Enhanced synaptic connectivity and epilepsy in C1q knockout mice. - Wikidata - awgldk - TriplyDB

Enhanced synaptic connectivity and epilepsy in C1q knockout mice.

Enhanced synaptic connectivity and epilepsy in C1q knockout mice.

http://www.wikidata.org/entity/Q30494456

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A New Outlook on Mental Illnesses: Glial Involvement Beyond the Glue Complement activation in the context of stem cells and tissue repair Complement in the Homeostatic and Ischemic Brain Emerging and Novel Functions of Complement Protein C1q The choreography of neuroinflammation in Huntington's disease Antimicrobial peptides and complement in neonatal hypoxia-ischemia induced brain damage From development to dysfunction: microglia and the complement cascade in CNS homeostasis Glial cells in (patho)physiology Do Microglia Default on Network Maintenance in Alzheimer's Disease?The Indispensable Roles of Microglia and Astrocytes during Brain Development LTD-like molecular pathways in developmental synaptic pruning Phagocytosis of apoptotic cells in homeostasis Autoimmune diseases, gastrointestinal disorders and the microbiome in schizophrenia: more than a gut feeling Microglia sculpt postnatal neural circuits in an activity and complement-dependent manner Microglial Function across the Spectrum of Age and Gender Function and Dysfunction of Microglia during Brain Development: Consequences for Synapses and Neural Circuits Excitatory and inhibitory synaptic connectivity to layer V fast-spiking interneurons in the freeze lesion model of cortical microgyria.A lack of immune system genes causes loss in high frequency hearing but does not disrupt cochlear synapse maturation in mice.C1 Complex: An Adaptable Proteolytic Module for Complement and Non-Complement Functions A complement-microglial axis drives synapse loss during virus-induced memory impairment.Fosb gene products contribute to excitotoxic microglial activation by regulating the expression of complement C5a receptors in microglia Sexually divergent induction of microglial-associated neuroinflammation with hippocampal aging.The intersection of amyloid beta and tau at synapses in Alzheimer's disease The role of the immune system in central nervous system plasticity after acute injury.Nuclear calcium signaling in spinal neurons drives a genomic program required for persistent inflammatory pain Complement in animal development: unexpected roles of a highly conserved pathway.DNA methylation analysis of the autistic brain reveals multiple dysregulated biological pathways.Versatility of the complement system in neuroinflammation, neurodegeneration and brain homeostasis Transcriptomic profiling of human peritumoral neocortex tissues revealed genes possibly involved in tumor-induced epilepsy Features of microglia and neuroinflammation relevant to environmental exposure and neurotoxicity.Complement protein C1q modulates neurite outgrowth in vitro and spinal cord axon regeneration in vivo.Experimental Malaria in Pregnancy Induces Neurocognitive Injury in Uninfected Offspring via a C5a-C5a Receptor Dependent Pathway.Interaction of HmC1q with leech microglial cells: involvement of C1qBP-related molecule in the induction of cell chemotaxis.Microglia in the developing brain: a potential target with lifetime effects.LSPS/Optogenetics to Improve Synaptic Connectivity Mapping: Unmasking the Role of Basket Cell-Mediated Feedforward Inhibition.Complement Activation in Placental Malaria.Regulation of microglia by ionotropic glutamatergic and GABAergic neurotransmission Targeting glia cells: novel perspectives for the treatment of neuropsychiatric diseases.Fundamental role of C1q in autoimmunity and inflammation Microglia during development and aging.

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P2860

Enhanced synaptic connectivity and epilepsy in C1q knockout mice.

http://www.wikidata.org/entity/Q30494456

description

2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Enhanced synaptic connectivity and epilepsy in C1q knockout mice.

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Enhanced synaptic connectivity and epilepsy in C1q knockout mice.

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type

label

Enhanced synaptic connectivity and epilepsy in C1q knockout mice.

@ast

Enhanced synaptic connectivity and epilepsy in C1q knockout mice.

@en

prefLabel

Enhanced synaptic connectivity and epilepsy in C1q knockout mice.

@ast

Enhanced synaptic connectivity and epilepsy in C1q knockout mice.

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Proceedings of the National Academy of Sciences of the United States of America

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Enhanced synaptic connectivity and epilepsy in C1q knockout mice.

@en

P2093

Alexei Pesic

http://www.w3.org/2001/XMLSchema#string

Ben Barres

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Beth Stevens

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David A Prince

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Isabel Parada

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Xiaoming Jin

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Yunxiang Chu

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P2860

Postnatal development of the visual cortex and the influence of environment

Abnormal dendritic spines in fragile X knockout mice: maturation and pruning deficits

Functional requirement for class I MHC in CNS development and plasticity

Mutations in the LGI1/Epitempin gene on 10q24 cause autosomal dominant lateral temporal epilepsy

The classical complement cascade mediates CNS synapse elimination

Evidence of functional mossy fiber sprouting in hippocampal formation of kainic acid-treated rats

Inherited epilepsy: spike-wave and focal motor seizures in the mutant mouse tottering

Increased expression of brain-derived neurotrophic factor induces formation of basal dendrites and axonal branching in dentate granule cells in hippocampal explant cultures

Selective expression of clusterin (SGP-2) and complement C1qB and C4 during responses to neurotoxins in vivo and in vitro

Enhanced excitatory synaptic connectivity in layer v pyramidal neurons of chronically injured epileptogenic neocortex in rats.

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