Analysis of the subgroup A avian sarcoma and leukosis virus receptor: the 40-residue, cysteine-rich, low-density lipoprotein receptor repeat motif of Tva is sufficient to mediate viral entry.
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Mutational analysis of the subgroup A avian sarcoma and leukosis virus putative fusion peptide domain.Solution structure of the viral receptor domain of Tva and its implications in viral entryThe avian retrovirus avian sarcoma/leukosis virus subtype A reaches the lipid mixing stage of fusion at neutral pH.Activation of a retroviral membrane fusion protein: soluble receptor-induced liposome binding of the ALSV envelope glycoprotein.Antibody binding in proximity to the receptor/glycoprotein complex leads to a basal level of virus neutralization.Stoichiometry of envelope glycoprotein trimers in the entry of human immunodeficiency virus type 1Heptad repeat 2-based peptides inhibit avian sarcoma and leukosis virus subgroup a infection and identify a fusion intermediateThe spacing between cysteines two and three of the LDL-A module of Tva is important for subgroup A avian sarcoma and leukosis virus entryIdentification of a new region of SARS-CoV S protein critical for viral entryProduction and characterization of a soluble, active form of Tva, the subgroup A avian sarcoma and leukosis virus receptorEfficient infection mediated by viral receptors incorporated into retroviral particles.Genetic mapping of the cloned subgroup A avian sarcoma and leukosis virus receptor gene to the TVA locus.Characterization of Ebola virus entry by using pseudotyped viruses: identification of receptor-deficient cell lines.Characterization of determinants for envelope binding and infection in tva, the subgroup A avian sarcoma and leukosis virus receptorSoluble receptor-induced retroviral infection of receptor-deficient cellsSoluble forms of the subgroup A avian leukosis virus [ALV(A)] receptor Tva significantly inhibit ALV(A) infection in vitro and in vivo.Identification of key residues in subgroup A avian leukosis virus envelope determining receptor binding affinity and infectivity of cells expressing chicken or quail Tva receptor.Role of calcium in protein folding and function of Tva, the receptor of subgroup A avian sarcoma and leukosis virusThe receptor for the subgroup C avian sarcoma and leukosis viruses, Tvc, is related to mammalian butyrophilins, members of the immunoglobulin superfamily.Synthesis and anti-HIV activity of 2'-deoxy-2'-fluoro-4'-C-ethynyl nucleoside analogs.Identification of two residues within the LDL-A module of Tva that dictate the altered receptor specificity of mutant subgroup A avian sarcoma and leukosis viruses.Murine coronavirus receptors are differentially expressed in the central nervous system and play virus strain-dependent roles in neuronal spreadResistance to infection by subgroups B, D, and E avian sarcoma and leukosis viruses is explained by a premature stop codon within a resistance allele of the tvb receptor gene.Two different molecular defects in the Tva receptor gene explain the resistance of two tvar lines of chickens to infection by subgroup A avian sarcoma and leukosis viruses.Kinetic analysis of binding interaction between the subgroup A Rous sarcoma virus glycoprotein SU and its cognate receptor Tva: calcium is not required for ligand binding.Synthesis of new 2'-deoxy-2'-fluoro-4'-azido nucleoside analogues as potent anti-HIV agents.The mature avian leukosis virus subgroup A envelope glycoprotein is metastable, and refolding induced by the synergistic effects of receptor binding and low pH is coupled to infectionRole of basic residues in the subgroup-determining region of the subgroup A avian sarcoma and leukosis virus envelope in receptor binding and infection.Conversion of a human low-density lipoprotein receptor ligandbinding repeat to a virus receptor: identification of residues important for ligand specificityThe Role of the Charged Residues of the GP2 Helical Regions in Ebola Entry()Cell-specific viral targeting mediated by a soluble retroviral receptor-ligand fusion proteinCharacterization of the LDL-A module mutants of Tva, the subgroup A Rous sarcoma virus receptor, and the implications in protein folding.Nef can enhance the infectivity of receptor-pseudotyped human immunodeficiency virus type 1 particles.Efficient subgroup C avian sarcoma and leukosis virus receptor activity requires the IgV domain of the Tvc receptor and proper display on the cell membraneProtein expression profiles of C3H 10T1/2 murine fibroblasts and of isogenic cells transformed by the H1047R mutant of phosphoinositide 3-kinase (PI3K).Residue Trp-48 of Tva is critical for viral entry but not for high-affinity binding to the SU glycoprotein of subgroup A avian leukosis and sarcoma viruses.Model of the TVA receptor determinants required for efficient infection by subgroup A avian sarcoma and leukosis viruses.Residues 28 to 39 of the Extracellular Loop 1 of Chicken Na+/H+ Exchanger Type I Mediate Cell Binding and Entry of Subgroup J Avian Leukosis Virus.
P2860
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P2860
Analysis of the subgroup A avian sarcoma and leukosis virus receptor: the 40-residue, cysteine-rich, low-density lipoprotein receptor repeat motif of Tva is sufficient to mediate viral entry.
description
1995 nî lūn-bûn
@nan
1995 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Analysis of the subgroup A avi ...... icient to mediate viral entry.
@ast
Analysis of the subgroup A avi ...... icient to mediate viral entry.
@en
type
label
Analysis of the subgroup A avi ...... icient to mediate viral entry.
@ast
Analysis of the subgroup A avi ...... icient to mediate viral entry.
@en
prefLabel
Analysis of the subgroup A avi ...... icient to mediate viral entry.
@ast
Analysis of the subgroup A avi ...... icient to mediate viral entry.
@en
P2860
P1433
P1476
Analysis of the subgroup A avi ...... ficient to mediate viral entry
@en
P2093
P2860
P304
P50
P577
1995-08-01T00:00:00Z