c-Jun in Schwann cells promotes axonal regeneration and motoneuron survival via paracrine signaling. - Wikidata - awgldk - TriplyDB

c-Jun in Schwann cells promotes axonal regeneration and motoneuron survival via paracrine signaling.

c-Jun in Schwann cells promotes axonal regeneration and motoneuron survival via paracrine signaling.

http://www.wikidata.org/entity/Q30519558

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Physiological Importance of Hydrogen Sulfide: Emerging Potent Neuroprotector and Neuromodulator Deletion of Jun proteins in adult oligodendrocytes does not perturb cell survival, or myelin maintenance in vivo Microprocessor complex subunit DiGeorge syndrome critical region gene 8 (Dgcr8) is required for schwann cell myelination and myelin maintenance Role of Schwann cells in the regeneration of penile and peripheral nerves cAMP-responsive element-binding protein (CREB) and cAMP co-regulate activator protein 1 (AP1)-dependent regeneration-associated gene expression and neurite growth M2 muscarinic receptor activation regulates Schwann cell differentiation and myelin organization.cJun promotes CNS axon growth.STAT3 Controls the Long-Term Survival and Phenotype of Repair Schwann Cells during Nerve Regeneration.Time-dependent differential expression of long non-coding RNAs following peripheral nerve injury.Mitogen Activated Protein Kinase Family Proteins and c-jun Signaling in Injury-induced Schwann Cell Plasticity.Astrocyte response to motor neuron injury promotes structural synaptic plasticity via STAT3-regulated TSP-1 expression.Nerve injury induces glial cell line-derived neurotrophic factor (GDNF) expression in Schwann cells through purinergic signaling and the PKC-PKD pathway Gene expression profiling studies in regenerating nerves in a mouse model for CMT1X: uninjured Cx32-knockout peripheral nerves display expression profile of injured wild type nerves.Coordinated cell type-specific epigenetic remodeling in prefrontal cortex begins before birth and continues into early adulthood.Dynamic regulation of Schwann cell enhancers after peripheral nerve injury c-Jun gene-modified Schwann cells: upregulating multiple neurotrophic factors and promoting neurite outgrowth Genetic analysis of genes causing hypertension and stroke in spontaneously hypertensive rats: Gene expression profiles in the kidneys.Temporal Analysis of Gene Expression in the Murine Schwann Cell Lineage and the Acutely Injured Postnatal Nerve.Strategies to promote peripheral nerve regeneration: electrical stimulation and/or exercise.Induction of heat shock protein 70 (Hsp70) prevents neuregulin-induced demyelination by enhancing the proteasomal clearance of c-Jun.Arginine methylation of the c-Jun coactivator RACO-1 is required for c-Jun/AP-1 activation Schwann cell phenotype is regulated by axon modality and central-peripheral location, and persists in vitro.Epigenomic Regulation of Schwann Cell Reprogramming in Peripheral Nerve Injury.Loss of Schwann cell plasticity in chronic inflammatory demyelinating polyneuropathy (CIDP).Signals regulating myelination in peripheral nerves and the Schwann cell response to injury.PI3K-GSK3 signalling regulates mammalian axon regeneration by inducing the expression of Smad1.The Expression implication of GDNF in ventral horn and associated remote cortex in rhesus monkeys with hemisected spinal cord injury Epigenetic profiling reveals a developmental decrease in promoter accessibility during cortical maturation in vivo.Artemin induced functional recovery and reinnervation after partial nerve injury.Delaying histone deacetylase response to injury accelerates conversion into repair Schwann cells and nerve regeneration.Negative regulators of schwann cell differentiation-novel targets for peripheral nerve therapies?Plastic fantastic: Schwann cells and repair of the peripheral nervous system.Concepts for regulation of axon integrity by enwrapping glia.Molecules involved in the crosstalk between immune- and peripheral nerve Schwann cells.Peripheral nerve injury modulates neurotrophin signaling in the peripheral and central nervous system.Repair of the Peripheral Nerve-Remyelination that Works.Schwann Cells: Development and Role in Nerve Repair The repair Schwann cell and its function in regenerating nerves.Gene delivery to rat and human Schwann cells and nerve segments: a comparison of AAV 1-9 and lentiviral vectors.Molecular Mechanisms Involved in Schwann Cell Plasticity

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c-Jun in Schwann cells promotes axonal regeneration and motoneuron survival via paracrine signaling.

http://www.wikidata.org/entity/Q30519558

description

2012 nî lūn-bûn

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2012 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2012 թվականի հուլիսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

c-Jun in Schwann cells promote ...... vival via paracrine signaling.

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c-Jun in Schwann cells promote ...... vival via paracrine signaling.

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Journal of Cell Biology

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c-Jun in Schwann cells promote ...... rvival via paracrine signaling

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Bradley Spencer-Dene

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Clive Da Costa

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Gennadij Raivich

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Kristjan R Jessen

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Mariya Hristova

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Morwena Latouche

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Rhona Mirsky

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Smriti Patodia

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Xavier Fontana

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P2860

Absence of Ret signaling in mice causes progressive and late degeneration of the nigrostriatal system

Artemin, a novel member of the GDNF ligand family, supports peripheral and central neurons and signals through the GFRalpha3-RET receptor complex

Interaction of phosphorylated c-Jun with TCF4 regulates intestinal cancer development

GDNF-induced activation of the ret protein tyrosine kinase is mediated by GDNFR-alpha, a novel receptor for GDNF

Neurotrophin signal transduction in the nervous system.

The GDNF family ligands and receptors - implications for neural development

The GDNF family: signalling, biological functions and therapeutic value

Neurturin and glial cell line-derived neurotrophic factor receptor-beta (GDNFR-beta), novel proteins related to GDNF and GDNFR-alpha with specific cellular patterns of expression suggesting roles in the developing and adult nervous system and in per

GDNF family receptor complexes are emerging drug targets

The jun proto-oncogene is positively autoregulated by its product, Jun/AP-1

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127-141

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scholarly article

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10.1083/JCB.201205025

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1

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198

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2012-07-02T00:00:00Z

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228105677

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22753894

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3392945

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