Mammalian target of rapamycin complex 1 activation negatively regulates Polo-like kinase 2-mediated homeostatic compensation following neonatal seizures.
about
Cdk5 is a New Rapid Synaptic Homeostasis Regulator Capable of Initiating the Early Alzheimer-Like Pathology.AMPA receptor antagonist NBQX attenuates later-life epileptic seizures and autistic-like social deficits following neonatal seizuresNeonatal seizures alter NMDA glutamate receptor GluN2A and 3A subunit expression and function in hippocampal CA1 neurons.Bortezomib alleviates drug-induced liver injury by regulating CYP2E1 gene transcription.Mechanisms regulating neuronal excitability and seizure development following mTOR pathway hyperactivation.Early-life seizures alter synaptic calcium-permeable AMPA receptor function and plasticity.What goes up must come down: homeostatic synaptic plasticity strategies in neurological disease.Environment-dependent striatal gene expression in the BACHD rat model for Huntington disease.
P2860
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P2860
Mammalian target of rapamycin complex 1 activation negatively regulates Polo-like kinase 2-mediated homeostatic compensation following neonatal seizures.
description
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2013年の論文
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2013年論文
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2013年論文
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name
Mammalian target of rapamycin ...... n following neonatal seizures.
@ast
Mammalian target of rapamycin ...... n following neonatal seizures.
@en
type
label
Mammalian target of rapamycin ...... n following neonatal seizures.
@ast
Mammalian target of rapamycin ...... n following neonatal seizures.
@en
prefLabel
Mammalian target of rapamycin ...... n following neonatal seizures.
@ast
Mammalian target of rapamycin ...... n following neonatal seizures.
@en
P2093
P2860
P356
P1476
Mammalian target of rapamycin ...... on following neonatal seizures
@en
P2093
Bela Kosaras
Frances E Jensen
Hongyu Sun
P2860
P304
P356
10.1073/PNAS.1208010110
P407
P577
2013-03-11T00:00:00Z