comH, a novel gene essential for natural transformation of Helicobacter pylori.
about
Peptidoglycan crosslinking relaxation promotes Helicobacter pylori's helical shape and stomach colonization.Structural insights into the unique single-stranded DNA-binding mode of Helicobacter pylori DprACag3 is a novel essential component of the Helicobacter pylori Cag type IV secretion system outer membrane subcomplexHelicobacter pylori AddAB helicase-nuclease and RecA promote recombination-related DNA repair and survival during stomach colonizationThe Helicobacter pylori chemotaxis receptor TlpB (HP0103) is required for pH taxis and for colonization of the gastric mucosaPhylogeographic origin of Helicobacter pylori determines host-adaptive responses upon coculture with gastric epithelial cells.Global transposon mutagenesis and essential gene analysis of Helicobacter pylori.Deciphering the unusual acylation pattern of Helicobacter pylori lipid A.Recombination-based in vivo expression technology identifies Helicobacter pylori genes important for host colonizationColonization and inflammation deficiencies in Mongolian gerbils infected by Helicobacter pylori chemotaxis mutants.A Helicobacter pylori Homolog of Eukaryotic Flotillin Is Involved in Cholesterol Accumulation, Epithelial Cell Responses and Host ColonizationAnalysis of protein expression regulated by the Helicobacter pylori ArsRS two-component signal transduction system.Chemotaxis plays multiple roles during Helicobacter pylori animal infection.Evolution in an oncogenic bacterial species with extreme genome plasticity: Helicobacter pylori East Asian genomes.Molecular analyses of the natural transformation machinery and identification of pilus structures in the extremely thermophilic bacterium Thermus thermophilus strain HB27.Helicobacter pylori versus the host: remodeling of the bacterial outer membrane is required for survival in the gastric mucosa.In vitro and in vivo complementation of the Helicobacter pylori arginase mutant using an intergenic chromosomal site.Vitamin B6 is required for full motility and virulence in Helicobacter pylori.The lipid A 1-phosphatase of Helicobacter pylori is required for resistance to the antimicrobial peptide polymyxin.Change is good: variations in common biological mechanisms in the epsilonproteobacterial genera Campylobacter and HelicobacterThe stringent response is required for Helicobacter pylori survival of stationary phase, exposure to acid, and aerobic shock.Identification of a broad family of lipid A late acyltransferases with non-canonical substrate specificity.Characterization of a ComE3 homologue essential for DNA transformation in Helicobacter pylori.Chemorepulsion from the Quorum Signal Autoinducer-2 Promotes Helicobacter pylori Biofilm Dispersal.Natural transformation of an engineered Helicobacter pylori strain deficient in type II restriction endonucleases.Dual nuclease and helicase activities of Helicobacter pylori AddAB are required for DNA repair, recombination, and mouse infectivity.Evidence for the active role of a novel nuclease from Helicobacter pylori in the horizontal transfer of genetic information.Recent Advances in Helicobacter pylori Replication: Possible Implications in Adaptation to a Pathogenic Lifestyle and Perspectives for Drug Design.Characterization of two cryptic Helicobacter pylori plasmids: a putative source for horizontal gene transfer and gene shufflingTIFA Signaling in Gastric Epithelial Cells Initiates the cag Type 4 Secretion System-Dependent Innate Immune Response to Helicobacter pylori Infection.Increased Outer Membrane Vesicle Formation in a Helicobacter pylori tolB Mutant.The Helicobacter pylori cag pathogenicity island protein CagN is a bacterial membrane-associated protein that is processed at its C terminus.Detection of H.pylori DNA in gastric epithelial cells by in situ hybridization.Functional and topological characterization of novel components of the comB DNA transformation competence system in Helicobacter pyloriThe quorum-sensing molecule autoinducer 2 regulates motility and flagellar morphogenesis in Helicobacter pylori.Natural transformation competence in Helicobacter pylori is mediated by the basic components of a type IV secretion system.Interspecies transfer of antibiotic resistance between Helicobacter pylori and Helicobacter acinonychis.Natural transformation increases the rate of adaptation in the human pathogen Helicobacter pylori.Aldo-keto reductase from Helicobacter pylori--role in adaptation to growth at acid pH.Type IV secretion in Gram-negative and Gram-positive bacteria.
P2860
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P2860
comH, a novel gene essential for natural transformation of Helicobacter pylori.
description
2000 nî lūn-bûn
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2000 թուականի Յուլիսին հրատարակուած գիտական յօդուած
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2000年の論文
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2000年論文
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2000年論文
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2000年論文
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2000年論文
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2000年論文
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name
comH, a novel gene essential for natural transformation of Helicobacter pylori.
@ast
comH, a novel gene essential for natural transformation of Helicobacter pylori.
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type
label
comH, a novel gene essential for natural transformation of Helicobacter pylori.
@ast
comH, a novel gene essential for natural transformation of Helicobacter pylori.
@en
prefLabel
comH, a novel gene essential for natural transformation of Helicobacter pylori.
@ast
comH, a novel gene essential for natural transformation of Helicobacter pylori.
@en
P2093
P2860
P1476
comH, a novel gene essential for natural transformation of Helicobacter pylori.
@en
P2093
Bijlsma JJ
Boomkens SY
Kusters JG
Vandenbroucke-Grauls CM
P2860
P304
P356
10.1128/JB.182.14.3948-3954.2000
P407
P577
2000-07-01T00:00:00Z