Neurotensin stimulates mitogenesis of prostate cancer cells through a novel c-Src/Stat5b pathway.
about
Transactivation of Epidermal Growth Factor Receptor by G Protein-Coupled Receptors: Recent Progress, Challenges and Future ResearchCell-surface receptors transactivation mediated by g protein-coupled receptorsNeuroendocrine differentiation in prostate cancer: a mechanism of radioresistance and treatment failureSRC: marker or actor in prostate cancer aggressivenessβ-Adrenergic Receptor Signaling in Prostate CancerAFAP-110 is overexpressed in prostate cancer and contributes to tumorigenic growth by regulating focal contactsNeuroendocrine-derived peptides promote prostate cancer cell survival through activation of IGF-1R signaling.The neuroendocrine-derived peptide parathyroid hormone-related protein promotes prostate cancer cell growth by stabilizing the androgen receptor.Signal transducer and activator of transcription 5b, c-Src, and epidermal growth factor receptor signaling play integral roles in estrogen-stimulated proliferation of estrogen receptor-positive breast cancer cells.Activation of EGFR, HER2 and HER3 by neurotensin/neurotensin receptor 1 renders breast tumors aggressive yet highly responsive to lapatinib and metformin in mice.Radiosensitization in prostate cancer: mechanisms and targetsDysfunctional activation of neurotensin/IL-8 pathway in hepatocellular carcinoma is associated with increased inflammatory response in microenvironment, more epithelial mesenchymal transition in cancer and worse prognosis in patientsInsulin-like growth factor-1 receptor transactivation modulates the inflammatory and proliferative responses of neurotensin in human colonic epithelial cells.Neurotensin promotes the progression of malignant glioma through NTSR1 and impacts the prognosis of glioma patients.The significance of NTR1 expression and its correlation with β-catenin and EGFR in gastric cancer.Muscarinic type 3 receptor induces cytoprotective signaling in salivary gland cells through epidermal growth factor receptor transactivation.Neurotensin and its high affinity receptor 1 as a potential pharmacological target in cancer therapyGastrointestinal hormones stimulate growth of Foregut Neuroendocrine Tumors by transactivating the EGF receptor.Src regulates cell cycle protein expression and renal epithelial cell proliferation via PI3K/Akt signaling-dependent and -independent mechanisms.STAT5A/B gene locus undergoes amplification during human prostate cancer progression.SR48692 inhibits non-small cell lung cancer proliferation in an EGF receptor-dependent mannerCellular functions regulated by phosphorylation of EGFR on Tyr845.Agonist-induced activation of human FFA1 receptor signals to extracellular signal-regulated kinase 1 and 2 through Gq- and Gi-coupled signaling cascades.The potential of neurotensin secreted from neuroendocrine tumor cells to promote gelsolin-mediated invasiveness of prostate adenocarcinoma cells.Neuronal signaling repertoire in the mammalian sperm functionality.Antitumor mechanisms of targeting the PDK1 pathway in head and neck cancer.Heterotrimeric G proteins directly regulate MMP14/membrane type-1 matrix metalloprotease: a novel mechanism for GPCR-EGFR transactivation.Agonist-induced activation of histamine H3 receptor signals to extracellular signal-regulated kinases 1 and 2 through PKC-, PLD-, and EGFR-dependent mechanisms.The role of SRC family kinases in prostate cancer.Autophagy Blockade Sensitizes Prostate Cancer Cells towards Src Family Kinase Inhibitors.c-Src tyrosine kinase co-associates with and phosphorylates signal transducer and activator of transcription 5b which mediates the proliferation of normal human B lymphocytes.Inhibition of Src kinase blocks high glucose-induced EGFR transactivation and collagen synthesis in mesangial cells and prevents diabetic nephropathy in mice.Targeting Lyn tyrosine kinase through protein fusions encompassing motifs of Cbp (Csk-binding protein) and the SOCS box of SOCS1.New extracellular factors in glioblastoma multiforme development: neurotensin, growth differentiation factor-15, sphingosine-1-phosphate and cytomegalovirus infection.Androgen receptor signaling regulates T-type Ca2+ channel expression and neuroendocrine differentiation in prostate cancer cells.17β-Estradiol and ICI182,780 Differentially Regulate STAT5 Isoforms in Female Mammary Epithelium, With Distinct Outcomes.
P2860
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P2860
Neurotensin stimulates mitogenesis of prostate cancer cells through a novel c-Src/Stat5b pathway.
description
2006 nî lūn-bûn
@nan
2006 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Neurotensin stimulates mitogen ...... a novel c-Src/Stat5b pathway.
@ast
Neurotensin stimulates mitogen ...... a novel c-Src/Stat5b pathway.
@en
type
label
Neurotensin stimulates mitogen ...... a novel c-Src/Stat5b pathway.
@ast
Neurotensin stimulates mitogen ...... a novel c-Src/Stat5b pathway.
@en
prefLabel
Neurotensin stimulates mitogen ...... a novel c-Src/Stat5b pathway.
@ast
Neurotensin stimulates mitogen ...... a novel c-Src/Stat5b pathway.
@en
P2093
P2860
P356
P1433
P1476
Neurotensin stimulates mitogen ...... a novel c-Src/Stat5b pathway.
@en
P2093
P2860
P2888
P304
P356
10.1038/SJ.ONC.1209814
P407
P577
2006-07-24T00:00:00Z
P5875
P6179
1025977731