Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.
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Innate immunity in the central nervous systemChemokine receptor CXCR2: physiology regulator and neuroinflammation controller?The inflammasome pyrin contributes to pertussis toxin-induced IL-1β synthesis, neutrophil intravascular crawling and autoimmune encephalomyelitisQuetiapine, an atypical antipsychotic, is protective against autoimmune-mediated demyelination by inhibiting effector T cell proliferationELR(+) chemokine signaling in host defense and disease in a viral model of central nervous system diseaseA regenerative approach to the treatment of multiple sclerosis.Promoting remyelination: utilizing a viral model of demyelination to assess cell-based therapiesHydroxycarboxylic acid receptor 2 mediates dimethyl fumarate's protective effect in EAE.Central nervous system pathology progresses independently of KC and CXCR2 in globoid-cell leukodystrophyDesign, synthesis, and evaluation of coumarin-based molecular probes for imaging of myelination.Mediators of oligodendrocyte differentiation during remyelinationAcute skeletal muscle injury: CCL2 expression by both monocytes and injured muscle is required for repair.Chemokines, macrophage inflammatory protein-2 and stromal cell-derived factor-1α, suppress amyloid β-induced neurotoxicity.18β-glycyrrhetinic acid suppresses experimental autoimmune encephalomyelitis through inhibition of microglia activation and promotion of remyelination.Subcutaneous Transplantation of Neural Precursor Cells in Experimental Autoimmune Encephalomyelitis Reduces Chemotactic Signals in the Central Nervous SystemSpatiotemporal ablation of CXCR2 on oligodendrocyte lineage cells: Role in myelin repair.Astrocyte TNFR2 is required for CXCL12-mediated regulation of oligodendrocyte progenitor proliferation and differentiation within the adult CNS.CXCL5 signaling is a shared pathway of neuroinflammation and blood-brain barrier injury contributing to white matter injury in the immature brain.The chemokine receptor CXCR2 and coronavirus-induced neurologic disease.Accelerated and enhanced effect of CCR5-transduced bone marrow neural stem cells on autoimmune encephalomyelitis.CCR5 knockout suppresses experimental autoimmune encephalomyelitis in C57BL/6 mice.Functional defect of peripheral neutrophils in mice with induced deletion of CXCR2Mesenchymal Stem Cells Reversed Morphine Tolerance and Opioid-induced Hyperalgesia.CXCR4 signaling regulates remyelination by endogenous oligodendrocyte progenitor cells in a viral model of demyelination.International Union of Basic and Clinical Pharmacology. [corrected]. LXXXIX. Update on the extended family of chemokine receptors and introducing a new nomenclature for atypical chemokine receptors.Interactions between neutrophils, Th17 cells, and chemokines during the initiation of experimental model of multiple sclerosis.The complex world of oligodendroglial differentiation inhibitors.New insights into ADAMs regulation of the GRO-α/CXCR2 system: focus on Sjögren's syndrome.Attempts to Overcome Remyelination Failure: Toward Opening New Therapeutic Avenues for Multiple Sclerosis.Induction of Experimental Autoimmune Encephalomyelitis in Mice and Evaluation of the Disease-dependent Distribution of Immune Cells in Various Tissues.Discovery of CNS Penetrant CXCR2 Antagonists for the Potential Treatment of CNS Demyelinating Disorders.A mouse model for testing remyelinating therapies.Exacerbation of experimental autoimmune encephalomyelitis in ceramide synthase 6 knockout mice is associated with enhanced activation/migration of neutrophils.CXCL1 can be regulated by IL-6 and promotes granulocyte adhesion to brain capillaries during bacterial toxin exposure and encephalomyelitis.Interleukin-36γ is expressed by neutrophils and can activate microglia, but has no role in experimental autoimmune encephalomyelitis.Polyomavirus JC infection inhibits differentiation of oligodendrocyte progenitor cells.Induced CNS expression of CXCL1 augments neurologic disease in a murine model of multiple sclerosis via enhanced neutrophil recruitment.
P2860
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P2860
Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.
@ast
Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.
@en
type
label
Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.
@ast
Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.
@en
prefLabel
Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.
@ast
Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.
@en
P2093
P2860
P1476
Myelin repair is accelerated by inactivating CXCR2 on nonhematopoietic cells.
@en
P2093
Karen Choi
LiPing Liu
Lindsey Darnall
Richard M Ransohoff
Taofang Hu
Thomas E Lane
P2860
P304
P356
10.1523/JNEUROSCI.1238-10.2010
P407
P577
2010-07-01T00:00:00Z