Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells. - Wikidata - awgldk - TriplyDB

Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells.

Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells.

http://www.wikidata.org/entity/Q30500386

about

The "virgin birth", polyploidy, and the origin of cancer DNA damage causes TP53-dependent coupling of self-renewal and senescence pathways in embryonal carcinoma cells Three steps to the immortality of cancer cells: senescence, polyploidy and self-renewal.De-regulated expression of the BRG1 chromatin remodeling factor in bone marrow mesenchymal stromal cells induces senescence associated with the silencing of NANOG and changes in the levels of chromatin proteins A multi-stage process including transient polyploidization and EMT precedes the emergence of chemoresistent ovarian carcinoma cells with a dedifferentiated and pro-inflammatory secretory phenotype Low-dose etoposide-treatment induces endoreplication and cell death accompanied by cytoskeletal alterations in A549 cells: Does the response involve senescence? The possible role of vimentin.Role of stress-activated OCT4A in the cell fate decisions of embryonal carcinoma cells treated with etoposide.Disentangling the aneuploidy and senescence paradoxes: a study of triploid breast cancers non-responsive to neoadjuvant therapy.

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P2860

Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells.

http://www.wikidata.org/entity/Q30500386

description

2011 nî lūn-bûn

@nan

2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի մայիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

@zh-hk

2011年論文

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2011年論文

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2011年论文

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name

Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells.

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Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells.

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type

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Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells.

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Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells.

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Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells.

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Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells.

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Journal of Aging Research

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Self-Renewal Signalling in Presenescent Tetraploid IMR90 Cells.

@en

P2093

Anda Huna

http://www.w3.org/2001/XMLSchema#string

Elina Jascenko

http://www.w3.org/2001/XMLSchema#string

Gunars Duburs

http://www.w3.org/2001/XMLSchema#string

Inna Inashkina

http://www.w3.org/2001/XMLSchema#string

Jekaterina Erenpreisa

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Kristine Salmina

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P2860

Premature senescence involving p53 and p16 is activated in response to constitutive MEK/MAPK mitogenic signaling

PML regulates p53 stability by sequestering Mdm2 to the nucleolus

Endopolyploidy in irradiated p53-deficient tumour cell lines: persistence of cell division activity in giant cells expressing Aurora-B kinase

A nucleolar mechanism controlling cell proliferation in stem cells and cancer cells

From polyploidy to aneuploidy, genome instability and cancer

Characterization of the cyclin-dependent kinase inhibitory domain of the INK4 family as a model for a synthetic tumour suppressor molecule

DNA polymerase alpha inhibition by aphidicolin induces gaps and breaks at common fragile sites in human chromosomes

Identification of aneuploidy-tolerating mutations

p53 induces differentiation of mouse embryonic stem cells by suppressing Nanog expression

The Grb2/Mek pathway represses Nanog in murine embryonic stem cells

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103253

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scholarly article

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10.4061/2011/103253

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2011

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2011-05-11T00:00:00Z

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