Mechanisms involved in systemic nicotine-induced glutamatergic synaptic plasticity on dopamine neurons in the ventral tegmental area.
about
The "stop" and "go" of nicotine dependence: role of GABA and glutamateReview of pharmacological treatment in mood disorders and future directions for drug development.Alcohol and nicotine interactions: pre-clinical models of dependenceMechanistic insights into nicotine withdrawal.Dopaminergic and cholinergic learning mechanisms in nicotine addictionDrug-driven AMPA receptor redistribution mimicked by selective dopamine neuron stimulationMechanisms of metabonomic for a gateway drug: nicotine priming enhances behavioral response to cocaine with modification in energy metabolism and neurotransmitter levelNicotinic modulation of hippocampal cell signaling and associated effects on learning and memoryNicotine receptors mediating sensorimotor gating and its enhancement by systemic nicotine.Decreased sensitivity of NMDA receptors on dopaminergic neurons from the posterior ventral tegmental area following chronic nondependent alcohol consumptionDopamine D₂ and acetylcholine α7 nicotinic receptors have subcellular distributions favoring mediation of convergent signaling in the mouse ventral tegmental area.Reward, addiction, withdrawal to nicotine.α7 nicotinic acetylcholine receptor-mediated neuroprotection against dopaminergic neuron loss in an MPTP mouse model via inhibition of astrocyte activation.Potential substrates for nicotine and alcohol interactions: a focus on the mesocorticolimbic dopamine system.Cannabinoid CB2 receptors modulate midbrain dopamine neuronal activity and dopamine-related behavior in miceEndogenous cholinergic inputs and local circuit mechanisms govern the phasic mesolimbic dopamine response to nicotine.Short- and long-lasting consequences of in vivo nicotine treatment on hippocampal excitability.Nicotine and ethanol cooperate to enhance ventral tegmental area AMPA receptor function via α6-containing nicotinic receptorsNicotine potentiation of excitatory inputs to ventral tegmental area dopamine neurons.Age-related changes in nicotine response of cholinergic and non-cholinergic laterodorsal tegmental neurons: implications for the heightened adolescent susceptibility to nicotine addictionRecent advances in understanding nicotinic receptor signaling mechanisms that regulate drug self-administration behavior.Presynaptic ionotropic receptors controlling and modulating the rules for spike timing-dependent plasticity.Nicotine excites hypothalamic arcuate anorexigenic proopiomelanocortin neurons and orexigenic neuropeptide Y neurons: similarities and differences.α6-Containing nicotinic acetylcholine receptors in midbrain dopamine neurons are poised to govern dopamine-mediated behaviors and synaptic plasticityNeuronal nicotinic acetylcholine receptors: neuroplastic changes underlying alcohol and nicotine addictionsEffects of nicotinic acetylcholine receptor agonists in assays of acute pain-stimulated and pain-depressed behaviors in rats.Diacylglycerol lipase disinhibits VTA dopamine neurons during chronic nicotine exposure.Nicotine decreases ethanol-induced dopamine signaling and increases self-administration via stress hormones.Nicotine aversion: Neurobiological mechanisms and relevance to tobacco dependence vulnerability.α4α6β2* nicotinic acetylcholine receptor activation on ventral tegmental area dopamine neurons is sufficient to stimulate a depolarizing conductance and enhance surface AMPA receptor functionNeuroplasticity in addiction: cellular and transcriptional perspectives.Possible contributions of a novel form of synaptic plasticity in Aplysia to reward, memory, and their dysfunctions in mammalian brain.Altered nicotine reward-associated behavior following α4 nAChR subunit deletion in ventral midbrain.Complexity of VTA DA neural activities in response to PFC transection in nicotine treated rats.Modulation of AMPA receptor mediated current by nicotinic acetylcholine receptor in layer I neurons of rat prefrontal cortex.Dl-3n-butylphthalide reduces epileptiform activity through GluA2-lacking calcium-permeable AMPARs in epilepsy models.Nicotinic receptors mediate stress-nicotine detrimental interplay via dopamine cells' activity.Modulation of dopamine release in the striatum by physiologically relevant levels of nicotine.Nicotine-induced anxiogenic-like behaviours of rats in the elevated plus-maze: possible role of NMDA receptors of the central amygdala.Electrophysiological phenotypes of MeCP2 A140V mutant mouse model.
P2860
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P2860
Mechanisms involved in systemic nicotine-induced glutamatergic synaptic plasticity on dopamine neurons in the ventral tegmental area.
description
2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Mechanisms involved in systemi ...... in the ventral tegmental area.
@ast
Mechanisms involved in systemi ...... in the ventral tegmental area.
@en
type
label
Mechanisms involved in systemi ...... in the ventral tegmental area.
@ast
Mechanisms involved in systemi ...... in the ventral tegmental area.
@en
prefLabel
Mechanisms involved in systemi ...... in the ventral tegmental area.
@ast
Mechanisms involved in systemi ...... in the ventral tegmental area.
@en
P2093
P2860
P1476
Mechanisms involved in systemi ...... in the ventral tegmental area.
@en
P2093
P2860
P304
13814-13825
P356
10.1523/JNEUROSCI.1943-10.2010
P407
P577
2010-10-01T00:00:00Z