Hypermethylation and loss of expression of glutathione peroxidase-3 in Barrett's tumorigenesis. - Wikidata - awgldk - TriplyDB

Hypermethylation and loss of expression of glutathione peroxidase-3 in Barrett's tumorigenesis.

Hypermethylation and loss of expression of glutathione peroxidase-3 in Barrett's tumorigenesis.

http://www.wikidata.org/entity/Q30438403

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Role of epigenetic alterations in the pathogenesis of Barrett's esophagus and esophageal adenocarcinoma One-Carbon Metabolism in Prostate Cancer: The Role of Androgen Signaling Hepatoprotective Effects of Chinese Medicinal Herbs: A Focus on Anti-Inflammatory and Anti-Oxidative Activities Early events during neoplastic progression in Barrett's esophagus A review of the past, present, and future directions of neoplasia The Roles of Glutathione Peroxidases during Embryo Development Neoplasia: Where We Have Been and Where We Are Going Early involvement of death-associated protein kinase promoter hypermethylation in the carcinogenesis of Barrett's esophageal adenocarcinoma and its association with clinical progression Alterations in Barrett's-related adenocarcinomas: a proteomic approach.Expression analysis of Barrett's esophagus-associated high-grade dysplasia in laser capture microdissected archival tissue.Methylation of the Tumor Suppressor Gene RUNX3 in Human Gastric Carcinoma.Esophageal adenocarcinoma: treatment modalities in the era of targeted therapy.Identification of a DNA methylome profile of esophageal squamous cell carcinoma and potential plasma epigenetic biomarkers for early diagnosis.Epigenetic and genetic silencing of CHFR in esophageal adenocarcinomas.Selenium, selenoenzymes, oxidative stress and risk of neoplastic progression from Barrett's esophagus: results from biomarkers and genetic variants.Silencing of glutathione peroxidase 3 through DNA hypermethylation is associated with lymph node metastasis in gastric carcinomas Loss of glutathione peroxidase 3 expression is correlated with epigenetic mechanisms in endometrial adenocarcinoma.Epigenetic silencing of somatostatin in gastric cancer.Clinical significance and therapeutic value of glutathione peroxidase 3 (GPx3) in hepatocellular carcinoma Selenium. Role of the essential metalloid in health Decreased levels of serum glutathione peroxidase 3 are associated with papillary serous ovarian cancer and disease progression.Down-regulation of GPX3 is associated with favorable/intermediate karyotypes in de novo acute myeloid leukemia Frequent epigenetic suppression of tumor suppressor gene glutathione peroxidase 3 by promoter hypermethylation and its clinical implication in clear cell renal cell carcinoma GPX3 hypermethylation serves as an independent prognostic biomarker in non-M3 acute myeloid leukemia.GPX3 promoter is methylated in chronic myeloid leukemia.Diagnostic value of serum glutathione peroxidase 3 levels in patients with lung cancer.Dietary supplement use and risk of neoplastic progression in esophageal adenocarcinoma: a prospective study Food-chain selenium and human health: emphasis on intake.Esophageal adenocarcinoma arising in Barrett esophagus Epigenetic and Glucocorticoid Receptor-Mediated Regulation of Glutathione Peroxidase 3 in Lung Cancer Cells Gene expression profiling predicts a three-gene expression signature of endometrial adenocarcinoma in a rat model Glutathione peroxidase 7 has potential tumour suppressor functions that are silenced by location-specific methylation in oesophageal adenocarcinoma.Barrett's esophagus: can biomarkers predict progression to malignancy?Molecular mechanisms by which selenoproteins affect cancer risk and progression.GPX3 methylation in bone marrow predicts adverse prognosis and leukemia transformation in myelodysplastic syndrome Promoter hypermethylation and suppression of glutathione peroxidase 3 are associated with inflammatory breast carcinogenesis.Epigenetic-dependent regulation of drug transport and metabolism: an update.EURRECA-Estimating selenium requirements for deriving dietary reference values.Cellular origins and molecular mechanisms of Barrett's esophagus and esophageal adenocarcinoma.Overview of major molecular alterations during progression from Barrett's esophagus to esophageal adenocarcinoma.

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P2860

Hypermethylation and loss of expression of glutathione peroxidase-3 in Barrett's tumorigenesis.

http://www.wikidata.org/entity/Q30438403

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2005 nî lūn-bûn

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2005 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2005年論文

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Hypermethylation and loss of e ...... -3 in Barrett's tumorigenesis.

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Hypermethylation and loss of e ...... -3 in Barrett's tumorigenesis.

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Hypermethylation and loss of e ...... -3 in Barrett's tumorigenesis.

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Hypermethylation and loss of e ...... -3 in Barrett's tumorigenesis.

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Hypermethylation and loss of e ...... -3 in Barrett's tumorigenesis.

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Doerthe Kuester

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Michael Vieth

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Ok-Jae Lee

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Patricia A McChesney

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Wa'el El-Rifai

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P2860

Characterization and partial amino acid sequence of human plasma glutathione peroxidase

Free radicals in the physiological control of cell function

Purification and characterization of human plasma glutathione peroxidase: a selenoglycoprotein distinct from the known cellular enzyme

Oxidative DNA damage: mechanisms, mutation, and disease

The biology and pathology of oxygen radicals

Tissue-specific functions of individual glutathione peroxidases

Cancer epigenetics comes of age

Gastric cancers overexpress S100A calcium-binding proteins.

Incidence of adenocarcinoma and mortality in patients with Barrett's oesophagus diagnosed between 1976 and 1986: implications for endoscopic surveillance.

Oxidative stress: oxidants and antioxidants.

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