c-Ret-mediated hearing loss in mice with Hirschsprung disease - Wikidata - awgldk - TriplyDB

c-Ret-mediated hearing loss in mice with Hirschsprung disease

c-Ret-mediated hearing loss in mice with Hirschsprung disease

http://www.wikidata.org/entity/Q30477530

about

Partial requirement of endothelin receptor B in spiral ganglion neurons for postnatal development of hearing Manganese-mediated acceleration of age-related hearing loss in mice.Chronic exposure to low frequency noise at moderate levels causes impaired balance in mice.Risk Assessment of Neonatal Exposure to Low Frequency Noise Based on Balance in Mice Genetic background strongly modifies the severity of symptoms of Hirschsprung disease, but not hearing loss in rats carrying Ednrb(sl) mutations.Deltex-3-like (DTX3L) stimulates metastasis of melanoma through FAK/PI3K/AKT but not MEK/ERK pathway.Reduced GNG2 expression levels in mouse malignant melanomas and human melanoma cell lines.Glial cell line-derived neurotrophic factor (GDNF) induces neuritogenesis in the cochlear spiral ganglion via neural cell adhesion molecule (NCAM)RAS/RAF/MEK/ERK and PI3K/PTEN/AKT Signaling in Malignant Melanoma Progression and Therapy.Molecular Network Associated with MITF in Skin Melanoma Development and Progression.c-Ret-mediated hearing losses.Hearing impairments caused by genetic and environmental factors.GNG2 inhibits invasion of human malignant melanoma cells with decreased FAK activity.Transcription factor LSF (TFCP2) inhibits melanoma growth.Enhanced carcinogenicity by coexposure to arsenic and iron and a novel remediation system for the elements in well drinking water.Barium inhibits arsenic-mediated apoptotic cell death in human squamous cell carcinoma cells.Correlated expression levels of endothelin receptor B and Plexin C1 in melanoma.Oral exposure to arsenic causes hearing loss in young people aged 12-29 years and in young mice.Commentary to Gorelenkova Miller and Mieyal (2015): sulfhydryl-mediated redox signaling in inflammation: role in neurodegenerative diseases.Characterization of retinal ganglion cell, horizontal cell, and amacrine cell types expressing the neurotrophic receptor tyrosine kinase Ret.Different biological effects of a constant dose for single UVB irradiation with different intensities and exposure times.Manganese-Mediated Decrease in Levels of c-RET and Tyrosine Hydroxylase Expression In Vitro.

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c-Ret-mediated hearing loss in mice with Hirschsprung disease

http://www.wikidata.org/entity/Q30477530

description

2010 nî lūn-bûn

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2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2010 թվականի հունիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

c-Ret-mediated hearing loss in mice with Hirschsprung disease

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c-Ret-mediated hearing loss in mice with Hirschsprung disease

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c-Ret-mediated hearing loss in mice with Hirschsprung disease

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c-Ret-mediated hearing loss in mice with Hirschsprung disease

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c-Ret-mediated hearing loss in mice with Hirschsprung disease

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c-Ret-mediated hearing loss in mice with Hirschsprung disease

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PNAS.1004520107

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Proceedings of the National Academy of Sciences of the United States of America

P1476

c-Ret-mediated hearing loss in mice with Hirschsprung disease

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P2093

Akira Hara

http://www.w3.org/2001/XMLSchema#string

Atsuyoshi Shimada

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Keiji Tabuchi

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Masahide Takahashi

http://www.w3.org/2001/XMLSchema#string

Masahiko Yamamoto

http://www.w3.org/2001/XMLSchema#string

Masashi Kato

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Mayumi Jijiwa

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Michihiko Sone

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Michiru Ida-Eto

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Naomi Sakashita

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P2860

The GDNF family: signalling, biological functions and therapeutic value

SOX10 mutations in patients with Waardenburg-Hirschsprung disease

Mutation of the endothelin-receptor B gene in Waardenburg-Hirschsprung disease

Expression of the GDNF family members and their receptors in the mature rat cochlea

Assessment of hearing in 80 inbred strains of mice by ABR threshold analyses.

Defects in the kidney and enteric nervous system of mice lacking the tyrosine kinase receptor Ret

Spiral ganglion neurons are protected from degeneration by GDNF gene therapy.

Nuclear factor kappaB deficiency is associated with auditory nerve degeneration and increased noise-induced hearing loss.

Diminished Ret expression compromises neuronal survival in the colon and causes intestinal aganglionosis in mice.

NF-kappaB in neuronal plasticity and neurodegenerative disorders

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scholarly article

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20616061

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2919946

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