Role of NO and K(+)(ATP) channels in adenosine-induced vasodilation on in vivo canine subendocardial arterioles.
about
Altered mechanism of adenosine-induced coronary arteriolar dilation in early-stage metabolic syndrome.Increased inactivation of nitric oxide is involved in impaired coronary flow reserve in heart failure.Pulsation-induced dilation of subendocardial and subepicardial arterioles: effect on vasodilator sensitivity.Regulation of Coronary Blood Flow.
P2860
Role of NO and K(+)(ATP) channels in adenosine-induced vasodilation on in vivo canine subendocardial arterioles.
description
1999 nî lūn-bûn
@nan
1999 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Role of NO and K(+)(ATP) chann ...... ine subendocardial arterioles.
@ast
Role of NO and K(+)(ATP) chann ...... ine subendocardial arterioles.
@en
type
label
Role of NO and K(+)(ATP) chann ...... ine subendocardial arterioles.
@ast
Role of NO and K(+)(ATP) chann ...... ine subendocardial arterioles.
@en
prefLabel
Role of NO and K(+)(ATP) chann ...... ine subendocardial arterioles.
@ast
Role of NO and K(+)(ATP) chann ...... ine subendocardial arterioles.
@en
P2093
P2860
P1476
Role of NO and K(+)(ATP) chann ...... ine subendocardial arterioles.
@en
P2093
P2860
P304
P356
10.1152/AJPHEART.1999.277.5.H1931
P407
P433
P577
1999-11-01T00:00:00Z