Transsynaptic progression of amyloid-β-induced neuronal dysfunction within the entorhinal-hippocampal network. - Wikidata - awgldk - TriplyDB

Transsynaptic progression of amyloid-β-induced neuronal dysfunction within the entorhinal-hippocampal network.

Transsynaptic progression of amyloid-β-induced neuronal dysfunction within the entorhinal-hippocampal network.

http://www.wikidata.org/entity/Q30498362

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Inhibitory interneuron deficit links altered network activity and cognitive dysfunction in Alzheimer model.Levetiracetam suppresses neuronal network dysfunction and reverses synaptic and cognitive deficits in an Alzheimer's disease model.At the interface of sensory and motor dysfunctions and Alzheimer's disease.Neuropathological alterations in Alzheimer disease Amyloid β precursor protein as a molecular target for amyloid β--induced neuronal degeneration in Alzheimer's disease Cortical odor processing in health and disease Entorhinal cortical defects in Tg2576 mice are present as early as 2-4 months of age.Memory loss in Alzheimer's disease Spreading of pathology in neurodegenerative diseases: a focus on human studies Alzheimer's disease and prion protein Mitochondrial dysfunction in neurodegenerative diseases Chronic Anatabine Treatment Reduces Alzheimer's Disease (AD)-Like Pathology and Improves Socio-Behavioral Deficits in a Transgenic Mouse Model of AD Propagation of tau pathology in a model of early Alzheimer's disease Alzheimer disease: a tale of two prions Pyroglutamate amyloid β (Aβ) aggravates behavioral deficits in transgenic amyloid mouse model for Alzheimer disease.Central CRF system perturbation in an Alzheimer's disease knockin mouse model.Physiologic brain activity causes DNA double-strand breaks in neurons, with exacerbation by amyloid-β ProNGF\NGF imbalance triggers learning and memory deficits, neurodegeneration and spontaneous epileptic-like discharges in transgenic mice Origins of Alzheimer's disease: reconciling cerebrospinal fluid biomarker and neuropathology data regarding the temporal sequence of amyloid-beta and tau involvement.Altered memory-related functional connectivity of the anterior and posterior hippocampus in older adults at increased genetic risk for Alzheimer's disease.Neurobiology of Alzheimer's Disease: Integrated Molecular, Physiological, Anatomical, Biomarker, and Cognitive Dimensions PAK inactivation impairs social recognition in 3xTg-AD Mice without increasing brain deposition of tau and Aβ Translocator Protein-18 kDa (TSPO) Positron Emission Tomography (PET) Imaging and Its Clinical Impact in Neurodegenerative Diseases Axonal BACE1 dynamics and targeting in hippocampal neurons: a role for Rab11 GTPase Ligand-based and structure-based investigation for Alzheimer's disease from traditional chinese medicine.Molecular drivers and cortical spread of lateral entorhinal cortex dysfunction in preclinical Alzheimer's disease Axonal degeneration in an Alzheimer mouse model is PS1 gene dose dependent and linked to intraneuronal Aβ accumulation.The intersection of amyloid beta and tau at synapses in Alzheimer's disease Notoginsenoside R1 increases neuronal excitability and ameliorates synaptic and memory dysfunction following amyloid elevation.Sleep is related to neuron numbers in the ventrolateral preoptic/intermediate nucleus in older adults with and without Alzheimer's disease.Excitability and synaptic alterations in the cerebellum of APP/PS1 mice Life extension factor klotho enhances cognition.p38 MAP kinase-mediated NMDA receptor-dependent suppression of hippocampal hypersynchronicity in a mouse model of Alzheimer's disease Human P301L-mutant tau expression in mouse entorhinal-hippocampal network causes tau aggregation and presynaptic pathology but no cognitive deficits Life extension factor klotho prevents mortality and enhances cognition in hAPP transgenic mice.Oligomeric Aβ-induced synaptic dysfunction in Alzheimer's disease.Gastrodin suppresses the amyloid β-induced increase of spontaneous discharge in the entorhinal cortex of rats.The sleep-wake cycle and Alzheimer's disease: what do we know?Cognitive and cerebrovascular improvements following kinin B1 receptor blockade in Alzheimer's disease mice.Inhibitory neuron and hippocampal circuit dysfunction in an aged mouse model of Alzheimer's disease.

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P2860

Transsynaptic progression of amyloid-β-induced neuronal dysfunction within the entorhinal-hippocampal network.

http://www.wikidata.org/entity/Q30498362

description

2010 nî lūn-bûn

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2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Transsynaptic progression of a ...... ntorhinal-hippocampal network.

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J.NEURON.2010.10.020

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P1476

Transsynaptic progression of a ...... ntorhinal-hippocampal network.

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Brian Halabisky

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Daniel Kim

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Gui-Qiu Yu

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Iris Lo

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Jorge J Palop

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Julie A Harris

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Kaitlyn Ho

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Lennart Mucke

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Myo T Thwin

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Nino Devidze

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P2860

Persistent amyloidosis following suppression of Abeta production in a transgenic model of Alzheimer disease

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

APP binds DR6 to trigger axon pruning and neuron death via distinct caspases

Alzheimer amyloid protein precursor in the rat hippocampus: transport and processing through the perforant path

Amyloid-beta-induced neuronal dysfunction in Alzheimer's disease: from synapses toward neural networks

Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

Soluble oligomers of the amyloid beta-protein impair synaptic plasticity and behavior

Transmission and spreading of tauopathy in transgenic mouse brain

Neuropathological stageing of Alzheimer-related changes

Involvement of caspases in proteolytic cleavage of Alzheimer's amyloid-beta precursor protein and amyloidogenic A beta peptide formation

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428-441

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scholarly article

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10.1016/J.NEURON.2010.10.020

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3

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68

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Eliezer Masliah

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2010-11-01T00:00:00Z

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47644142

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21040845

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3050043

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