Helicobacter pylori VacA cytotoxin: a probe for a clathrin-independent and Cdc42-dependent pinocytic pathway routed to late endosomes.
about
Relationship between VacA Toxin and Host Cell Autophagy in Helicobacter pylori Infection of the Human Stomach: A Few Answers, Many QuestionsAn Overview of Helicobacter pylori VacA Toxin BiologyA role for altered phagosome maturation in the long-term persistence of Helicobacter pylori infectionBacillus sphaericus binary toxin elicits host cell autophagy as a response to intoxicationEndocytic crosstalk: cavins, caveolins, and caveolae regulate clathrin-independent endocytosisA heparan sulfate-facilitated and raft-dependent macropinocytosis of eosinophil cationic proteinEarly endosomes associated with dynamic F-actin structures are required for late trafficking of H. pylori VacA toxin.Plasma membrane area increases with spread area by exocytosis of a GPI-anchored protein compartment.Sphingomyelin functions as a novel receptor for Helicobacter pylori VacA.Lipid rafts and clathrin cooperate in the internalization of PrP in epithelial FRT cellsAnalysis of endocytic pathways in Drosophila cells reveals a conserved role for GBF1 in internalization via GEECs.Helicobacter pylori counteracts the apoptotic action of its VacA toxin by injecting the CagA protein into gastric epithelial cells.Insight into nanoparticle cellular uptake and intracellular targetingSphingomyelin is important for the cellular entry and intracellular localization of Helicobacter pylori VacA.Endocytic Trafficking of Nanoparticles Delivered by Cell-penetrating Peptides Comprised of Nona-arginine and a Penetration Accelerating SequenceDistinct mechanisms of clathrin-independent endocytosis have unique sphingolipid requirements.Role of connexin 43 in Helicobacter pylori VacA-induced cell death.Cholesterol depletion reduces entry of Campylobacter jejuni cytolethal distending toxin and attenuates intoxication of host cellsClustering of Helicobacter pylori VacA in lipid rafts, mediated by its receptor, receptor-like protein tyrosine phosphatase beta, is required for intoxication in AZ-521 Cells.Helicobacter pylori VacA induces programmed necrosis in gastric epithelial cells.the versatility of the Helicobacter pylori vacuolating cytotoxin vacA in signal transduction and molecular crosstalk.Vacuolating cytotoxin in Helicobacter pylori water-soluble proteins upregulates chemokine expression in human eosinophils via Ca2+ influx, mitochondrial reactive oxygen intermediates, and NF-kappaB activation.S-palmitoylation and ubiquitination differentially regulate interferon-induced transmembrane protein 3 (IFITM3)-mediated resistance to influenza virusAck1 is a dopamine transporter endocytic brake that rescues a trafficking-dysregulated ADHD coding variant.About lipids and toxins.Bacterial protein toxins and lipids: role in toxin targeting and activity.Gold nanoparticle conjugated Rad6 inhibitor induces cell death in triple negative breast cancer cells by inducing mitochondrial dysfunction and PARP-1 hyperactivation: Synthesis and characterization.Helicobacter pylori VacA disrupts apical membrane-cytoskeletal interactions in gastric parietal cellsHelicobacter pylori VacA suppresses Lactobacillus acidophilus-induced interferon beta signaling in macrophages via alterations in the endocytic pathwayEndocytosis as a mechanism of regulating natural killer cell function: unique endocytic and trafficking pathway for CD94/NKG2A.Steric and not structure-specific factors dictate the endocytic mechanism of glycosylphosphatidylinositol-anchored proteinsEndocytosis unplugged: multiple ways to enter the cell.Multifaceted interactions of bacterial toxins with the gastrointestinal mucosa.Remodeling the host environment: modulation of the gastric epithelium by the Helicobacter pylori vacuolating toxin (VacA).Endocytosis of gene delivery vectors: from clathrin-dependent to lipid raft-mediated endocytosis.Characterization of Helicobacter pylori VacA-containing vacuoles (VCVs), VacA intracellular trafficking and interference with calcium signalling in T lymphocytes.Uptake of Helicobacter pylori vesicles is facilitated by clathrin-dependent and clathrin-independent endocytic pathways.PKC-dependent endocytosis of the Helicobacter pylori vacuolating cytotoxin in primary T lymphocytes.Pore-forming epsilon toxin causes membrane permeabilization and rapid ATP depletion-mediated cell death in renal collecting duct cells.The cytotoxic necrotizing factors from Yersinia pseudotuberculosis and from Escherichia coli bind to different cellular receptors but take the same route to the cytosol.
P2860
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P2860
Helicobacter pylori VacA cytotoxin: a probe for a clathrin-independent and Cdc42-dependent pinocytic pathway routed to late endosomes.
description
2005 nî lūn-bûn
@nan
2005 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Helicobacter pylori VacA cytot ...... hway routed to late endosomes.
@ast
Helicobacter pylori VacA cytot ...... hway routed to late endosomes.
@en
type
label
Helicobacter pylori VacA cytot ...... hway routed to late endosomes.
@ast
Helicobacter pylori VacA cytot ...... hway routed to late endosomes.
@en
prefLabel
Helicobacter pylori VacA cytot ...... hway routed to late endosomes.
@ast
Helicobacter pylori VacA cytot ...... hway routed to late endosomes.
@en
P2093
P2860
P356
P1476
Helicobacter pylori VacA cytot ...... hway routed to late endosomes.
@en
P2093
Nils C Gauthier
Pascale Monzo
Patrice Boquet
Vincent Kaddai
Vittorio Ricci
P2860
P304
P356
10.1091/MBC.E05-05-0398
P577
2005-07-29T00:00:00Z