Francisella tularensis subsp. tularensis Schu S4 disulfide bond formation protein B, but not an RND-type efflux pump, is required for virulence - Wikidata - awgldk - TriplyDB

Francisella tularensis subsp. tularensis Schu S4 disulfide bond formation protein B, but not an RND-type efflux pump, is required for virulence

Francisella tularensis subsp. tularensis Schu S4 disulfide bond formation protein B, but not an RND-type efflux pump, is required for virulence

http://www.wikidata.org/entity/Q30440960

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Comparative review of Francisella tularensis and Francisella novicida Characterization of Francisella tularensis Schu S4 defined mutants as live-attenuated vaccine candidates Impact of Francisella tularensis pilin homologs on pilus formation and virulence Azithromycin effectiveness against intracellular infections of Francisella.Rationally designed tularemia vaccines.Identification of genes contributing to the virulence of Francisella tularensis SCHU S4 in a mouse intradermal infection model A Francisella tularensis Schu S4 purine auxotroph is highly attenuated in mice but offers limited protection against homologous intranasal challenge.Francisella tularensis DeltapyrF mutants show that replication in nonmacrophages is sufficient for pathogenesis in vivo.EmrA1 membrane fusion protein of Francisella tularensis LVS is required for resistance to oxidative stress, intramacrophage survival and virulence in mice.Directed screen of Francisella novicida virulence determinants using Drosophila melanogaster.Discovery of a novel and potent class of F. tularensis enoyl-reductase (FabI) inhibitors by molecular shape and electrostatic matching Low dose vaccination with attenuated Francisella tularensis strain SchuS4 mutants protects against tularemia independent of the route of vaccination.Identification of disulfide bond isomerase substrates reveals bacterial virulence factors.Genetic identification of unique immunological responses in mice infected with virulent and attenuated Francisella tularensis Sensitivity of Francisella tularensis to ultrapure water and deoxycholate: implications for bacterial intracellular growth assay in macrophages.Targeting virulence not viability in the search for future antibacterials.The francisella intracellular life cycle: toward molecular mechanisms of intracellular survival and proliferation.Vaccines against tularemia.A Francisella tularensis locus required for spermine responsiveness is necessary for virulence.Identifying Francisella tularensis genes required for growth in host cells.A Novel Glycolipid Biosurfactant Confers Grazing Resistance upon Pantoea ananatis BRT175 against the Social Amoeba Dictyostelium discoideum.Contribution of citrulline ureidase to Francisella tularensis strain Schu S4 pathogenesis Characterization of rationally attenuated Francisella tularensis vaccine strains that harbor deletions in the guaA and guaB genes.Vaccination of Fischer 344 rats against pulmonary infections by Francisella tularensis type A strains.Working toward the future: insights into Francisella tularensis pathogenesis and vaccine development.Proteins involved in Francisella tularensis survival and replication inside macrophages.Morphological analysis of Francisella novicida epithelial cell infections in the absence of functional FipA.Outer membrane vesicle-associated lipase FtlA enhances cellular invasion and virulence in Francisella tularensis LVS.Benzoxazoles, phthalazinones, and arylurea-based compounds with IMPDH-independent antibacterial activity against Francisella tularensis.Gluconeogenesis, an essential metabolic pathway for pathogenic Francisella.Characterization of a Unique Outer Membrane Protein Required for Oxidative Stress Resistance and Virulence of Francisella tularensis.Deletion of the Major Facilitator Superfamily Transporter fptB Alters Host Cell Interactions and Attenuates Virulence of Type A Francisella tularensis.Live Attenuated Tularemia Vaccines for Protection Against Respiratory Challenge With Virulent F. tularensis subsp. tularensis.

P2860

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P2860

Francisella tularensis subsp. tularensis Schu S4 disulfide bond formation protein B, but not an RND-type efflux pump, is required for virulence

http://www.wikidata.org/entity/Q30440960

description

2008 nî lūn-bûn

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2008 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2008 թվականի մայիսին հրատարակված գիտական հոդված

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2008年の論文

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Francisella tularensis subsp. ...... ump, is required for virulence

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Francisella tularensis subsp. ...... ump, is required for virulence

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Francisella tularensis subsp. ...... ump, is required for virulence

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Aiping Qin

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Barbara J Mann

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David W Scott

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P2860

The complete genome sequence of Francisella tularensis, the causative agent of tularemia

Construction of Escherichia coli K-12 in-frame, single-gene knockout mutants: the Keio collection

CLUSTAL W: improving the sensitivity of progressive multiple sequence alignment through sequence weighting, position-specific gap penalties and weight matrix choice

Twin RNA polymerase-associated proteins control virulence gene expression in Francisella tularensis

Identification of transposon insertion mutants of Francisella tularensis tularensis strain Schu S4 deficient in intracellular replication in the hepatic cell line HepG2.

The Francisella pathogenicity island protein IglA localizes to the bacterial cytoplasm and is needed for intracellular growth

DsbA: a protein-folding catalyst contributing to bacterial virulence.

The Burkholderia pseudomallei BpeAB-OprB efflux pump: expression and impact on quorum sensing and virulence.

Oxidative protein folding in bacteria.

Virulent and avirulent strains of Francisella tularensis prevent acidification and maturation of their phagosomes and escape into the cytoplasm in human macrophages.

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3086-3092

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10.1128/IAI.00363-08

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7

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76

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2008-05-05T00:00:00Z

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