Brain amyloid-β oligomers in ageing and Alzheimer's disease.
about
The Role of Amyloid-β Oligomers in Toxicity, Propagation, and ImmunotherapyCalcium-Sensing Receptors of Human Neural Cells Play Crucial Roles in Alzheimer's DiseaseFluid Biomarkers in Clinical Trials of Alzheimer's Disease TherapeuticsEarly failure of the default-mode network and the pathogenesis of Alzheimer's diseaseBreaking the Code of Amyloid-β OligomersOn the Environmental Factors Affecting the Structural and Cytotoxic Properties of IAPP PeptidesIs L-methionine a trigger factor for Alzheimer's-like neurodegeneration?: Changes in Aβ oligomers, tau phosphorylation, synaptic proteins, Wnt signaling and behavioral impairment in wild-type miceTreatment of beta amyloid 1-42 (Aβ(1-42))-induced basal forebrain cholinergic damage by a non-classical estrogen signaling activator in vivoIVIg protects the 3xTg-AD mouse model of Alzheimer’s disease from memory deficit and Aβ pathologyMolecular Structure of Aggregated Amyloid-β: Insights from Solid-State Nuclear Magnetic ResonanceThree dimensions of the amyloid hypothesis: time, space and 'wingmen'APOE-modulated Aβ-induced neuroinflammation in Alzheimer's disease: current landscape, novel data, and future perspectiveAnalyzing dendritic spine pathology in Alzheimer's disease: problems and opportunitiesAlzheimer's therapeutics targeting amyloid beta 1-42 oligomers I: Abeta 42 oligomer binding to specific neuronal receptors is displaced by drug candidates that improve cognitive deficitsAlzheimer's therapeutics targeting amyloid beta 1-42 oligomers II: Sigma-2/PGRMC1 receptors mediate Abeta 42 oligomer binding and synaptotoxicityDBA/2J genetic background exacerbates spontaneous lethal seizures but lessens amyloid deposition in a mouse model of Alzheimer's disease.Plaque-associated lipids in Alzheimer's diseased brain tissue visualized by nonlinear microscopyChronic adiponectin deficiency leads to Alzheimer's disease-like cognitive impairments and pathologies through AMPK inactivation and cerebral insulin resistance in aged mice.Highly potent soluble amyloid-β seeds in human Alzheimer brain but not cerebrospinal fluidPhysiological IgM class catalytic antibodies selective for transthyretin amyloid.The essential role of p53-up-regulated modulator of apoptosis (Puma) and its regulation by FoxO3a transcription factor in β-amyloid-induced neuron death.The Hsp70/Hsp90 Chaperone Machinery in Neurodegenerative DiseasesTat-haFGF14-154 Upregulates ADAM10 to Attenuate the Alzheimer Phenotype of APP/PS1 Mice through the PI3K-CREB-IRE1α/XBP1 Pathway.Building a pipeline to discover and validate novel therapeutic targets and lead compounds for Alzheimer's disease.Amyloid β Protein and Alzheimer's Disease: When Computer Simulations Complement Experimental Studies.Smoking and increased Alzheimer's disease risk: a review of potential mechanismsAlzheimer's disease: relevant molecular and physiopathological events affecting amyloid-β brain balance and the putative role of PPARs.Knock-in of human BACE1 cleaves murine APP and reiterates Alzheimer-like phenotypesDevelopment of a novel cellular model of Alzheimer's disease utilizing neurosphere cultures derived from B6C3-Tg(APPswe,PSEN1dE9)85Dbo/J embryonic mouse brainAmyloid β and Tau Alzheimer's disease related pathology is reduced by Toll-like receptor 9 stimulation.Disruption of amyloid plaques integrity affects the soluble oligomers content from Alzheimer disease brains.Multifunctional Effects of Mangosteen Pericarp on Cognition in C57BL/6J and Triple Transgenic Alzheimer's Mice.Longitudinal testing of hippocampal plasticity reveals the onset and maintenance of endogenous human Aß-induced synaptic dysfunction in individual freely behaving pre-plaque transgenic rats: rapid reversal by anti-Aß agents.Increased Alzheimer's disease-like pathology in the APP/ PS1ΔE9 mouse model lacking Nrf2 through modulation of autophagyThe elusive nature and diagnostics of misfolded Aβ oligomers.Specific amyloid β clearance by a catalytic antibody construct.Characterization of a Novel Mouse Model of Alzheimer's Disease--Amyloid Pathology and Unique β-Amyloid Oligomer Profile.Amyloid polymorphism: structural basis and neurobiological relevance.Soluble amyloid-β oligomers as synaptotoxins leading to cognitive impairment in Alzheimer's diseaseAβ43 is neurotoxic and primes aggregation of Aβ40 in vivo.
P2860
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P2860
Brain amyloid-β oligomers in ageing and Alzheimer's disease.
description
2013 nî lūn-bûn
@nan
2013 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Brain amyloid-β oligomers in ageing and Alzheimer's disease.
@ast
Brain amyloid-β oligomers in ageing and Alzheimer's disease.
@en
type
label
Brain amyloid-β oligomers in ageing and Alzheimer's disease.
@ast
Brain amyloid-β oligomers in ageing and Alzheimer's disease.
@en
prefLabel
Brain amyloid-β oligomers in ageing and Alzheimer's disease.
@ast
Brain amyloid-β oligomers in ageing and Alzheimer's disease.
@en
P2093
P2860
P356
P1433
P1476
Brain amyloid-β oligomers in ageing and Alzheimer's disease.
@en
P2093
Julie A Schneider
Karen H Ashe
Marianne Grant
Mathew A Sherman
Michael Kuskowski
Sylvain E Lesné
P2860
P304
P356
10.1093/BRAIN/AWT062
P407
P577
2013-04-09T00:00:00Z