fowler

http://www.w3.org/2001/XMLSchema#string

Eugene J Barrett

http://www.w3.org/2001/XMLSchema#string

Jia Liu

http://www.w3.org/2001/XMLSchema#string

Linda A Jahn

http://www.w3.org/2001/XMLSchema#string

Zhenqi Liu

http://www.w3.org/2001/XMLSchema#string

P2860

Roles for insulin receptor, PI3-kinase, and Akt in insulin-signaling pathways related to production of nitric oxide in human vascular endothelial cells

Quantification of myocardial blood flow with ultrasound-induced destruction of microbubbles administered as a constant venous infusion

Physiologic hyperinsulinemia enhances human skeletal muscle perfusion by capillary recruitment.

Inhibiting NOS blocks microvascular recruitment and blunts muscle glucose uptake in response to insulin.

The vasodilatory actions of insulin on resistance and terminal arterioles and their impact on muscle glucose uptake.

Microvascular recruitment is an early insulin effect that regulates skeletal muscle glucose uptake in vivo.

Insulin at physiological concentrations selectively activates insulin but not insulin-like growth factor I (IGF-I) or insulin/IGF-I hybrid receptors in endothelial cells.

Obesity blunts insulin-mediated microvascular recruitment in human forearm muscle.

p38 mitogen-activated protein kinase mediates palmitate-induced apoptosis but not inhibitor of nuclear factor-kappaB degradation in human coronary artery endothelial cells.

Tumor necrosis factor-alpha induces insulin resistance in endothelial cells via a p38 mitogen-activated protein kinase-dependent pathway.

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3543-3549

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scholarly article

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10.1210/JC.2009-0027

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9

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94

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2009-06-30T00:00:00Z

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19567533

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P932

2741712

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