Trans-synaptic zinc mobilization improves social interaction in two mouse models of autism through NMDAR activation - Wikidata - awgldk - TriplyDB

Trans-synaptic zinc mobilization improves social interaction in two mouse models of autism through NMDAR activation

Trans-synaptic zinc mobilization improves social interaction in two mouse models of autism through NMDAR activation

http://www.wikidata.org/entity/Q30655543

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Brain-specific transcriptional regulator T-brain-1 controls brain wiring and neuronal activity in autism spectrum disorders Autism phenotypes in ZnT3 null mice: Involvement of zinc dyshomeostasis, MMP-9 activation and BDNF upregulation.Actin-Dependent Alterations of Dendritic Spine Morphology in Shankopathies Advancing the understanding of autism disease mechanisms through genetics SHANK proteins: roles at the synapse and in autism spectrum disorder.Synaptic Actin Dysregulation, a Convergent Mechanism of Mental Disorders?An intra-amygdala circuit specifically regulates social fear learning.Behavioural Phenotypes and Neural Circuit Dysfunctions in Mouse Models of Autism Spectrum Disorder.Extracerebral Dysfunction in Animal Models of Autism Spectrum Disorder.Functional significance of rare neuroligin 1 variants found in autism The role of postsynaptic density proteins in neural degeneration and regeneration.D-Cycloserine Ameliorates Autism-Like Deficits by Removing GluA2-Containing AMPA Receptors in a Valproic Acid-Induced Rat Model.Alleviation of N-Methyl-D-Aspartate Receptor-Dependent Long-Term Depression via Regulation of the Glycogen Synthase Kinase-3β Pathway in the Amygdala of a Valproic Acid-Induced Animal Model of Autism.Dynamical features in fetal and postnatal zinc-copper metabolic cycles predict the emergence of autism spectrum disorder.Prospects of Zinc Supplementation in Autism Spectrum Disorders and Shankopathies Such as Phelan McDermid Syndrome.Silica nanoparticle exposure during the neonatal period impairs hippocampal precursor proliferation and social behavior later in life.Real Time Imaging and Dynamics of Hippocampal Zn2+ under Epileptic Condition Using a Ratiometric Fluorescent Probe.Defective Synapse Maturation and Enhanced Synaptic Plasticity in Shank2 Δex7–/– Mice.Distinct Phenotypes of Mouse Models Reflect Neuropsychiatric Spectrum Disorders of Human Patients With Variants Dietary Zinc Supplementation Prevents Autism Related Behaviors and Striatal Synaptic Dysfunction in Exon 13-16 Mutant Mice Mutant Mice Display Hyperactivity Insensitive to Methylphenidate and Reduced Flexibility in Social Motivation, but Normal Social Recognition

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P2860

Trans-synaptic zinc mobilization improves social interaction in two mouse models of autism through NMDAR activation

http://www.wikidata.org/entity/Q30655543

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2015 nî lūn-bûn

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2015 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2015 թվականի մայիսին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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Trans-synaptic zinc mobilizati ...... utism through NMDAR activation

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Trans-synaptic zinc mobilizati ...... utism through NMDAR activation

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Trans-synaptic zinc mobilizati ...... utism through NMDAR activation

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Trans-synaptic zinc mobilizati ...... utism through NMDAR activation

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Trans-synaptic zinc mobilizati ...... utism through NMDAR activation

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Trans-synaptic zinc mobilizati ...... utism through NMDAR activation

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Nature Communications

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Trans-synaptic zinc mobilizati ...... utism through NMDAR activation

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Changuk Chung

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Eun-Jae Lee

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Eunjoon Kim

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Hyejin Lee

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Jae-Young Koh

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Kyungdeok Kim

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Tzyy-Nan Huang

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Wangyong Shin

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P2860

Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice

Haploinsufficiency of the autism-associated Shank3 gene leads to deficits in synaptic function, social interaction, and social communication

Neuroligins and neurexins link synaptic function to cognitive disease

Searching for ways out of the autism maze: genetic, epigenetic and environmental clues

Meta-analysis of SHANK Mutations in Autism Spectrum Disorders: a gradient of severity in cognitive impairments

Mutations in the SHANK2 synaptic scaffolding gene in autism spectrum disorder and mental retardation

Importance of Shank3 protein in regulating metabotropic glutamate receptor 5 (mGluR5) expression and signaling at synapses

Mutations in the gene encoding the synaptic scaffolding protein SHANK3 are associated with autism spectrum disorders

Autistic-like social behaviour in Shank2-mutant mice improved by restoring NMDA receptor function

Clioquinol down-regulates mutant huntingtin expression in vitro and mitigates pathology in a Huntington's disease mouse model.

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