Toll-like receptor 1 polymorphisms affect innate immune responses and outcomes in sepsis. - Wikidata - awgldk - TriplyDB

Toll-like receptor 1 polymorphisms affect innate immune responses and outcomes in sepsis.

Toll-like receptor 1 polymorphisms affect innate immune responses and outcomes in sepsis.

http://www.wikidata.org/entity/Q30483818

about

A TIR domain variant of MyD88 adapter-like (Mal)/TIRAP results in loss of MyD88 binding and reduced TLR2/TLR4 signaling Proinflammatory responses by glycosylphosphatidylinositols (GPIs) of Plasmodium falciparum are mainly mediated through the recognition of TLR2/TLR1 Single nucleotide polymorphisms of Toll-like receptors and susceptibility to infectious diseases A genome-wide in vitro bacterial-infection screen reveals human variation in the host response associated with inflammatory disease Genetic variation in the FAS gene and associations with acute lung injury.Toll-like receptor 4 region genetic variants are associated with susceptibility to melioidosis.Immunogenomics and systems biology of vaccines.Characterizing sepsis: Another small piece of the puzzle.Homozygosity and risk of childhood death due to invasive bacterial disease.Toll-like receptor polymorphisms and susceptibility to urinary tract infections in adult women.Genetic variation of the human urinary tract innate immune response and asymptomatic bacteriuria in women.Genetic polymorphisms associated with acute lung injury.Leprosy and the adaptation of human toll-like receptor 1.Common variants of TLR1 associate with organ dysfunction and sustained pro-inflammatory responses during sepsis.Genetic variants in the TIRAP gene are associated with increased risk of sepsis-associated acute lung injury Effects of the TLR2 agonists MALP-2 and Pam3Cys in isolated mouse lungs Role of innate host defenses in susceptibility to early-onset neonatal sepsis.Pathophysiology and treatment of septic shock in neonates.Association of human TLR1 and TLR6 deficiency with altered immune responses to BCG vaccination in South African infants.APOε4 is associated with enhanced in vivo innate immune responses in human subjects Association of genetic variants in the IRAK-4 gene with susceptibility to severe sepsis.Polymorphisms in toll-like receptor 4 and toll-like receptor 9 influence viral load in a seroincident cohort of HIV-1-infected individuals Genetic variants of innate immune receptors and infections after liver transplantation.Genome wide association identifies PPFIA1 as a candidate gene for acute lung injury risk following major trauma.SNP-set analysis replicates acute lung injury genetic risk factors.A functional synonymous coding variant in the IL1RN gene is associated with survival in septic shock.Leprosy and the human genome Secreted phosphoprotein 1 is a determinant of lung function development in mice.Genetic polymorphisms and posttraumatic complications.Association of Toll-like receptor polymorphisms with HIV status in North Americans Cutting edge: Genetic variation in TLR1 is associated with Pam3CSK4-induced effector T cell resistance to regulatory T cell suppression.ANGPT2 genetic variant is associated with trauma-associated acute lung injury and altered plasma angiopoietin-2 isoform ratio.Survey of innate immune responses to Burkholderia pseudomallei in human blood identifies a central role for lipopolysaccharide.Common TLR1 genetic variation is not associated with death from melioidosis, a common cause of sepsis in rural Thailand.Experimental models and emerging hypotheses for acute lung injury.A nonsynonymous polymorphism of IRAK4 associated with increased prevalence of gram-positive infection and decreased response to toll-like receptor ligands Variants in the Toll-interacting protein gene are associated with susceptibility to sepsis in the Chinese Han population.Role of type 2 deiodinase in response to acute lung injury (ALI) in mice.Variants in toll-like receptor 1 and 4 genes are associated with Chlamydia trachomatis among women with pelvic inflammatory disease Toll-like receptor 1 polymorphisms increase susceptibility to candidemia.

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P2860

Toll-like receptor 1 polymorphisms affect innate immune responses and outcomes in sepsis.

http://www.wikidata.org/entity/Q30483818

description

2008 nî lūn-bûn

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2008 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի հուլիսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Toll-like receptor 1 polymorphisms affect innate immune responses and outcomes in sepsis.

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Toll-like receptor 1 polymorphisms affect innate immune responses and outcomes in sepsis.

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type

label

Toll-like receptor 1 polymorphisms affect innate immune responses and outcomes in sepsis.

@ast

Toll-like receptor 1 polymorphisms affect innate immune responses and outcomes in sepsis.

@en

prefLabel

Toll-like receptor 1 polymorphisms affect innate immune responses and outcomes in sepsis.

@ast

Toll-like receptor 1 polymorphisms affect innate immune responses and outcomes in sepsis.

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American Journal of Respiratory and Critical Care Medicine

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Toll-like receptor 1 polymorphisms affect innate immune responses and outcomes in sepsis.

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P2093

Adeline M Hajjar

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Deborah A Nickerson

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Frank Radella

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Gail Rona

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James A Russell

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Joe G N Garcia

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John T Ruzinski

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Jonathan Sevransky

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P2860

TLR4 mutations are associated with endotoxin hyporesponsiveness in humans

Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. The ACCP/SCCM Consensus Conference Committee. American College of Chest Physicians/Society of Critical Care Medicine

Cutting edge: role of Toll-like receptor 1 in mediating immune response to microbial lipoproteins

Statistical significance for genomewide studies

Toll-like receptors

Signalling pathways and molecular interactions of NOD1 and NOD2

A Mal functional variant is associated with protection against invasive pneumococcal disease, bacteremia, malaria and tuberculosis

Cutting edge: functional interactions between toll-like receptor (TLR) 2 and TLR1 or TLR6 in response to phenol-soluble modulin

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710-720

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178

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Carl B Shanholtz

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