Modulation of the Rho/ROCK pathway in heart and lung after thorax irradiation reveals targets to improve normal tissue toxicity.
about
Pathology and biology of radiation-induced cardiac diseaseRac1-regulated endothelial radiation response stimulates extravasation and metastasis that can be blocked by HMG-CoA reductase inhibitorsMolecular mechanisms and treatment of radiation-induced lung fibrosisEnhanced sensitivity to low dose irradiation of ApoE-/- mice mediated by early pro-inflammatory profile and delayed activation of the TGFβ1 cascade involved in fibrogenesis.Relaxin regulates myofibroblast contractility and protects against lung fibrosisRole of Interleukin-1 in Radiation-Induced Cardiomyopathy.Radiosensitization by a novel Bcl-2 and Bcl-XL inhibitor S44563 in small-cell lung cancer.Therapeutic management of intestinal fibrosis induced by radiation therapy: from molecular profiling to new intervention strategies et vice et versaNormal tissues toxicities triggered by combined anti-angiogenic and radiation therapies: hurdles might be aheadProtection from Radiation-Induced Pulmonary Fibrosis by Peripheral Targeting of Cannabinoid Receptor-1.Cardiovascular complications of radiation therapy for thoracic malignancies: the role for non-invasive imaging for detection of cardiovascular disease.Potential use of HMG-CoA reductase inhibitors (statins) as radioprotective agents.Inhibition of Rac1 signaling by lovastatin protects against anthracycline-induced cardiac toxicity.Rho inhibition by lovastatin affects apoptosis and DSB repair of primary human lung cells in vitro and lung tissue in vivo following fractionated irradiationTAZ contributes to pulmonary fibrosis by activating profibrotic functions of lung fibroblasts.Effects of ionizing radiation on the heart.X-ray dose delivered during a longitudinal micro-CT study has no adverse effect on cardiac and pulmonary tissue in C57BL/6 mice.Syndecan-2 Attenuates Radiation-induced Pulmonary Fibrosis and Inhibits Fibroblast Activation by Regulating PI3K/Akt/ROCK Pathway via CD148.Proteomics landscape of radiation-induced cardiovascular disease: somewhere over the paradigm.Ultrahigh dose-rate FLASH irradiation increases the differential response between normal and tumor tissue in mice.Molecular Mechanisms and Treatment of Radiation-Induced Lung Fibrosis.
P2860
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P2860
Modulation of the Rho/ROCK pathway in heart and lung after thorax irradiation reveals targets to improve normal tissue toxicity.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Modulation of the Rho/ROCK pat ...... mprove normal tissue toxicity.
@ast
Modulation of the Rho/ROCK pat ...... mprove normal tissue toxicity.
@en
type
label
Modulation of the Rho/ROCK pat ...... mprove normal tissue toxicity.
@ast
Modulation of the Rho/ROCK pat ...... mprove normal tissue toxicity.
@en
prefLabel
Modulation of the Rho/ROCK pat ...... mprove normal tissue toxicity.
@ast
Modulation of the Rho/ROCK pat ...... mprove normal tissue toxicity.
@en
P2093
P50
P1433
P1476
Modulation of the Rho/ROCK pat ...... improve normal tissue toxicity
@en
P2093
Charlotte Schupp
Paule Opolon
Virginie Monceau
P304
P356
10.2174/1389450111009011395
P577
2010-11-01T00:00:00Z