Connexin30 deficiency causes instrastrial fluid-blood barrier disruption within the cochlear stria vascularis. - Wikidata - awgldk - TriplyDB

Connexin30 deficiency causes instrastrial fluid-blood barrier disruption within the cochlear stria vascularis.

Connexin30 deficiency causes instrastrial fluid-blood barrier disruption within the cochlear stria vascularis.

http://www.wikidata.org/entity/Q30479250

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Gap junction mediated intercellular metabolite transfer in the cochlea is compromised in connexin30 null mice Identification and characterization of pannexin expression in the mammalian cochlea The endocochlear potential depends on two K+ diffusion potentials and an electrical barrier in the stria vascularis of the inner ear Human adenylate kinase 2 deficiency causes a profound hematopoietic defect associated with sensorineural deafness Early development of the vertebrate inner ear Folic acid deficiency induces premature hearing loss through mechanisms involving cochlear oxidative stress and impairment of homocysteine metabolism Na+/K+-ATPase α1 identified as an abundant protein in the blood-labyrinth barrier that plays an essential role in the barrier integrity The unique electrical properties in an extracellular fluid of the mammalian cochlea; their functional roles, homeostatic processes, and pathological significance.Coordinated control of connexin 26 and connexin 30 at the regulatory and functional level in the inner ear The human deafness-associated connexin 30 T5M mutation causes mild hearing loss and reduces biochemical coupling among cochlear non-sensory cells in knock-in mice.A connexin30 mutation rescues hearing and reveals roles for gap junctions in cochlear amplification and micromechanics Organ of Corti and Stria Vascularis: Is there an Interdependence for Survival?Immunohistochemical localization of OCT2 in the cochlea of various species.Structural changes in thestrial blood-labyrinth barrier of aged C57BL/6 mice.Potassium ion movement in the inner ear: insights from genetic disease and mouse models Lipopolysaccharide-induced middle ear inflammation disrupts the cochlear intra-strial fluid-blood barrier through down-regulation of tight junction proteins.Genetics of auditory mechano-electrical transduction.Perivascular macrophage-like melanocyte responsiveness to acoustic trauma--a salient feature of strial barrier associated hearing loss.Feline deafness.Endothelial cell, pericyte, and perivascular resident macrophage-type melanocyte interactions regulate cochlear intrastrial fluid-blood barrier permeability Physiopathology of the cochlear microcirculation Calcium signaling in the cochlea - Molecular mechanisms and physiopathological implications.Perivascular-resident macrophage-like melanocytes in the inner ear are essential for the integrity of the intrastrial fluid-blood barrier.BAAV mediated GJB2 gene transfer restores gap junction coupling in cochlear organotypic cultures from deaf Cx26Sox10Cre mice.Advances in Auditory and Vestibular Medicine.Gap-junction channels dysfunction in deafness and hearing loss.ATP-mediated cell-cell signaling in the organ of Corti: the role of connexin channels.Cochlear Homocysteine Metabolism at the Crossroad of Nutrition and Sensorineural Hearing Loss.Matrix imbalance by inducing expression of metalloproteinase and oxidative stress in cochlea of hyperhomocysteinemic mice.Gap junctions and blood-tissue barriers Competitive antagonism of fluorescent gentamicin uptake in the cochlea The role of gap junction channels during physiologic and pathologic conditions of the human central nervous system Serum homocysteine and folate concentrations are associated with prevalent age-related hearing loss.Spinster homolog 2 (spns2) deficiency causes early onset progressive hearing loss.Molecular architecture of the stria vascularis membrane transport system, which is essential for physiological functions of the mammalian cochlea.Deletion of astroglial connexins weakens the blood-brain barrier.Folic acid improves inner ear vascularization in hyperhomocysteinemic mice Strial microvascular pathology and age-associated endocochlear potential decline in NOD congenic mice.Isolation and culture of endothelial cells, pericytes and perivascular resident macrophage-like melanocytes from the young mouse ear.Cx36 makes channels coupling human pancreatic beta-cells, and correlates with insulin expression.

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P2860

Connexin30 deficiency causes instrastrial fluid-blood barrier disruption within the cochlear stria vascularis.

http://www.wikidata.org/entity/Q30479250

description

2007 nî lūn-bûn

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2007 թուականի Մարտին հրատարակուած գիտական յօդուած

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2007 թվականի մարտին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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PNAS.0605108104

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Proceedings of the National Academy of Sciences of the United States of America

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Connexin30 deficiency causes i ...... the cochlear stria vascularis.

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Béatrice Regnault

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Dorothée Caille

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Karine Demuth

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Nadège Cayet

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Paolo Meda

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P2860

Deafness and imbalance associated with inactivation of the secretory Na-K-2Cl co-transporter

Mutations in GJB6 cause nonsyndromic autosomal dominant deafness at DFNA3 locus

The enzymatic synthesis of S-adenosyl-L-homocysteine from adenosine and homocysteine

Transgenic expression of a dominant-negative connexin26 causes degeneration of the organ of Corti and non-syndromic deafness

Expression of the gap-junction connexins 26 and 30 in the rat cochlea

Gap junctions in the inner ear: comparison of distribution patterns in different vertebrates and assessement of connexin composition in mammals

Localization of connexin 30 in the luminal membrane of cells in the distal nephron

Endothelial dysfunction in a murine model of mild hyperhomocyst(e)inemia

Connexin30 (Gjb6)-deficiency causes severe hearing impairment and lack of endocochlear potential

Gap junctional hemichannel-mediated ATP release and hearing controls in the inner ear

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15

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104

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Martine Cohen-Salmon

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51382492

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17400755

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1851033

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