Stimulation of the noradrenergic system during memory formation impairs extinction learning but not the disruption of reconsolidation. - Wikidata - awgldk - TriplyDB

Stimulation of the noradrenergic system during memory formation impairs extinction learning but not the disruption of reconsolidation.

Stimulation of the noradrenergic system during memory formation impairs extinction learning but not the disruption of reconsolidation.

http://www.wikidata.org/entity/Q30455937

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Regulation of excitatory synapses and fearful memories by stress hormones New avenues for treating emotional memory disorders: towards a reconsolidation intervention for posttraumatic stress disorder Modulation of Fear Extinction by Stress, Stress Hormones and Estradiol: A Review.Behavioral and neurobiological mechanisms of extinction in Pavlovian and instrumental learning Targeting memory processes with drugs to prevent or cure PTSD Noradrenergic regulation of fear and drug-associated memory reconsolidation Mechanisms to medicines: elucidating neural and molecular substrates of fear extinction to identify novel treatments for anxiety disorders Revisiting propranolol and PTSD: Memory erasure or extinction enhancement?Modification of Fear Memory by Pharmacological and Behavioural Interventions during Reconsolidation.Retrieval cues that trigger reconsolidation of associative fear memory are not necessarily an exact replica of the original learning experience Can fear extinction be enhanced? A review of pharmacological and behavioral findings.Noradrenergic blockade of memory reconsolidation: a failure to reduce conditioned fear responding Fear inhibition in high trait anxiety High trait anxiety: a challenge for disrupting fear memory reconsolidation.Contextual control over expression of fear is affected by cortisol In Search for Boundary Conditions of Reconsolidation: A Failure of Fear Memory Interference.Evidence for alterations in central noradrenergic signaling in irritable bowel syndrome Modulating reconsolidation: a link to causal systems-level dynamics of human memories.Translational Approaches Targeting Reconsolidation.Effects of Prereactivation Propranolol on Cocaine Craving Elicited by Imagery Script/Cue Sets in Opioid-dependent Polydrug Users: A Randomized Study.Effects of Propranolol, a β-noradrenergic Antagonist, on Memory Consolidation and Reconsolidation in Mice.A double blind, placebo-controlled study of the effects of post-retrieval propranolol on reconsolidation of memory for craving and cue reactivity in cocaine dependent humans.A bout of voluntary running enhances context conditioned fear, its extinction, and its reconsolidation Noradrenergic modulation of cognition: therapeutic implications.Memory reconsolidation in aversive and appetitive settings.Effect of a Brief Memory Updating Intervention on Smoking Behavior: A Randomized Clinical Trial.Why Forget? On the Adaptive Value of Memory Loss.Augmentation of Evidence-Based Psychotherapy for PTSD With Cognitive Enhancers.The potential of epigenetics in stress-enhanced fear learning models of PTSD.First Steps in Using Multi-Voxel Pattern Analysis to Disentangle Neural Processes Underlying Generalization of Spider Fear.Fearing shades of grey: individual differences in fear responding towards generalisation stimuli.Disrupting reconsolidation of fear memory in humans by a noradrenergic β-blocker.Young and old Pavlovian fear memories can be modified with extinction training during reconsolidation in humans.Prediction error demarcates the transition from retrieval, to reconsolidation, to new learning.Memory Reconsolidation Interference as an Emerging Treatment for Emotional Disorders: Strengths, Limitations, Challenges, and Opportunities.PTSD-like memory generated through enhanced noradrenergic activity is mitigated by a dual step pharmacological intervention targeting its reconsolidation.No evidence for enhanced extinction memory consolidation through noradrenergic reuptake inhibition-delayed memory test and reinstatement in human fMRI.Prazosin during threat discrimination boosts memory of the safe stimulus.Memory creation and modification: Enhancing the treatment of psychological disorders.Multiple memory systems, multiple time points: how science can inform treatment to control the expression of unwanted emotional memories.

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P2860

Stimulation of the noradrenergic system during memory formation impairs extinction learning but not the disruption of reconsolidation.

http://www.wikidata.org/entity/Q30455937

description

2011 nî lūn-bûn

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2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Stimulation of the noradrenerg ...... disruption of reconsolidation.

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Stimulation of the noradrenerg ...... disruption of reconsolidation.

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Stimulation of the noradrenerg ...... disruption of reconsolidation.

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Stimulation of the noradrenerg ...... disruption of reconsolidation.

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Stimulation of the noradrenerg ...... disruption of reconsolidation.

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Neuropsychopharmacology

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Stimulation of the noradrenerg ...... disruption of reconsolidation.

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Marieke Soeter

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Merel Kindt

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P2860

Fear memories require protein synthesis in the amygdala for reconsolidation after retrieval

Phobias and preparedness: the selective, automatic, and encapsulated nature of fear.

Conditioned fear extinction and reinstatement in a human fear-potentiated startle paradigm

Increasing CREB in the auditory thalamus enhances memory and generalization of auditory conditioned fear

Cognitive neuroscience of emotional memory.

A requirement for memory retrieval during and after long-term extinction learning

Meta-analysis of risk factors for posttraumatic stress disorder in trauma-exposed adults.

Yohimbine increases impulsivity through activation of cAMP response element binding in the orbitofrontal cortex

Long-term memory is facilitated by cAMP response element-binding protein overexpression in the amygdala.

Effect of post-retrieval propranolol on psychophysiologic responding during subsequent script-driven traumatic imagery in post-traumatic stress disorder.

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10.1038/NPP.2011.307

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