Disruption of vascular Ca2+-activated chloride currents lowers blood pressure. - Wikidata - awgldk - TriplyDB

Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.

Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.

http://www.wikidata.org/entity/Q30569762

about

The bestrophin- and TMEM16A-associated Ca(2+)- activated Cl(–) channels in vascular smooth muscles Renal autoregulation in health and disease Mechanism of allosteric activation of TMEM16A/ANO1 channels by a commonly used chloride channel blocker Modulating Ca²⁺ signals: a common theme for TMEM16, Ist2, and TMC.Molecular and functional significance of Ca(2+)-activated Cl(-) channels in pulmonary arterial smooth muscle.Effects of new-generation inhibitors of the calcium-activated chloride channel anoctamin 1 on slow waves in the gastrointestinal tract.Synthesis and evaluation of 5,6-disubstituted thiopyrimidine aryl aminothiazoles as inhibitors of the calcium-activated chloride channel TMEM16A/Ano1.TMEM16A is associated with voltage-gated calcium channels in mouse retina and its function is disrupted upon mutation of the auxiliary α2δ4 subunit.The molecular basis of the genesis of basal tone in internal anal sphincter.Local coupling of TRPC6 to ANO1/TMEM16A channels in smooth muscle cells amplifies vasoconstriction in cerebral arteries.Regulation of vascular tone and arterial blood pressure: role of chloride transport in vascular smooth muscle.Negative News: Cl- and HCO3- in the Vascular Wall.Smooth Muscle Ion Channels and Regulation of Vascular Tone in Resistance Arteries and Arterioles.AT1 receptor signaling pathways in the cardiovascular system.Cell-specific impact of nitric oxide-dependent guanylyl cyclase on arteriogenesis and angiogenesis in mice.Contrasting effects of phosphatidylinositol 4,5-bisphosphate on cloned TMEM16A and TMEM16B channels.ANO1 taking center stage: blood pressure regulation in SHRs.Inhibitory role of phosphatidylinositol 4,5-bisphosphate on TMEM16A-encoded calcium-activated chloride channels in rat pulmonary artery.Neurotrophin Signaling Is Required for Glucose-Induced Insulin Secretion.Conditional genetic deletion of Ano1 in interstitial cells of Cajal impairs Ca2+ transients and slow waves in adult mouse small intestine.Nitric oxide-sensitive guanylyl cyclase stimulation improves experimental heart failure with preserved ejection fraction.CLCA2 is a positive regulator of store-operated calcium entry and TMEM16A.Ca-activated Cl currents in the murine vomeronasal organ enhance neuronal spiking but are dispensable for male-male aggression Inositol 1,4,5-Trisphosphate Receptors in Hypertension

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P2860

Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.

http://www.wikidata.org/entity/Q30569762

description

2014 nî lūn-bûn

@nan

2014 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հունվարին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.

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Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.

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type

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Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.

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Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.

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Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.

@ast

Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.

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Journal of Clinical Investigation

P1476

Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.

@en

P2093

Agnieszka E Klementowicz

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Anika Seniuk

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Antje K Huebner

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Björn C Schroeder

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Christian A Hübner

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Christoph Heinze

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Heimo Ehmke

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Helga Vitzthum

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István A Szijártó

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Johanna Schleifenbaum

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P2860

Calcium-dependent phospholipid scrambling by TMEM16F

Mutations in ANO3 cause dominant craniocervical dystonia: ion channel implicated in pathogenesis

Retinovascular physiology and pathophysiology: new experimental approach/new insights

Pericytes: developmental, physiological, and pathological perspectives, problems, and promises

Global burden of hypertension: analysis of worldwide data

TMEM16A/anoctamin 1 protein mediates calcium-activated chloride currents in pulmonary arterial smooth muscle cells

Dominant role of smooth muscle L-type calcium channel Cav1.2 for blood pressure regulation

The transmembrane protein TMEM16A is required for normal development of the murine trachea

TMEM16A confers receptor-activated calcium-dependent chloride conductance

A cre-transgenic mouse strain for the ubiquitous deletion of loxP-flanked gene segments including deletion in germ cells

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675-686

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10.1172/JCI70025

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2

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124

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2014-01-09T00:00:00Z

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24401273

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3904609

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