Deletion of densin-180 results in abnormal behaviors associated with mental illness and reduces mGluR5 and DISC1 in the postsynaptic density fraction.
about
CaMKII binding to GluN2B is critical during memory consolidationPhosphorylation of synaptic GTPase-activating protein (synGAP) by Ca2+/calmodulin-dependent protein kinase II (CaMKII) and cyclin-dependent kinase 5 (CDK5) alters the ratio of its GAP activity toward Ras and Rap GTPasesFmr1 deficiency promotes age-dependent alterations in the cortical synaptic proteome.Analysis of Gene Expression Profiles in the Human Brain Stem, Cerebellum and Cerebral CortexCaMKII regulation in information processing and storage.Translational Concepts of mGluR5 in Synaptic Diseases of the BrainGenetic variant analysis of the putative regulatory regions of the LRRC7 gene in bipolar disorder.DISC1 genetics, biology and psychiatric illnessThe CaMKII/NMDAR complex as a molecular memoryHeterozygous deletion of the LRFN2 gene is associated with working memory deficits.Modulation of behavior by scaffolding proteins of the post-synaptic density.Splice variants of the CaV1.3 L-type calcium channel regulate dendritic spine morphology.Synaptic Signaling in Learning and Memory.CaMKII: claiming center stage in postsynaptic function and organization.Disrupted in schizophrenia 1 and synaptic function in the mammalian central nervous system.The glutamatergic aspects of schizophrenia molecular pathophysiology: role of the postsynaptic density, and implications for treatmentRegulation of Postsynaptic Stability by the L-type Calcium Channel CaV1.3 and its Interaction with PDZ ProteinsGenetics of aggressive behavior: An overview.A Disc1 mutation differentially affects neurites and spines in hippocampal and cortical neurons.Densin-180 Controls the Trafficking and Signaling of L-Type Voltage-Gated Cav1.2 Ca2+ Channels at Excitatory Synapses.Densin-180 is not a transmembrane protein.Unique sensory and motor behavior in Thy1-GFP-M mice before and after spinal cord injury.Role of DISC1 interacting proteins in schizophrenia risk from genome-wide analysis of missense SNPs.NEURODEVELOPMENT. Adult cortical plasticity depends on an early postnatal critical period.
P2860
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P2860
Deletion of densin-180 results in abnormal behaviors associated with mental illness and reduces mGluR5 and DISC1 in the postsynaptic density fraction.
description
2011 nî lūn-bûn
@nan
2011 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Deletion of densin-180 results ...... postsynaptic density fraction.
@ast
Deletion of densin-180 results ...... postsynaptic density fraction.
@en
type
label
Deletion of densin-180 results ...... postsynaptic density fraction.
@ast
Deletion of densin-180 results ...... postsynaptic density fraction.
@en
prefLabel
Deletion of densin-180 results ...... postsynaptic density fraction.
@ast
Deletion of densin-180 results ...... postsynaptic density fraction.
@en
P2093
P2860
P1476
Deletion of densin-180 results ...... postsynaptic density fraction.
@en
P2093
Andrew D Steele
Andrew Medina-Marino
Eugenia Khorosheva
Holly J Carlisle
Keith M Gunapala
Leslie Schenker
Mary B Kennedy
Paul H Patterson
Thomas J O'Dell
Tim Indersmitten
P2860
P304
16194-16207
P356
10.1523/JNEUROSCI.5877-10.2011
P407
P577
2011-11-01T00:00:00Z