DNA-AP sites generation by etoposide in whole blood cells.
about
DNA damaging agents and p53 do not cause senescence in quiescent cells, while consecutive re-activation of mTOR is associated with conversion to senescenceMouse embryonic stem cells undergo charontosis, a novel programmed cell death pathway dependent upon cathepsins, p53, and EndoG, in response to etoposide treatment.Role of oxidative stress in transformation induced by metal mixture.Evaluation of cytogenetic and DNA damage caused by thallium(I) acetate in human blood cells.
P2860
DNA-AP sites generation by etoposide in whole blood cells.
description
2009 nî lūn-bûn
@nan
2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
DNA-AP sites generation by etoposide in whole blood cells.
@ast
DNA-AP sites generation by etoposide in whole blood cells.
@en
type
label
DNA-AP sites generation by etoposide in whole blood cells.
@ast
DNA-AP sites generation by etoposide in whole blood cells.
@en
prefLabel
DNA-AP sites generation by etoposide in whole blood cells.
@ast
DNA-AP sites generation by etoposide in whole blood cells.
@en
P2860
P356
P1433
P1476
DNA-AP sites generation by etoposide in whole blood cells
@en
P2093
Efrain Tovar
Patricia Mussali
P2860
P2888
P356
10.1186/1471-2407-9-398
P407
P577
2009-11-16T00:00:00Z
P5875
P6179
1003945705