Cardiomyocyte-specific IκB kinase (IKK)/NF-κB activation induces reversible inflammatory cardiomyopathy and heart failure. - Wikidata - awgldk - TriplyDB

Cardiomyocyte-specific IκB kinase (IKK)/NF-κB activation induces reversible inflammatory cardiomyopathy and heart failure.

Cardiomyocyte-specific IκB kinase (IKK)/NF-κB activation induces reversible inflammatory cardiomyopathy and heart failure.

http://www.wikidata.org/entity/Q30523246

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Repair Injured Heart by Regulating Cardiac Regenerative Signals N-acetylcysteine reduces oxidative stress, nuclear factor‑κB activity and cardiomyocyte apoptosis in heart failure Sustained, neuron-specific IKK/NF-κB activation generates a selective neuroinflammatory response promoting local neurodegeneration with aging.Tumor necrosis factor-α confers cardioprotection through ectopic expression of keratins K8 and K18.Cardiac-specific suppression of NF-κB signaling prevents diabetic cardiomyopathy via inhibition of the renin-angiotensin system.BET bromodomains mediate transcriptional pause release in heart failure Sodium tanshinone IIA silate inhibits oxygen-glucose deprivation/recovery-induced cardiomyocyte apoptosis via suppression of the NF-κB/TNF-α pathway Conditional knockout of prolyl hydroxylase domain protein 2 attenuates high fat-diet-induced cardiac dysfunction in mice.Cardiomyocyte p65 nuclear factor-κB is necessary for compensatory adaptation to pressure overload Dynamic reorganization of the AC16 cardiomyocyte transcriptome in response to TNFα signaling revealed by integrated genomic analyses.Signal regulatory protein-α protects against cardiac hypertrophy via the disruption of toll-like receptor 4 signaling Cardiac-Specific Activation of IKK2 Leads to Defects in Heart Development and Embryonic Lethality.Bone marrow cell characteristics associated with patient profile and cardiac performance outcomes in the LateTIME-Cardiovascular Cell Therapy Research Network (CCTRN) trial Myocardial NF-κB activation is essential for zebrafish heart regeneration.TAK1 Regulates Myocardial Response to Pathological Stress via NFAT, NFκB, and Bnip3 Pathways.The NO/ONOO-cycle as the central cause of heart failure.Identification of Bone Marrow Cell Subpopulations Associated With Improved Functional Outcomes in Patients With Chronic Left Ventricular Dysfunction: An Embedded Cohort Evaluation of the FOCUS-CCTRN Trial Smad Nuclear Interacting Protein 1 Acts as a Protective Regulator of Pressure Overload-Induced Pathological Cardiac Hypertrophy.p53 regulates the cardiac transcriptome.Interferon regulatory factor 7 functions as a novel negative regulator of pathological cardiac hypertrophy Fluid overload and inflammation--a vicious cycle.Do multiple nuclear factor kappa B activation mechanisms explain its varied effects in the heart?TWEAK-Fn14 Cytokine-Receptor Axis: A New Player of Myocardial Remodeling and Cardiac Failure Ubiquitin and ubiquitin-like proteins in cardiac disease and protection.NF-κB activation is cell type-specific in the heart.Signaling Pathways in Cardiac Myocyte Apoptosis.Redirecting cardiac growth mechanisms for therapeutic regeneration.Cardiac-restricted Overexpression of TRAF3 Interacting Protein 2 (TRAF3IP2) Results in Spontaneous Development of Myocardial Hypertrophy, Fibrosis, and Dysfunction.Inhibition of IκB Kinase at 24 Hours After Acute Kidney Injury Improves Recovery of Renal Function and Attenuates Fibrosis Adipocyte-specific IKKβ signaling suppresses adipose tissue inflammation through an IL-13-dependent paracrine feedback pathway.Sang-qi Granula Reduces Blood Pressure and Myocardial Fibrosis by Suppressing Inflammatory Responses Associated with the Peroxisome Proliferator-Activated Receptors and Nuclear Factor κ B Protein in Spontaneously Hypertensive Rats.Neferine ameliorates cardiomyoblast apoptosis induced by doxorubicin: possible role in modulating NADPH oxidase/ROS-mediated NFκB redox signaling cascade.Therapeutic effect of MG-132 on diabetic cardiomyopathy is associated with its suppression of proteasomal activities: roles of Nrf2 and NF-κB.Therapeutic targeting of the oncostatin M receptor-β prevents inflammatory heart failure.BEX1 is an RNA-dependent mediator of cardiomyopathy.The Role of Toll-Like Receptor Signaling in the Progression of Heart Failure.NF-κB-mediated miR-30b regulation in cardiomyocytes cell death by targeting Bcl-2.MCP-1-induced protein attenuates post-infarct cardiac remodeling and dysfunction through mitigating NF-κB activation and suppressing inflammation-associated microRNA expression.Inflammatory Mediators Drive Adverse Right Ventricular Remodeling and Dysfunction and Serve as Potential Biomarkers.Chemogenetic generation of hydrogen peroxide in the heart induces severe cardiac dysfunction

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Cardiomyocyte-specific IκB kinase (IKK)/NF-κB activation induces reversible inflammatory cardiomyopathy and heart failure.

http://www.wikidata.org/entity/Q30523246

description

2012 nî lūn-bûn

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2012 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի հուլիսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Cardiomyocyte-specific IκB kin ...... diomyopathy and heart failure.

@ast

Cardiomyocyte-specific IκB kin ...... diomyopathy and heart failure.

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type

label

Cardiomyocyte-specific IκB kin ...... diomyopathy and heart failure.

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Cardiomyocyte-specific IκB kin ...... diomyopathy and heart failure.

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prefLabel

Cardiomyocyte-specific IκB kin ...... diomyopathy and heart failure.

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Cardiomyocyte-specific IκB kin ...... diomyopathy and heart failure.

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PNAS.1116584109

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Proceedings of the National Academy of Sciences of the United States of America

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Cardiomyocyte-specific IκB kin ...... rdiomyopathy and heart failure

@en

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Astrid Wietelmann

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Cornelia Brunner

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Harald J Maier

http://www.w3.org/2001/XMLSchema#string

Marcus Krüger

http://www.w3.org/2001/XMLSchema#string

Thomas Böttger

http://www.w3.org/2001/XMLSchema#string

Thomas Wirth

http://www.w3.org/2001/XMLSchema#string

Tobias G Schips

http://www.w3.org/2001/XMLSchema#string

P2860

IKK mediates ischemia-induced neuronal death

Dichotomous actions of NF-kappaB signaling pathways in heart.

Interferon-stimulated gene 15 (ISG15) conjugates proteins in dermatomyositis muscle with perifascicular atrophy.

Organ-specific roles for transcription factor NF-kappaB in reovirus-induced apoptosis and disease.

Targeted ablation of IKK2 improves skeletal muscle strength, maintains mass, and promotes regeneration

Ongoing enterovirus-induced myocarditis is associated with persistent heart muscle infection: quantitative analysis of virus replication, tissue damage, and inflammation

Blockade of NF-kappaB using IkappaB alpha dominant-negative mice ameliorates cardiac hypertrophy in myotrophin-overexpressed transgenic mice.

The NF-kappaB family of transcription factors and its regulation.

Role of nuclear factor kappaB in cardiovascular health and disease.

Multiple facets of NF-κB in the heart: to be or not to NF-κB.

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11794-11799

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10.1073/PNAS.1116584109

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3406816

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