Deficits in adult prefrontal cortex neurons and behavior following early post-natal NMDA antagonist treatment
about
GABAergic interneuron origin of schizophrenia pathophysiology.Spatial and temporal boundaries of NMDA receptor hypofunction leading to schizophrenia.Region-specific alteration in brain glutamate: possible relationship to risk-taking behavior.Adolescent binge drinking alters adult brain neurotransmitter gene expression, behavior, brain regional volumes, and neurochemistry in mice.Postnatal day 7 ethanol treatment causes persistent reductions in adult mouse brain volume and cortical neurons with sex specific effects on neurogenesis.Adolescent binge ethanol treatment alters adult brain regional volumes, cortical extracellular matrix protein and behavioral flexibility.Clinical physiology and mechanism of dizocilpine (MK-801): electron transfer, radicals, redox metabolites and bioactivityAntioxidant Treatment with N-acetyl Cysteine Prevents the Development of Cognitive and Social Behavioral Deficits that Result from Perinatal Ketamine TreatmentNeonatal NMDA receptor blockade disrupts spike timing and glutamatergic synapses in fast spiking interneurons in a NMDA receptor hypofunction model of schizophrenia.Prolonged exposure to NMDAR antagonist induces cell-type specific changes of glutamatergic receptors in rat prefrontal cortex.Periadolescent exposure to the NMDA receptor antagonist MK-801 impairs the functional maturation of local GABAergic circuits in the adult prefrontal cortexNonsteroidal anti-inflammatory treatment prevents delayed effects of early life stress in rats.Current hypotheses on the mechanisms of alcoholism.Neuronal survival in the brain: neuron type-specific mechanisms.Beyond dopamine: glutamate as a target for future antipsychotics.Antioxidant Treatment in Male Mice Prevents Mitochondrial and Synaptic Changes in an NMDA Receptor Dysfunction Model of Schizophrenia.Npas4 deficiency and prenatal stress interact to affect social recognition in mice.HMGB1/IL-1β complexes regulate neuroimmune responses in alcoholism.Short-Term Exposure to Enriched Environment in Adult Rats Restores MK-801-Induced Cognitive Deficits and GABAergic Interneuron Immunoreactivity Loss.Sub-chronic Antipsychotic Drug Administration Reverses the Expression of Neuregulin 1 and ErbB4 in a Cultured MK801-Induced Mouse Primary Hippocampal Neuron or a Neurodevelopmental Schizophrenia Model.Neonatal alcohol exposure reduces number of parvalbumin-positive interneurons in the medial prefrontal cortex and impairs passive avoidance acquisition in mice deficits not rescued from exercise.
P2860
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P2860
Deficits in adult prefrontal cortex neurons and behavior following early post-natal NMDA antagonist treatment
description
2009 nî lūn-bûn
@nan
2009 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Deficits in adult prefrontal c ...... atal NMDA antagonist treatment
@ast
Deficits in adult prefrontal c ...... atal NMDA antagonist treatment
@en
type
label
Deficits in adult prefrontal c ...... atal NMDA antagonist treatment
@ast
Deficits in adult prefrontal c ...... atal NMDA antagonist treatment
@en
prefLabel
Deficits in adult prefrontal c ...... atal NMDA antagonist treatment
@ast
Deficits in adult prefrontal c ...... atal NMDA antagonist treatment
@en
P2093
P2860
P1476
Deficits in adult prefrontal c ...... atal NMDA antagonist treatment
@en
P2093
Fulton T Crews
L Fredrik Jarskog
Leon G Coleman
Sheryl S Moy
P2860
P304
P356
10.1016/J.PBB.2009.04.017
P407
P577
2009-05-03T00:00:00Z