Hypothyroidism prolongs mitotic activity in the post-natal mouse brain.
about
Molecular basis of physiological heart growth: fundamental concepts and new playersThyroid hormone receptor alpha is a molecular switch of cardiac function between fetal and postnatal life.The Krüppel-like factor 9 cistrome in mouse hippocampal neurons reveals predominant transcriptional repression via proximal promoter bindingA bimodal influence of thyroid hormone on cerebellum oligodendrocyte differentiation.Thyroid hormone function in the rat testis.Unliganded thyroid hormone receptor α controls developmental timing in Xenopus tropicalisDepletion of the p43 mitochondrial T3 receptor increases Sertoli cell proliferation in mice.TR alpha 2 exerts dominant negative effects on hypothalamic Trh transcription in vivo.The timecourse of apoptotic cell death during postnatal remodeling of the mouse cochlea and its premature onset by triiodothyronine (T3)A Novel Population of Inner Cortical Cells in the Adrenal Gland That Displays Sexually Dimorphic Expression of Thyroid Hormone Receptor-β1.An evaluation of neurobehavioral tests used to assess the neurodevelopmental effects of early ammonium perchlorate exposure.Profound morphological and functional changes of rodent Purkinje cells between the first and the second postnatal weeks: a metamorphosis?A role of unliganded thyroid hormone receptor in postembryonic development in Xenopus laevis.Thyroid hormone triggers the developmental loss of axonal regenerative capacity via thyroid hormone receptor α1 and krüppel-like factor 9 in Purkinje cells.Thyroid hormone promotes neuronal differentiation of embryonic neural stem cells by inhibiting STAT3 signaling through TRα1A Mechanism to Enhance Cellular Responsivity to Hormone Action: Krüppel-Like Factor 9 Promotes Thyroid Hormone Receptor-β Autoinduction During Postembryonic Brain Development.Delayed development of specific thyroid hormone-regulated events in transthyretin null mice.Thyroid hormones and their nuclear receptors: new players in intestinal epithelium stem cell biology?Excess thyroid hormone inhibits embryonic neural stem/progenitor cells proliferation and maintenance through STAT3 signalling pathway.Liganded Thyroid Hormone Receptors Transactivate the DNA Methyltransferase 3a Gene in Mouse Neuronal CellsReceptors and signaling pathways involved in proliferation and differentiation of Sertoli cells.Thyroid hormone receptor alpha1 directly controls transcription of the beta-catenin gene in intestinal epithelial cells.Thyroid hormone participates in the regulation of neural stem cells and oligodendrocyte precursor cells in the central nervous system of adult rat.Thyroid Hormone Receptor α Controls Developmental Timing and Regulates the Rate and Coordination of Tissue-Specific Metamorphosis in Xenopus tropicalis.Deafness and loss of cochlear hair cells in the absence of thyroid hormone transporters Slc16a2 (Mct8) and Slc16a10 (Mct10).Regulation of T3 Availability in the Developing Brain: The Mouse Genetics Contribution.
P2860
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P2860
Hypothyroidism prolongs mitotic activity in the post-natal mouse brain.
description
2000 nî lūn-bûn
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2000年の論文
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name
Hypothyroidism prolongs mitotic activity in the post-natal mouse brain.
@ast
Hypothyroidism prolongs mitotic activity in the post-natal mouse brain.
@en
type
label
Hypothyroidism prolongs mitotic activity in the post-natal mouse brain.
@ast
Hypothyroidism prolongs mitotic activity in the post-natal mouse brain.
@en
prefLabel
Hypothyroidism prolongs mitotic activity in the post-natal mouse brain.
@ast
Hypothyroidism prolongs mitotic activity in the post-natal mouse brain.
@en
P2093
P1433
P1476
Hypothyroidism prolongs mitotic activity in the post-natal mouse brain.
@en
P2093
Demeneix B
Ghorbel MT
Hadj-Sahraoui N
P356
10.1016/S0304-3940(00)00768-0
P407
P577
2000-02-01T00:00:00Z