about
Priming the inflammatory pump of the CNS after traumatic brain injury.Oxidation of KCNB1 Potassium Channels Causes Neurotoxicity and Cognitive Impairment in a Mouse Model of Traumatic Brain Injury.Novel rodent models of penetrating traumatic brain injury.Selective activation of cannabinoid receptor-2 reduces neuroinflammation after traumatic brain injury via alternative macrophage polarization.
P2860
description
2014 nî lūn-bûn
@nan
2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
A novel mouse model of penetrating brain injury
@ast
A novel mouse model of penetrating brain injury
@en
type
label
A novel mouse model of penetrating brain injury
@ast
A novel mouse model of penetrating brain injury
@en
prefLabel
A novel mouse model of penetrating brain injury
@ast
A novel mouse model of penetrating brain injury
@en
P2093
P2860
P356
P1476
A novel mouse model of penetrating brain injury
@en
P2093
Ian D Wing
Ibolja Cernak
Johan Davidsson
Stefan Plantman
P2860
P356
10.3389/FNEUR.2014.00209
P577
2014-10-22T00:00:00Z