High copy number suppression of the meiotic arrest caused by a dmc1 mutation: REC114 imposes an early recombination block and RAD54 promotes a DMC1-independent DSB repair pathway.
about
Frequent and efficient use of the sister chromatid for DNA double-strand break repair during budding yeast meiosisClarifying the mechanics of DNA strand exchange in meiotic recombinationTid1/Rdh54 promotes colocalization of rad51 and dmc1 during meiotic recombinationMnd1/Hop2 facilitates Dmc1-dependent interhomolog crossover formation in meiosis of budding yeast.Csm4-dependent telomere movement on nuclear envelope promotes meiotic recombination.Hed1 regulates Rad51-mediated recombination via a novel mechanism.Role for the silencing protein Dot1 in meiotic checkpoint control.A role for MMS4 in the processing of recombination intermediates during meiosis in Saccharomyces cerevisiaeMek1 kinase activity functions downstream of RED1 in the regulation of meiotic double strand break repair in budding yeast.The Mnd1 protein forms a complex with hop2 to promote homologous chromosome pairing and meiotic double-strand break repair.Meiotic condensin is required for proper chromosome compaction, SC assembly, and resolution of recombination-dependent chromosome linkagesMeiotic crossover control by concerted action of Rad51-Dmc1 in homolog template bias and robust homeostatic regulation.ZIP4H (TEX11) deficiency in the mouse impairs meiotic double strand break repair and the regulation of crossing overRole of RAD52 epistasis group genes in homologous recombination and double-strand break repairA comparative analysis of Dmc1 and Rad51 nucleoprotein filamentsA dominant, recombination-defective allele of Dmc1 causing male-specific sterility.Excess single-stranded DNA inhibits meiotic double-strand break repair.Roles of Hop1 and Mek1 in meiotic chromosome pairing and recombination partner choice in Schizosaccharomyces pombeSpontaneous and double-strand break-induced recombination, and gene conversion tract lengths, are differentially affected by overexpression of wild-type or ATPase-defective yeast Rad54.Mek1 suppression of meiotic double-strand break repair is specific to sister chromatids, chromosome autonomous and independent of Rec8 cohesin complexesPartner choice during meiosis is regulated by Hop1-promoted dimerization of Mek1.Chromosome-wide regulation of meiotic crossover formation in Caenorhabditis elegans requires properly assembled chromosome axes.Analysis of close stable homolog juxtaposition during meiosis in mutants of Saccharomyces cerevisiae.Spo11 and the Formation of DNA Double-Strand Breaks in Meiosis.Genetic evidence that synaptonemal complex axial elements govern recombination pathway choice in micePch2 modulates chromatid partner choice during meiotic double-strand break repair in Saccharomyces cerevisiae.Prelude to a divisionNovel attributes of Hed1 affect dynamics and activity of the Rad51 presynaptic filament during meiotic recombination.Analysis of the Relationships between DNA Double-Strand Breaks, Synaptonemal Complex and Crossovers Using the Atfas1-4 Mutant.ASY1 mediates AtDMC1-dependent interhomolog recombination during meiosis in Arabidopsis.Mek1 kinase is regulated to suppress double-strand break repair between sister chromatids during budding yeast meiosisASY1 coordinates early events in the plant meiotic recombination pathway.Brca2 is involved in meiosis in Arabidopsis thaliana as suggested by its interaction with Dmc1.An essential role of DmRad51/SpnA in DNA repair and meiotic checkpoint control.On the role of AtDMC1, AtRAD51 and its paralogs during Arabidopsis meiosis.A non-sister act: recombination template choice during meiosis.The mitotic DNA damage checkpoint proteins Rad17 and Rad24 are required for repair of double-strand breaks during meiosis in yeast.Down-regulation of Rad51 activity during meiosis in yeast prevents competition with Dmc1 for repair of double-strand breaks.Regulation of meiotic recombination via Mek1-mediated Rad54 phosphorylation.Crossover interference in Saccharomyces cerevisiae requires a TID1/RDH54- and DMC1-dependent pathway.
P2860
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P2860
High copy number suppression of the meiotic arrest caused by a dmc1 mutation: REC114 imposes an early recombination block and RAD54 promotes a DMC1-independent DSB repair pathway.
description
1999 nî lūn-bûn
@nan
1999 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
High copy number suppression o ...... ndependent DSB repair pathway.
@ast
High copy number suppression o ...... ndependent DSB repair pathway.
@en
type
label
High copy number suppression o ...... ndependent DSB repair pathway.
@ast
High copy number suppression o ...... ndependent DSB repair pathway.
@en
prefLabel
High copy number suppression o ...... ndependent DSB repair pathway.
@ast
High copy number suppression o ...... ndependent DSB repair pathway.
@en
P2093
P2860
P1433
P1476
High copy number suppression o ...... independent DSB repair pathway
@en
P2093
P2860
P304
P356
10.1046/J.1365-2443.1999.00273.X
P50
P577
1999-08-01T00:00:00Z