about
Ischemic stroke injury is mediated by aberrant Cdk5Ischemic long-term-potentiation (iLTP): perspectives to set the threshold of neural plasticity toward therapyThrombin regulation of synaptic transmission and plasticity: implications for health and diseaseEffect of cardiac arrest on cognitive impairment and hippocampal plasticity in middle-aged rats.PKMζ knockdown disrupts post-ischemic long-term potentiation via inhibiting postsynaptic expression of aminomethyl phosphonic acid receptorsActive calcium/calmodulin-dependent protein kinase II (CaMKII) regulates NMDA receptor mediated postischemic long-term potentiation (i-LTP) by promoting the interaction between CaMKII and NMDA receptors in ischemiaNoninvasive brain stimulation in traumatic brain injury.Neurobiology of rehabilitationCARDIOVASCULAR RISK FACTORS AFFECT HIPPOCAMPAL MICROVASCULATURE IN EARLY AD.Opposite roles of NMDA receptors in relapsing and primary progressive multiple sclerosisTissue-type plasminogen activator triggers the synaptic vesicle cycle in cerebral cortical neurons.Hydroxysafflor Yellow A Protects Neurons From Excitotoxic Death through Inhibition of NMDARs.Late-onset epileptogenesis and seizure genesis: lessons from models of cerebral ischemia.Neuroplasticity and cellular therapy in cerebral infarction.Changes in striatal signaling induce remodeling of RGS complexes containing Gbeta5 and R7BP subunits.Expression of Nampt in hippocampal and cortical excitatory neurons is critical for cognitive function.New synaptic and molecular targets for neuroprotection in Parkinson's disease.Early Hippocampal i-LTP and LOX-1 Overexpression Induced by Anoxia: A Potential Role in Neurodegeneration in NPC Mouse Model.The Plasminogen Activation System Promotes Dendritic Spine Recovery and Improvement in Neurological Function After an Ischemic Stroke.Angiogenesis, neurogenesis and neuroplasticity in ischemic stroke.Thrombin induces ischemic LTP (iLTP): implications for synaptic plasticity in the acute phase of ischemic stroke.Endothelin-1-induced mini-stroke in the dorsal hippocampus or lateral amygdala results in deficits in learning and memory.Combination BMSC and Niaspan treatment of stroke enhances white matter remodeling and synaptic protein expression in diabetic ratsDecreased striatal dopamine release underlies increased expression of long-term synaptic potentiation at corticostriatal synapses 24 h after 3-nitropropionic-acid-induced chemical hypoxia.Ischemic-LTP in striatal spiny neurons of both direct and indirect pathway requires the activation of D1-like receptors and NO/soluble guanylate cyclase/cGMP transmission.Glycine bidirectionally regulates ischemic tolerance via different mechanisms including NR2A-dependent CREB phosphorylation.Ischemic LTP: NMDA-dependency and dorso/ventral distribution within the hippocampus.When ischaemia is not ischaemia.Striatal GABA receptor alterations in hypoxic neonatal rats: role of glucose, oxygen and epinephrine treatment.Cardiac Arrest Induces Ischemic Long-Term Potentiation of Hippocampal CA1 Neurons That Occludes Physiological Long-Term Potentiation.Synaptic Plasticity Changes: Hallmark for Neurological and Psychiatric Disorders
P2860
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P2860
description
2003 nî lūn-bûn
@nan
2003 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Synaptic plasticity in the ischaemic brain.
@ast
Synaptic plasticity in the ischaemic brain.
@en
type
label
Synaptic plasticity in the ischaemic brain.
@ast
Synaptic plasticity in the ischaemic brain.
@en
prefLabel
Synaptic plasticity in the ischaemic brain.
@ast
Synaptic plasticity in the ischaemic brain.
@en
P2093
P1433
P1476
Synaptic plasticity in the ischaemic brain.
@en
P2093
Antonio Pisani
Giorgio Bernardi
LetiziaM Cupini
Paolo Calabresi
P304
P356
10.1016/S1474-4422(03)00532-5
P577
2003-10-01T00:00:00Z