Phosphatidylserine receptor Tim-4 is essential for the maintenance of the homeostatic state of resident peritoneal macrophages - Wikidata - awgldk - TriplyDB

Phosphatidylserine receptor Tim-4 is essential for the maintenance of the homeostatic state of resident peritoneal macrophages

Phosphatidylserine receptor Tim-4 is essential for the maintenance of the homeostatic state of resident peritoneal macrophages

http://www.wikidata.org/entity/Q30494992

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The TIM and TAM families of phosphatidylserine receptors mediate dengue virus entry.Blocking monoclonal antibodies of TIM proteins as orchestrators of anti-tumor immune response Phosphatidylserine receptors: enhancers of enveloped virus entry and infection Phagocytosis of apoptotic cells in homeostasis Clearance of Dying Cells by Phagocytes: Mechanisms and Implications for Disease Pathogenesis.The phosphatidylserine receptor TIM4 utilizes integrins as coreceptors to effect phagocytosis.Beginnings of a good apoptotic meal: the find-me and eat-me signaling pathways Find-me and eat-me signals in apoptotic cell clearance: progress and conundrums.Engulfment signals and the phagocytic machinery for apoptotic cell clearance.p85α recruitment by the CD300f phosphatidylserine receptor mediates apoptotic cell clearance required for autoimmunity suppression Control of signaling-mediated clearance of apoptotic cells by the tumor suppressor p53 Conjugated bilirubin triggers anemia by inducing erythrocyte death.T-cell immunoglobulin and mucin domain 4 (TIM-4) signaling in innate immune-mediated liver ischemia-reperfusion injury.Tim-4 Inhibits NO Generation by Murine Macrophages.Animal Models of Interferon Signature Positive Lupus.Diurnal, localized exposure of phosphatidylserine by rod outer segment tips in wild-type but not Itgb5-/- or Mfge8-/- mouse retina.P2X7 receptor-mediated scavenger activity of mononuclear phagocytes toward non-opsonized particles and apoptotic cells is inhibited by serum glycoproteins but remains active in cerebrospinal fluid.Activation-Induced TIM-4 Expression Identifies Differential Responsiveness of Intestinal CD103+ CD11b+ Dendritic Cells to a Mucosal Adjuvant.Distinct Trafficking of Cell Surface and Endosomal TIM-1 to the Immune Synapse.TIM-4 promotes the growth of non-small-cell lung cancer in a RGD motif-dependent manner.Long-lived self-renewing bone marrow-derived macrophages displace embryo-derived cells to inhabit adult serous cavities The scavenger receptor SCARF1 mediates the clearance of apoptotic cells and prevents autoimmunity.Interruption of dendritic cell-mediated TIM-4 signaling induces regulatory T cells and promotes skin allograft survival.Identification of a tissue-specific, C/EBPβ-dependent pathway of differentiation for murine peritoneal macrophages.The role of T cell immunoglobulin mucin domains 1 and 4 in a herpes simplex virus-induced Behçet's disease mouse model.Innate immune functions of macrophage subpopulations in the spleen.Kidney injury molecule-1: more than just an injury marker of tubular epithelial cells?O death where is thy sting? Immunologic tolerance to apoptotic self.The impact of the TIM gene family on tumor immunity and immunosuppression.Novel insights into Tim-4 function in autoimmune diseases.Tim-4 protects mice against lipopolysaccharide-induced endotoxic shock by suppressing the NF-κB signaling pathway.Maxed out macs: physiologic cell clearance as a function of macrophage phagocytic capacity.Efferocytosis Signaling in the Regulation of Macrophage Inflammatory Responses.F4/80 as a Major Macrophage Marker: The Case of the Peritoneum and Spleen.Two-step engulfment of apoptotic cells.Role of TIM-4 in innate or adaptive immune response.The transcriptional signature of human ovarian carcinoma macrophages is associated with extracellular matrix reorganization.Tim-4 inhibition of T-cell activation and T helper type 17 differentiation requires both the immunoglobulin V and mucin domains and occurs via the mitogen-activated protein kinase pathway.Phagocytosis imprints heterogeneity in tissue-resident macrophages.Tissue-Resident Macrophages Are Locally Programmed for Silent Clearance of Apoptotic Cells.

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Phosphatidylserine receptor Tim-4 is essential for the maintenance of the homeostatic state of resident peritoneal macrophages

http://www.wikidata.org/entity/Q30494992

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Phosphatidylserine receptor Ti ...... esident peritoneal macrophages

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Phosphatidylserine receptor Ti ...... esident peritoneal macrophages

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type

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Phosphatidylserine receptor Ti ...... esident peritoneal macrophages

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Proceedings of the National Academy of Sciences of the United States of America

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Phosphatidylserine receptor Ti ...... esident peritoneal macrophages

@en

P2093

Christine Tan

http://www.w3.org/2001/XMLSchema#string

Jo-Anne Hongo

http://www.w3.org/2001/XMLSchema#string

Kit Wong

http://www.w3.org/2001/XMLSchema#string

Patricia A Valdez

http://www.w3.org/2001/XMLSchema#string

Sherry Yeh

http://www.w3.org/2001/XMLSchema#string

P2860

CRIg: a macrophage complement receptor required for phagocytosis of circulating pathogens

TIM-1 and TIM-4 glycoproteins bind phosphatidylserine and mediate uptake of apoptotic cells.

Structures of T Cell Immunoglobulin Mucin Protein 4 Show a Metal-Ion-Dependent Ligand Binding Site where Phosphatidylserine Binds

Identification of Tapr (an airway hyperreactivity regulatory locus) and the linked Tim gene family

Identification of a factor that links apoptotic cells to phagocytes

Kidney injury molecule-1 is a phosphatidylserine receptor that confers a phagocytic phenotype on epithelial cells

A receptor for phosphatidylserine-specific clearance of apoptotic cells

Interleukin-22, a T(H)17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis

BAI1 is an engulfment receptor for apoptotic cells upstream of the ELMO/Dock180/Rac module.

Engulfment of apoptotic cells: signals for a good meal.

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