Regenerative capacity and the number of satellite cells in soleus muscles of normal and mdx mice.
about
Analyses of the differentiation potential of satellite cells from myoD-/-, mdx, and PMP22 C22 mice.Muscular dystrophy begins early in embryonic development deriving from stem cell loss and disrupted skeletal muscle formationEnhancement of myogenic and muscle repair capacities of human adipose-derived stem cells with forced expression of MyoD.Identification of retinoic acid in a high content screen for agents that overcome the anti-myogenic effect of TGF-beta-1.Expression profiling reveals altered satellite cell numbers and glycolytic enzyme transcription in nemaline myopathy muscle.Angiotensin II type 1 receptor blockade attenuates TGF-beta-induced failure of muscle regeneration in multiple myopathic states.Challenges and opportunities in dystrophin-deficient cardiomyopathy gene therapy.Dystrophin expression in muscle stem cells regulates their polarity and asymmetric division.Increased sphingosine-1-phosphate improves muscle regeneration in acutely injured mdx mice.Mature adult dystrophic mouse muscle environment does not impede efficient engrafted satellite cell regeneration and self-renewal.Loss of niche-satellite cell interactions in syndecan-3 null mice alters muscle progenitor cell homeostasis improving muscle regeneration.Delayed bone regeneration is linked to chronic inflammation in murine muscular dystrophy.The deiodinases and the control of intracellular thyroid hormone signaling during cellular differentiation.Skeletal muscle activity and the fate of myonuclei.Satellite Cells and Skeletal Muscle Regeneration.Comparison of satellite cell-derived myoblasts and C2C12 differentiation in two- and three-dimensional cultures: changes in adhesion protein expression.Elastase levels and activity are increased in dystrophic muscle and impair myoblast cell survival, proliferation and differentiation.Testosterone-induced increase in muscle size in healthy young men is associated with muscle fiber hypertrophy.Testosterone-induced muscle hypertrophy is associated with an increase in satellite cell number in healthy, young men.Combined therapy with sonic hedgehog gene transfer and bone marrow-derived endothelial progenitor cells enhances angiogenesis and myogenesis in the ischemic skeletal muscle.Insulin-like growth factor-I analogue protects muscles of dystrophic mdx mice from contraction-mediated damage.Targeting muscle stem cell intrinsic defects to treat Duchenne muscular dystrophy.Synaptic Failure: Focus in an Integrative View of ALS.
P2860
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P2860
Regenerative capacity and the number of satellite cells in soleus muscles of normal and mdx mice.
description
2000 nî lūn-bûn
@nan
2000 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Regenerative capacity and the ...... uscles of normal and mdx mice.
@ast
Regenerative capacity and the ...... uscles of normal and mdx mice.
@en
type
label
Regenerative capacity and the ...... uscles of normal and mdx mice.
@ast
Regenerative capacity and the ...... uscles of normal and mdx mice.
@en
prefLabel
Regenerative capacity and the ...... uscles of normal and mdx mice.
@ast
Regenerative capacity and the ...... uscles of normal and mdx mice.
@en
P2093
P1476
Regenerative capacity and the ...... uscles of normal and mdx mice.
@en
P2093
P304
P356
10.1016/S0960-8966(99)00118-2
P577
2000-06-01T00:00:00Z