Oxidized phospholipids induce phenotypic switching of vascular smooth muscle cells in vivo and in vitro. - Wikidata - awgldk - TriplyDB

Oxidized phospholipids induce phenotypic switching of vascular smooth muscle cells in vivo and in vitro.

Oxidized phospholipids induce phenotypic switching of vascular smooth muscle cells in vivo and in vitro.

http://www.wikidata.org/entity/Q30444292

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TGF-β/Smad3 stimulates stem cell/developmental gene expression and vascular smooth muscle cell de-differentiation Mammalian Krüppel-like factors in health and diseases Role of vascular smooth muscle cell in the inflammation of atherosclerosis Regulation of an inflammatory disease: Krüppel-like factors and atherosclerosis Implications of autophagy for vascular smooth muscle cell function and plasticity Txnip ablation reduces vascular smooth muscle cell inflammation and ameliorates atherosclerosis in apolipoprotein E knockout mice Epigenetic Control of Smooth Muscle Cell Identity and Lineage Memory MicroRNA-1 regulates smooth muscle cell differentiation by repressing Kruppel-like factor 4 Lipid peroxidation generates biologically active phospholipids including oxidatively N-modified phospholipids Generation and biological activities of oxidized phospholipids Inhaled diesel emissions alter atherosclerotic plaque composition in ApoE(-/-) mice Krüppel-like factor 4 inhibits proliferation by platelet-derived growth factor receptor beta-mediated, not by retinoic acid receptor alpha-mediated, phosphatidylinositol 3-kinase and ERK signaling in vascular smooth muscle cells Suppression of JAK2/STAT3 signaling reduces end-to-end arterial anastomosis induced cell proliferation in common carotid arteries of rats Kruppel-like factor 4, Elk-1, and histone deacetylases cooperatively suppress smooth muscle cell differentiation markers in response to oxidized phospholipids.NCK Associated Protein 1 Modulated by miRNA-214 Determines Vascular Smooth Muscle Cell Migration, Proliferation, and Neointima Hyperplasia.The effect of oxidized phospholipids on phenotypic polarization and function of macrophages.Activation of the pluripotency factor OCT4 in smooth muscle cells is atheroprotective.Vascular Smooth Muscle Cells in Atherosclerosis.KLF4-dependent phenotypic modulation of smooth muscle cells has a key role in atherosclerotic plaque pathogenesis.TNF-α induces phenotypic modulation in cerebral vascular smooth muscle cells: implications for cerebral aneurysm pathology.KLF4 regulates abdominal aortic aneurysm morphology and deletion attenuates aneurysm formation Cooperative binding of KLF4, pELK-1, and HDAC2 to a G/C repressor element in the SM22α promoter mediates transcriptional silencing during SMC phenotypic switching in vivo.Cigarette smoke modulates vascular smooth muscle phenotype: implications for carotid and cerebrovascular disease Smooth muscle cell phenotypic switching in atherosclerosis Cigarette smoke and inflammation: role in cerebral aneurysm formation and rupture.Smooth and cardiac muscle-selective knock-out of Kruppel-like factor 4 causes postnatal death and growth retardation.Cyclosporine up-regulates Krüppel-like factor-4 (KLF4) in vascular smooth muscle cells and drives phenotypic modulation in vivo.Complex regulation and function of the inflammatory smooth muscle cell phenotype in atherosclerosis Oxidized phospholipid species promote in vivo differential cx43 phosphorylation and vascular smooth muscle cell proliferation Sp1-dependent activation of KLF4 is required for PDGF-BB-induced phenotypic modulation of smooth muscle.The role of oxidized phospholipids in atherosclerosis.Oxidized phospholipids induce type VIII collagen expression and vascular smooth muscle cell migration.Connexins in vascular physiology and pathology.Role of smooth muscle cells in the initiation and early progression of atherosclerosis.Molecular mechanisms of collagen isotype-specific modulation of smooth muscle cell phenotype Conditional deletion of Krüppel-like factor 4 delays downregulation of smooth muscle cell differentiation markers but accelerates neointimal formation following vascular injury Sphingosine-1-phosphate receptor subtypes differentially regulate smooth muscle cell phenotype.A novel approach to minimize false discovery rate in genome-wide data analysis.PDGF-mediated autophagy regulates vascular smooth muscle cell phenotype and resistance to oxidative stress c-Kit modifies the inflammatory status of smooth muscle cells

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P2860

Oxidized phospholipids induce phenotypic switching of vascular smooth muscle cells in vivo and in vitro.

http://www.wikidata.org/entity/Q30444292

description

2007 nî lūn-bûn

@nan

2007 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2007年の論文

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2007年論文

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2007年論文

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2007年論文

@zh-hk

2007年論文

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2007年論文

@zh-tw

2007年论文

@wuu

name

Oxidized phospholipids induce ...... le cells in vivo and in vitro.

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Oxidized phospholipids induce ...... le cells in vivo and in vitro.

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type

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Oxidized phospholipids induce ...... le cells in vivo and in vitro.

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Oxidized phospholipids induce ...... le cells in vivo and in vitro.

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Oxidized phospholipids induce ...... le cells in vivo and in vitro.

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CIRCRESAHA.107.152736

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Circulation Research

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Oxidized phospholipids induce ...... le cells in vivo and in vitro.

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Gary K Owens

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James A Thomas

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Matthew R Alexander

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Nataliya A Pidkovka

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Olga A Cherepanova

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Rebecca A Deaton

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Tadashi Yoshida

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792-801

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scholarly article

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10.1161/CIRCRESAHA.107.152736

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8

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101

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Norbert Leitinger

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2007-08-17T00:00:00Z

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17704209

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