CD44 is a marker for the outer pillar cells in the early postnatal mouse inner ear. - Wikidata - awgldk - TriplyDB

CD44 is a marker for the outer pillar cells in the early postnatal mouse inner ear.

CD44 is a marker for the outer pillar cells in the early postnatal mouse inner ear.

http://www.wikidata.org/entity/Q30474323

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Fgf10 is required for specification of non-sensory regions of the cochlear epithelium.Integration of transcriptomics, proteomics, and microRNA analyses reveals novel microRNA regulation of targets in the mammalian inner ear Inhibition of MMP-2 but not MMP-9 influences inner ear spiral ganglion neurons in vitro.Molecular profile of cochlear immunity in the resident cells of the organ of Corti.Genetic rescue of Muenke syndrome model hearing loss reveals prolonged FGF-dependent plasticity in cochlear supporting cell fates.Inner ear supporting cells: rethinking the silent majority.Correct timing of proliferation and differentiation is necessary for normal inner ear development and auditory hair cell viability.Intrinsic regenerative potential of murine cochlear supporting cells.Inner ear hair cell-like cells from human embryonic stem cells.Cell type-specific transcriptome analysis reveals a major role for Zeb1 and miR-200b in mouse inner ear morphogenesis.Thyroid hormone increases fibroblast growth factor receptor expression and disrupts cell mechanics in the developing organ of corti CD44 is required for spatial memory retention and sensorimotor functions Macrophage migration inhibitory factor acts as a neurotrophin in the developing inner ear.Cdc42-dependent structural development of auditory supporting cells is required for wound healing at adulthood.Quantitative Analysis of Supporting Cell Subtype Labeling Among CreER Lines in the Neonatal Mouse Cochlea.Hyaluronic acid pretreatment for Sendai virus-mediated cochlear gene transfer.The cytokine macrophage migration inhibitory factor (MIF) acts as a neurotrophin in the developing inner ear of the zebrafish, Danio rerio.Intracellular Regulome Variability Along the Organ of Corti: Evidence, Approaches, Challenges, and Perspective.

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P2860

CD44 is a marker for the outer pillar cells in the early postnatal mouse inner ear.

http://www.wikidata.org/entity/Q30474323

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

CD44 is a marker for the outer pillar cells in the early postnatal mouse inner ear.

@ast

CD44 is a marker for the outer pillar cells in the early postnatal mouse inner ear.

@en

type

label

CD44 is a marker for the outer pillar cells in the early postnatal mouse inner ear.

@ast

CD44 is a marker for the outer pillar cells in the early postnatal mouse inner ear.

@en

prefLabel

CD44 is a marker for the outer pillar cells in the early postnatal mouse inner ear.

@ast

CD44 is a marker for the outer pillar cells in the early postnatal mouse inner ear.

@en

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CD44 is a marker for the outer pillar cells in the early postnatal mouse inner ear.

@en

P2093

Caroline Timothy

http://www.w3.org/2001/XMLSchema#string

Chandrakala Puligilla

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David J Eisenman

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Didier A Depireux

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Jeffrey Wolf

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Matthew W Kelley

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Ronna Hertzano

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Scott E Strome

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Sheikh Riazuddin

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Siaw-Lin Chan

http://www.w3.org/2001/XMLSchema#string

P2860

Mammalian cochlear supporting cells can divide and trans-differentiate into hair cells

Mutations of the protocadherin gene PCDH15 cause Usher syndrome type 1F

The Ina and Inb blood group antigens are located on a glycoprotein of 80,000 MW (the CDw44 glycoprotein) whose expression is influenced by the In(Lu) gene

Expression in cochlea and retina of myosin VIIa, the gene product defective in Usher syndrome type 1B

CD44 cell adhesion molecules

Expression of Delta1 and Serrate1 (Jagged1) in the mouse inner ear

Structure and innervation of the cochlea

CD44 is the principal cell surface receptor for hyaluronate

Hey2 regulation by FGF provides a Notch-independent mechanism for maintaining pillar cell fate in the organ of Corti

Loss of Fgfr3 leads to excess hair cell development in the mouse organ of Corti

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s10162-010-0211-x

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407-418

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