Glutamate co-release at GABA/glycinergic synapses is crucial for the refinement of an inhibitory map. - Wikidata - awgldk - TriplyDB

Glutamate co-release at GABA/glycinergic synapses is crucial for the refinement of an inhibitory map.

Glutamate co-release at GABA/glycinergic synapses is crucial for the refinement of an inhibitory map.

http://www.wikidata.org/entity/Q30493499

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Fragile x syndrome Dynamic regulation of neurotransmitter specification: relevance to nervous system homeostasis A behavioral framework to guide research on central auditory development and plasticity.Dual-transmitter neurons: functional implications of co-release and co-transmission Rescue of inhibitory synapse strength following developmental hearing loss Plasticity of local GABAergic interneurons drives olfactory habituation Glutamatergic phenotype of glucagon-like peptide 1 neurons in the caudal nucleus of the solitary tract in rats Hyperpolarization-independent maturation and refinement of GABA/glycinergic connections in the auditory brain stem.Noradrenergic refinement of glutamatergic neuronal circuits in the lateral superior olivary nucleus before hearing onset.Excitation by Axon Terminal GABA Spillover in a Sound Localization Circuit Restoration of Mecp2 expression in GABAergic neurons is sufficient to rescue multiple disease features in a mouse model of Rett syndrome.Postnatal development of synaptic properties of the GABAergic projection from the inferior colliculus to the auditory thalamus.The precise temporal pattern of prehearing spontaneous activity is necessary for tonotopic map refinement Development of intrinsic connectivity in the central nucleus of the mouse inferior colliculus.Yes, there is a medial nucleus of the trapezoid body in humans.Glycinergic synaptic transmission in the cochlear nucleus of mice with normal hearing and age-related hearing loss.Time-dependent gene expression analysis of the developing superior olivary complex.Glutamatergic inputs and glutamate-releasing immature inhibitory inputs activate a shared postsynaptic receptor population in lateral superior olive.Lateral superior olive function in congenital deafness.Synaptic plasticity in inhibitory neurons of the auditory brainstem Synaptic changes underlying the strengthening of GABA/glycinergic connections in the developing lateral superior olive.GABA(A) Receptors: Post-Synaptic Co-Localization and Cross-Talk with Other Receptors.Functional refinement in the projection from ventral cochlear nucleus to lateral superior olive precedes hearing onset in rat.A population of glomerular glutamatergic neurons controls sensory information transfer in the mouse olfactory bulb.Fluorescent labeling of newborn dentate granule cells in GAD67-GFP transgenic mice: a genetic tool for the study of adult neurogenesis.Expression of GABAergic and glutamatergic phenotypic markers in hypothalamic proopiomelanocortin neurons.Activation of reciprocal pathways between arcuate nucleus and ventrolateral periaqueductal gray during electroacupuncture: involvement of VGLUT3 NMDA receptors in GABAergic synapses during postnatal development.The glutamatergic neurons in the spinal cord of the sea lamprey: an in situ hybridization and immunohistochemical study.NMDAR-Mediated Calcium Transients Elicited by Glutamate Co-Release at Developing Inhibitory Synapses.Genetic controls balancing excitatory and inhibitory synaptogenesis in neurodevelopmental disorder models.Effects of diazepam on glutamatergic synaptic transmission in the hippocampal CA1 area of rats with traumatic brain injury.Why are neurotransmitters neurotoxic? An evolutionary perspective NMDA receptors in hippocampal GABAergic synapses and their role in nitric oxide signaling Maturation of calcium-dependent GABA, glycine, and glutamate release in the glycinergic MNTB-LSO pathway Emerging themes in GABAergic synapse development Paired recordings from distant inhibitory neuron pairs by a sequential scanning approach Olfactory habituation: fresh insights from flies.Mapping Auditory Synaptic Circuits with Photostimulation of Caged Glutamate.Cannabinoid receptor expression at the MNTB-LSO synapse in developing rats.

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Glutamate co-release at GABA/glycinergic synapses is crucial for the refinement of an inhibitory map.

http://www.wikidata.org/entity/Q30493499

description

2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Glutamate co-release at GABA/g ...... finement of an inhibitory map.

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Glutamate co-release at GABA/g ...... finement of an inhibitory map.

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type

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Glutamate co-release at GABA/g ...... finement of an inhibitory map.

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Glutamate co-release at GABA/g ...... finement of an inhibitory map.

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Glutamate co-release at GABA/g ...... finement of an inhibitory map.

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Glutamate co-release at GABA/g ...... finement of an inhibitory map.

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Nature Neuroscience

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Glutamate co-release at GABA/g ...... finement of an inhibitory map.

@en

P2093

Jessica A Garver

http://www.w3.org/2001/XMLSchema#string

Jihyun Noh

http://www.w3.org/2001/XMLSchema#string

Karl Kandler

http://www.w3.org/2001/XMLSchema#string

Rebecca P Seal

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Robert H Edwards

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P2860

Impairment of SLC17A8 encoding vesicular glutamate transporter-3, VGLUT3, underlies nonsyndromic deafness DFNA25 and inner hair cell dysfunction in null mice.

Sensorineural deafness and seizures in mice lacking vesicular glutamate transporter 3.

The identification of vesicular glutamate transporter 3 suggests novel modes of signaling by glutamate

Otoferlin, defective in a human deafness form, is essential for exocytosis at the auditory ribbon synapse

A forward genetics screen in mice identifies recessive deafness traits and reveals that pejvakin is essential for outer hair cell function

Expression of the vesicular glutamate transporters during development indicates the widespread corelease of multiple neurotransmitters

Calcium- and otoferlin-dependent exocytosis by immature outer hair cells

The self-tuning neuron: synaptic scaling of excitatory synapses

The ontogeny of inhibition and excitation in the gerbil lateral superior olive.

A missense mutation in the conserved C2B domain of otoferlin causes deafness in a new mouse model of DFNB9

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10.1038/NN.2478

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2

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13

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41041505

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1052237588

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20081852

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2832847

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