Synthetic microRNA-mediated downregulation of Nogo-A in transgenic rats reveals its role as regulator of synaptic plasticity and cognitive function
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Nogo limits neural plasticity and recovery from injuryThe sphingolipid receptor S1PR2 is a receptor for Nogo-a repressing synaptic plasticityCell type-specific Nogo-A gene ablation promotes axonal regeneration in the injured adult optic nerveInsights into the physiological role of CNS regeneration inhibitorsReduced expression of nogo-a leads to motivational deficits in rats.Effective heritable gene knockdown in zebrafish using synthetic microRNAs.Neutralization of Nogo-A enhances synaptic plasticity in the rodent motor cortex and improves motor learning in vivo.Perineuronal nets and schizophrenia: the importance of neuronal coatings.Acquisition of a High-precision Skilled Forelimb Reaching Task in Rats.The Neurite Outgrowth Inhibitory Nogo-A-Δ20 Region Is an Intrinsically Disordered Segment Harbouring Three Stretches with Helical Propensity.Nogo-A-deficient Transgenic Rats Show Deficits in Higher Cognitive Functions, Decreased Anxiety, and Altered Circadian Activity Patterns.Emerging genotype-phenotype relationships in patients with large NF1 deletions.The role of reticulons in neurodegenerative diseases.Function of Nogo-A/Nogo-A receptor in Alzheimer's disease.New Insights into the Roles of Nogo-A in CNS Biology and Diseases.Recombinant Nogo-66 via soluble expression with SUMO fusion in Escherichia coli inhibits neurite outgrowth in vitro.Nogo-66 Restricts Synaptic Strengthening via Lingo1 and the ROCK2-Cofilin Pathway to Control Actin Dynamics.Anti-Nogo-A Immunotherapy Does Not Alter Hippocampal Neurogenesis after Stroke in Adult Rats.N-Methyl-d-Aspartate Receptor - Nitric Oxide Synthase Pathway in the Cortex of Nogo-A-Deficient Rats in Relation to Brain Laterality and Schizophrenia.Spatiotemporal and Long Lasting Modulation of 11 Key Nogo Signaling Genes in Response to Strong Neuroexcitation.Upregulation of axon guidance molecules in the adult central nervous system of Nogo-A knockout mice restricts neuronal growth and regeneration.Nogo-A deletion increases the plasticity of the optokinetic response and changes retinal projection organization in the adult mouse visual system.Forebrain-specific, conditional silencing of Staufen2 alters synaptic plasticity, learning, and memory in rats.Urokinase-type Plasminogen Activator Induces Neurorepair in the Ischemic Brain
P2860
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P2860
Synthetic microRNA-mediated downregulation of Nogo-A in transgenic rats reveals its role as regulator of synaptic plasticity and cognitive function
description
2013 nî lūn-bûn
@nan
2013 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Synthetic microRNA-mediated do ...... sticity and cognitive function
@ast
Synthetic microRNA-mediated do ...... sticity and cognitive function
@en
type
label
Synthetic microRNA-mediated do ...... sticity and cognitive function
@ast
Synthetic microRNA-mediated do ...... sticity and cognitive function
@en
prefLabel
Synthetic microRNA-mediated do ...... sticity and cognitive function
@ast
Synthetic microRNA-mediated do ...... sticity and cognitive function
@en
P2093
P2860
P356
P1476
Synthetic microRNA-mediated do ...... sticity and cognitive function
@en
P2093
Björn Tews
Dusan Bartsch
Hansjörg Kasper
Kai Schönig
Martin E Schwab
Mengia-Seraina Rioult-Pedotti
Miriam Schneider
Oliver Weinmann
Stefan M Berger
Stefano Clementi
P2860
P304
P356
10.1073/PNAS.1217665110
P407
P577
2013-04-01T00:00:00Z