Abnormal Capillary Vasodynamics Contribute to Ictal Neurodegeneration in Epilepsy. - Wikidata - awgldk - TriplyDB

Abnormal Capillary Vasodynamics Contribute to Ictal Neurodegeneration in Epilepsy.

Abnormal Capillary Vasodynamics Contribute to Ictal Neurodegeneration in Epilepsy.

http://www.wikidata.org/entity/Q30839982

about

Capillary pericytes mediate coronary no-reflow after myocardial ischaemia.Seizure epicenter depth and translaminar field potential synchrony underlie complex variations in tissue oxygenation during ictal initiation.The past, present and future challenges in epilepsy related and sudden deaths and biobanking.Capillary pericytes express α-smooth muscle actin, which requires prevention of filamentous-actin depolymerization for detection.Aspirin attenuates spontaneous recurrent seizures in the chronically epileptic mice.Targeting pericytes for therapeutic approaches to neurological disorders Bioenergetic Mechanisms of Seizure Control Long-term all-optical interrogation of cortical neurons in awake-behaving nonhuman primates

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Abnormal Capillary Vasodynamics Contribute to Ictal Neurodegeneration in Epilepsy.

http://www.wikidata.org/entity/Q30839982

description

2017 nî lūn-bûn

@nan

2017 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2017 թվականի փետրվարին հրատարակված գիտական հոդված

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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name

Abnormal Capillary Vasodynamics Contribute to Ictal Neurodegeneration in Epilepsy.

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Abnormal Capillary Vasodynamics Contribute to Ictal Neurodegeneration in Epilepsy.

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Abnormal Capillary Vasodynamics Contribute to Ictal Neurodegeneration in Epilepsy.

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Abnormal Capillary Vasodynamics Contribute to Ictal Neurodegeneration in Epilepsy.

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Abnormal Capillary Vasodynamics Contribute to Ictal Neurodegeneration in Epilepsy.

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Abnormal Capillary Vasodynamics Contribute to Ictal Neurodegeneration in Epilepsy.

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Scientific Reports

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Abnormal Capillary Vasodynamics Contribute to Ictal Neurodegeneration in Epilepsy

@en

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Alexander A Schlegel

http://www.w3.org/2001/XMLSchema#string

David M Treiman

http://www.w3.org/2001/XMLSchema#string

Jonathan LaMee

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Jong M Rho

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Landon Inge

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Leandro L Di Stasi

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Luis Alarcon-Martinez

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Michael E Calhoun

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Pamela J Osborn Popp

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Rocio Leal-Campanario

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P2860

Fiji: an open-source platform for biological-image analysis.

Ischemic cell death in brain neurons

A novel mutation in the human voltage-gated potassium channel gene (Kv1.1) associates with episodic ataxia type 1 and sometimes with partial epilepsy

The powerhouse takes control of the cell: is the mitochondrial permeability transition a viable therapeutic target against neuronal dysfunction and death?

Limbic seizure and brain damage produced by kainic acid: mechanisms and relevance to human temporal lobe epilepsy

Bcl-2 transfection via herpes simplex virus blocks apoptosis-inducing factor translocation after focal ischemia in the rat

A novel mutation in the human voltage-gated potassium channel gene (Kv1.1) associates with episodic ataxia type 1 and sometimes with partial epilepsy

Unbiased stereological estimation of the total number of neurons in thesubdivisions of the rat hippocampus using the optical fractionator

Glial and neuronal control of brain blood flow

Pericytes in capillaries are contractile in vivo, but arterioles mediate functional hyperemia in the mouse brain.

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43276

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scholarly article

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10.1038/SREP43276

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7

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Jorge Otero-Millan

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2017-02-27T00:00:00Z

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1084131270

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28240297

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5327474

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