Hippocampal unit activity after transient cerebral ischemia in rats.
about
Differential responses of progesterone receptor membrane component-1 (Pgrmc1) and the classical progesterone receptor (Pgr) to 17β-estradiol and progesterone in hippocampal subregions that support synaptic remodeling and neurogenesisHyperbaric hyperoxia and normobaric reoxygenation increase excitability and activate oxygen-induced potentiation in CA1 hippocampal neurons.Characterization of the anoxia-induced long-term synaptic potentiation in area CA1 of the rat hippocampus.The role of dendritic dysfunction in neurodegeneration.Changes in extracellular amino acids and spontaneous neuronal activity during ischemia and extended reflow in the CA1 of the rat hippocampus.Block by N6-L-phenylisopropyl-adenosine of the electrophysiological and morphological correlates of hippocampal ischaemic injury in the gerbil.The modulations of NCAM polysialylation state that follow transient global ischemia are brief on neurons but enduring on glia.Localization of 70 kDa stress protein mRNA induction in gerbil brain after ischemia.l-Lactate mediates neuroprotection against ischaemia by increasing TREK1 channel expression in rat hippocampal astrocytes in vitro.GABAA receptor function in the early period after transient forebrain ischaemia in the rat.Transient neurophysiological changes in CA3 neurons and dentate granule cells after severe forebrain ischemia in vivo.Hilar somatostatin neurons are more vulnerable to an ischemic insult than CA1 pyramidal neurons.Enhanced calcium uptake by CA1 pyramidal cell dendrites in the postischemic phase despite subnormal evoked field potentials: excitatory amino acid receptor dependency and relationship to neuronal damage.Postischemic alterations of complex spike cell discharges and evoked potentials in rat hippocampal CA1 region.Disturbance of membrane function preceding ischemic delayed neuronal death in the gerbil hippocampus.Transient ischemia induces an early decrease of synaptic transmission in CA1 neurons of rat hippocampus: electrophysiologic study in brain slices.Delayed neuronal death is induced without postischemic hyperexcitability: continuous multiple-unit recording from ischemic CA1 neurons.Enhancement of GABA neurotransmission after cerebral ischemia in the rat reduces loss of hippocampal CA1 pyramidal cells.Differential changes of potassium currents in CA1 pyramidal neurons after transient forebrain ischemia.
P2860
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P2860
Hippocampal unit activity after transient cerebral ischemia in rats.
description
1989 nî lūn-bûn
@nan
1989 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Hippocampal unit activity after transient cerebral ischemia in rats.
@ast
Hippocampal unit activity after transient cerebral ischemia in rats.
@en
type
label
Hippocampal unit activity after transient cerebral ischemia in rats.
@ast
Hippocampal unit activity after transient cerebral ischemia in rats.
@en
prefLabel
Hippocampal unit activity after transient cerebral ischemia in rats.
@ast
Hippocampal unit activity after transient cerebral ischemia in rats.
@en
P2093
P356
P1433
P1476
Hippocampal unit activity after transient cerebral ischemia in rats.
@en
P2093
P304
P356
10.1161/01.STR.20.8.1051
P407
P577
1989-08-01T00:00:00Z