In contrast to kindled seizures, the frequency of spontaneous epilepsy in the limbic status model correlates with greater aberrant fascia dentata excitatory and inhibitory axon sprouting, and increased staining for N-methyl-D-aspartate, AMPA and GAB
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Electrophysiological evidence of monosynaptic excitatory transmission between granule cells after seizure-induced mossy fiber sproutingA functional mouse retroposed gene Rps23r1 reduces Alzheimer's beta-amyloid levels and tau phosphorylationInfluence of neuropathology on convection-enhanced delivery in the rat hippocampusHypothalamic neuronal loss and altered circadian rhythm of temperature in a rat model of mesial temporal lobe epilepsy.The pathological substrate of limbic epilepsy: neuronal loss in the medial dorsal thalamic nucleus as the consistent change.The midline thalamus: alterations and a potential role in limbic epilepsy.Phenobarbital and MK-801, but not phenytoin, improve the long-term outcome of status epilepticus.Mesial temporal lobe epilepsy: How do we improve surgical outcome?Temporal lobe epilepsy: where do the seizures really begin?The relevance of kindling for human epilepsy.Surviving hilar somatostatin interneurons enlarge, sprout axons, and form new synapses with granule cells in a mouse model of temporal lobe epilepsy.Topological determinants of epileptogenesis in large-scale structural and functional models of the dentate gyrus derived from experimental data.Chronic epileptogenic cellular alterations in the limbic system after status epilepticus.Hippocampal granule cell pathology in epilepsy - a possible structural basis for comorbidities of epilepsy?Early MR diffusion and relaxation changes in the parahippocampal gyrus precede the onset of spontaneous seizures in an animal model of chronic limbic epilepsy.Postlesional epilepsy: the ultimate brain plasticity.Increased excitatory synaptic input to granule cells from hilar and CA3 regions in a rat model of temporal lobe epilepsyRapamycin suppresses mossy fiber sprouting but not seizure frequency in a mouse model of temporal lobe epilepsy.Initial loss but later excess of GABAergic synapses with dentate granule cells in a rat model of temporal lobe epilepsy.Control of synchronization of brain dynamics leads to control of epileptic seizures in rodents.Candidate drug targets for prevention or modification of epilepsyImpaired maturation of cortical GABA(A) receptor expression in pediatric epilepsyStructure of cortical microcircuit theory.Accumulation of abnormal adult-generated hippocampal granule cells predicts seizure frequency and severity.The risk of epilepsy after status epilepticus in children and adults.Synaptic input to dentate granule cell basal dendrites in a rat model of temporal lobe epilepsy.Disease modification in epilepsy: from animal models to clinical applications.Electrophysiologic analysis of a chronic seizure model after unilateral hippocampal KA injection.Posttraumatic Epilepsy: What's Contusion Got to Do With It?Short- and long-term changes in CA1 network excitability after kainate treatment in rats.Fluorescent tracer in pilocarpine-treated rats shows widespread aberrant hippocampal neuronal connectivity.Long-lasting increased excitability differs in dentate gyrus vs. CA1 in freely moving chronic epileptic rats after electrically induced status epilepticus.Damage, reorganization, and abnormal neocortical hyperexcitability in the pilocarpine model of temporal lobe epilepsy.Differential and long-lasting alterations of high-voltage activated calcium currents in CA1 and dentate granule neurons after status epilepticus.Chronic lithium treatment inhibits pilocarpine-induced mossy fiber sprouting in rat hippocampus.Contributions of mossy fiber and CA1 pyramidal cell sprouting to dentate granule cell hyperexcitability in kainic acid-treated hippocampal slice cultures.Role of mossy fiber sprouting and mossy cell loss in hyperexcitability: a network model of the dentate gyrus incorporating cell types and axonal topography.Increased fast ripple to ripple ratios correlate with reduced hippocampal volumes and neuron loss in temporal lobe epilepsy patients.Changes in mIPSCs and sIPSCs after kainate treatment: evidence for loss of inhibitory input to dentate granule cells and possible compensatory responses.Effect of theophylline and trimethobenzamide when given during kainate-induced status epilepticus: an improved histopathologic rat model of human hippocampal sclerosis.
P2860
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P2860
In contrast to kindled seizures, the frequency of spontaneous epilepsy in the limbic status model correlates with greater aberrant fascia dentata excitatory and inhibitory axon sprouting, and increased staining for N-methyl-D-aspartate, AMPA and GAB
description
1997 nî lūn-bûn
@nan
1997 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
name
In contrast to kindled seizure ...... thyl-D-aspartate, AMPA and GAB
@ast
In contrast to kindled seizure ...... thyl-D-aspartate, AMPA and GAB
@en
type
label
In contrast to kindled seizure ...... thyl-D-aspartate, AMPA and GAB
@ast
In contrast to kindled seizure ...... thyl-D-aspartate, AMPA and GAB
@en
prefLabel
In contrast to kindled seizure ...... thyl-D-aspartate, AMPA and GAB
@ast
In contrast to kindled seizure ...... thyl-D-aspartate, AMPA and GAB
@en
P2093
P1433
P1476
In contrast to kindled seizure ...... thyl-D-aspartate, AMPA and GAB
@en
P2093
Bertram EH 3rd
Kuhlman PA
Mathern GW
Pretorius JK
Spradlin S
P304
P356
10.1016/S0306-4522(96)00516-7
P407
P577
1997-04-01T00:00:00Z