Potential of ketamine and midazolam, individually or in combination, to induce apoptotic neurodegeneration in the infant mouse brain.
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Update on neonatal anesthetic neurotoxicity: insight into molecular mechanisms and relevance to humansFrom Drug-Induced Developmental Neuroapoptosis to Pediatric Anesthetic Neurotoxicity-Where Are We Now?Modeling anesthetic developmental neurotoxicity using human stem cellsReview: effects of anesthetics on brain circuit formationClinical pharmacology of midazolam in neonates and children: effect of disease-a reviewSedation in Critically Ill Children with Respiratory FailureBehavioral consequences of NMDA antagonist-induced neuroapoptosis in the infant mouse brain.Clonidine for sedation and analgesia for neonates receiving mechanical ventilation.Protective Effect of Minocycline Against Ketamine-Induced Injury in Neural Stem Cell: Involvement of PI3K/Akt and Gsk-3 Beta Pathway.Functional implications of an early exposure to general anesthesia: are we changing the behavior of our children?Behavioral disturbances in adult mice following neonatal virus infection or kynurenine treatment--role of brain kynurenic acid.Timing versus duration: determinants of anesthesia-induced developmental apoptosis in the young mammalian brainGeneral anesthesia causes long-lasting disturbances in the ultrastructural properties of developing synapses in young rats.Juvenile exposure to ketamine causes delayed emergence of EEG abnormalities during adulthood in miceAnesthesia-induced developmental neurodegeneration: the role of neuronal organelles.Isoflurane impairs immature astroglia development in vitro: the role of actin cytoskeletonDeveloping brain and general anesthesia - is there a cause for concern?Impact of anaesthetics and surgery on neurodevelopment: an update.Chronic ketamine impairs fear conditioning and produces long-lasting reductions in auditory evoked potentials.Atypical anti-schizophrenic drugs prevent changes in cortical N-methyl-D-aspartate receptors and behavior following sub-chronic phencyclidine administration in developing rat pups.Ketamine alters cortical integration of GABAergic interneurons and induces long-term sex-dependent impairments in transgenic Gad67-GFP miceDelayed emergence of behavioral and electrophysiological effects following juvenile ketamine exposure in miceUse of analgesic and sedative drugs in the NICU: integrating clinical trials and laboratory data.Acetyl L-carnitine protects motor neurons and Rohon-Beard sensory neurons against ketamine-induced neurotoxicity in zebrafish embryos.Ketamine-induced apoptosis in the mouse cerebral cortex follows similar characteristic of physiological apoptosis and can be regulated by neuronal activity.Validation of a preclinical spinal safety model: effects of intrathecal morphine in the neonatal ratEffects of intrathecal ketamine in the neonatal rat: evaluation of apoptosis and long-term functional outcomeModulation of GABAergic and glutamatergic transmission by ethanol in the developing neocortex: an in vitro test of the excessive inhibition hypothesis of fetal alcohol spectrum disorder.Early childhood general anaesthesia exposure and neurocognitive development.General anesthetics in pediatric anesthesia: influences on the developing brain.The effect of lidocaine on apoptotic neurodegeneration in the developing mouse brainKetamine anesthesia during the first week of life can cause long-lasting cognitive deficits in rhesus monkeys.Are anaesthetics toxic to the brain?Early childhood exposure to anesthesia and risk of developmental and behavioral disorders in a sibling birth cohortRepeated exposure to ketamine-xylazine during early development impairs motor learning-dependent dendritic spine plasticity in adulthood.Comparative evaluation of the effects of propofol and sevoflurane on cognitive function and memory in patients undergoing laparoscopic cholecystectomy: A randomised prospective study.Ketamine causes mitochondrial dysfunction in human induced pluripotent stem cell-derived neurons.Perils of paediatric anaesthesia and novel molecular approaches: An evidence-based review.Propofol neurotoxicity is mediated by p75 neurotrophin receptor activation.The clinically available NMDA receptor antagonist, memantine, exhibits relative safety in the developing rat brain.
P2860
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P2860
Potential of ketamine and midazolam, individually or in combination, to induce apoptotic neurodegeneration in the infant mouse brain.
description
2005 nî lūn-bûn
@nan
2005 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Potential of ketamine and mida ...... ion in the infant mouse brain.
@ast
Potential of ketamine and mida ...... ion in the infant mouse brain.
@en
type
label
Potential of ketamine and mida ...... ion in the infant mouse brain.
@ast
Potential of ketamine and mida ...... ion in the infant mouse brain.
@en
prefLabel
Potential of ketamine and mida ...... ion in the infant mouse brain.
@ast
Potential of ketamine and mida ...... ion in the infant mouse brain.
@en
P2093
P2860
P356
P1476
Potential of ketamine and mida ...... ion in the infant mouse brain.
@en
P2093
Chainllie Young
Haihui Wang
Joann Labruyere
John W Olney
Tatyana Tenkova
Yue-Qin Qin
P2860
P304
P356
10.1038/SJ.BJP.0706301
P407
P577
2005-09-01T00:00:00Z