COL25A1 triggers and promotes Alzheimer's disease-like pathology in vivo. - Wikidata - awgldk - TriplyDB

COL25A1 triggers and promotes Alzheimer's disease-like pathology in vivo.

COL25A1 triggers and promotes Alzheimer's disease-like pathology in vivo.

http://www.wikidata.org/entity/Q30492796

about

Galantamine slows down plaque formation and behavioral decline in the 5XFAD mouse model of Alzheimer's disease.Neuroimaging and genetic risk for Alzheimer's disease and addiction-related degenerative brain disorders.Gene expression profiling and association with prion-related lesions in the medulla oblongata of symptomatic natural scrapie animals.A role for collagen XXIII in cancer cell adhesion, anchorage-independence and metastasis A distinct subfraction of Aβ is responsible for the high-affinity Pittsburgh compound B-binding site in Alzheimer's disease brain Integrated model of de novo and inherited genetic variants yields greater power to identify risk genes.A peptide study of the relationship between the collagen triple-helix and amyloid Genome-wide methylation profiling identifies hypermethylated biomarkers in high-grade cervical intraepithelial neoplasia.Association of COL25A1 with comorbid antisocial personality disorder and substance dependence.Genome-wide analysis of a Wnt1-regulated transcriptional network implicates neurodegenerative pathways.Genome-wide identification of microRNA-related variants associated with risk of Alzheimer's disease.Unraveling the genes implicated in Alzheimer's disease.Genome-wide association analysis of age at onset in schizophrenia in a European-American sample.

P2860

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P2860

COL25A1 triggers and promotes Alzheimer's disease-like pathology in vivo.

http://www.wikidata.org/entity/Q30492796

description

2009 nî lūn-bûn

@nan

2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հունիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

@zh-hk

2009年論文

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2009年論文

@zh-tw

2009年论文

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name

COL25A1 triggers and promotes Alzheimer's disease-like pathology in vivo.

@ast

COL25A1 triggers and promotes Alzheimer's disease-like pathology in vivo.

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type

label

COL25A1 triggers and promotes Alzheimer's disease-like pathology in vivo.

@ast

COL25A1 triggers and promotes Alzheimer's disease-like pathology in vivo.

@en

prefLabel

COL25A1 triggers and promotes Alzheimer's disease-like pathology in vivo.

@ast

COL25A1 triggers and promotes Alzheimer's disease-like pathology in vivo.

@en

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COL25A1 triggers and promotes Alzheimer's disease-like pathology in vivo.

@en

P2093

Kimberly Scearce-Levie

http://www.w3.org/2001/XMLSchema#string

Louis J Ptácek

http://www.w3.org/2001/XMLSchema#string

Ying Tong

http://www.w3.org/2001/XMLSchema#string

Ying Xu

http://www.w3.org/2001/XMLSchema#string

Ying-Hui Fu

http://www.w3.org/2001/XMLSchema#string

P2860

CLAC: a novel Alzheimer amyloid plaque component derived from a transmembrane precursor, CLAC-P/collagen type XXV

Mostly separate distributions of CLAC- versus Abeta40- or thioflavin S-reactivities in senile plaques reveal two distinct subpopulations of beta-amyloid deposits

Characterization of the Alzheimer's disease-associated CLAC protein and identification of an amyloid beta-peptide-binding site

CLAC binds to amyloid beta peptides through the positively charged amino acid cluster within the collagenous domain 1 and inhibits formation of amyloid fibrils

Lipids revert inert Abeta amyloid fibrils to neurotoxic protofibrils that affect learning in mice

A hippocampal Cdk5 pathway regulates extinction of contextual fear

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

Conversion of p35 to p25 deregulates Cdk5 activity and promotes neurodegeneration

Genetic background changes the pattern of forebrain commissure defects in transgenic mice underexpressing the beta-amyloid-precursor protein

Common structure of soluble amyloid oligomers implies common mechanism of pathogenesis

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41-52

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1

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11

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2009-06-23T00:00:00Z

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26312504

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19548013

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Alzheimer's disease

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2807601

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