Blood-brain barrier disruption and matrix metalloproteinase-9 expression during reperfusion injury: mechanical versus embolic focal ischemia in spontaneously hypertensive rats.
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Vascular protection to increase the safety of tissue plasminogen activator for strokeEvidence-based review of oral traditional Chinese medicine compound recipe administration for treating weight drop-induced experimental traumatic brain injuryNADPH oxidase-mediated redox signal contributes to lipoteichoic acid-induced MMP-9 upregulation in brain astrocytesα2-Antiplasmin: New Insights and Opportunities for Ischemic Stroke.Acoustically enhanced Evans blue dye perfusion in neurological tissues.Measurement of blood-brain barrier permeability in acute ischemic stroke using standard first-pass perfusion CT dataAtorvastatin downregulates tissue plasminogen activator-aggravated genes mediating coagulation and vascular permeability in single cerebral endothelial cells captured by laser microdissection.Coadministration of FTY720 and rt-PA in an experimental model of large hemispheric stroke-no influence on functional outcome and blood-brain barrier disruptionNeuroprotective effects of overexpressing tissue inhibitor of metalloproteinase TIMP-1.Cortical spreading depression activates and upregulates MMP-9.Reversing the deleterious effects of α2-antiplasmin on tissue plasminogen activator therapy improves outcomes in experimental ischemic strokeAnnexin A2 combined with low-dose tPA improves thrombolytic therapy in a rat model of focal embolic stroke.Peroxynitrite decomposition catalyst prevents matrix metalloproteinase activation and neurovascular injury after prolonged cerebral ischemia in rats.Annexin A2: a tissue plasminogen activator amplifier for thrombolytic stroke therapy.Calmodulin kinase II-dependent transactivation of PDGF receptors mediates astrocytic MMP-9 expression and cell motility induced by lipoteichoic acidTransforming growth factor-β1 induces matrix metalloproteinase-9 and cell migration in astrocytes: roles of ROS-dependent ERK- and JNK-NF-κB pathways.The contribution of L-arginine to the neurotoxicity of recombinant tissue plasminogen activator following cerebral ischemia: a review of rtPA neurotoxicityHypoxia-ischemia or excitotoxin-induced tissue plasminogen activator- dependent gelatinase activation in mice neonate brain microvessels.Sex-independent neuroprotection with minocycline after experimental thromboembolic stroke.Japanese encephalitis virus induces matrix metalloproteinase-9 expression via a ROS/c-Src/PDGFR/PI3K/Akt/MAPKs-dependent AP-1 pathway in rat brain astrocytesMARCKS Signaling Differentially Regulates Vascular Smooth Muscle and Endothelial Cell Proliferation through a KIS-, p27kip1- Dependent Mechanism.Microglial cell activation is a source of metalloproteinase generation during hemorrhagic transformation.Endothelial cells and astrocytes: a concerto en duo in ischemic pathophysiology.Matrix Metalloproteinase Expression in Contusional Traumatic Brain Injury: A Paired Microdialysis Study.Anesthesia-Induced Hypothermia Attenuates Early-Phase Blood-Brain Barrier Disruption but Not Infarct Volume following Cerebral Ischemia.Evolution of blood-brain-barrier permeability after acute ischemic strokeSignaling, delivery and age as emerging issues in the benefit/risk ratio outcome of tPA For treatment of CNS ischemic disorders.Homocysteine alters cerebral microvascular integrity and causes remodeling by antagonizing GABA-A receptorDoes inhibiting Sur1 complement rt-PA in cerebral ischemia?HIV-1 gp120-induced injury to the blood-brain barrier: role of metalloproteinases 2 and 9 and relationship to oxidative stressExperimental models, neurovascular mechanisms and translational issues in stroke research.Targets of vascular protection in acute ischemic stroke differ in type 2 diabetes.Blood brain barrier breakdown as the starting point of cerebral small vessel disease? - New insights from a rat model.Mild hypothermia reduces tissue plasminogen activator-related hemorrhage and blood brain barrier disruption after experimental stroke.Tissue-type plasminogen activator induces opening of the blood-brain barrier via the LDL receptor-related protein.Disruption of the blood-brain barrier after generalized tonic-clonic seizures correlates with cerebrospinal fluid MMP-9 levels.Plasma and brain matrix metalloproteinase-9 after acute focal cerebral ischemia in rats.Malvidin's Effects on Rat Pial Microvascular Permeability Changes Due to Hypoperfusion and Reperfusion Injury.Neutrophils contribute to intracerebral haemorrhages after treatment with recombinant tissue plasminogen activator following cerebral ischaemiaAlteration of microvascular permeability in acute kidney injury
P2860
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P2860
Blood-brain barrier disruption and matrix metalloproteinase-9 expression during reperfusion injury: mechanical versus embolic focal ischemia in spontaneously hypertensive rats.
description
2002 nî lūn-bûn
@nan
2002 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Blood-brain barrier disruption ...... ontaneously hypertensive rats.
@ast
Blood-brain barrier disruption ...... ontaneously hypertensive rats.
@en
type
label
Blood-brain barrier disruption ...... ontaneously hypertensive rats.
@ast
Blood-brain barrier disruption ...... ontaneously hypertensive rats.
@en
prefLabel
Blood-brain barrier disruption ...... ontaneously hypertensive rats.
@ast
Blood-brain barrier disruption ...... ontaneously hypertensive rats.
@en
P2093
P1433
P1476
Blood-brain barrier disruption ...... ontaneously hypertensive rats.
@en
P2093
Tatsuro Mori
Toshiaki Aoki
Toshihisa Sumii
Xiaoying Wang
P304
P356
10.1161/01.STR.0000033932.34467.97
P407
P577
2002-11-01T00:00:00Z