Telomere dynamics in human cells reprogrammed to pluripotency. - Wikidata - awgldk - TriplyDB

Telomere dynamics in human cells reprogrammed to pluripotency.

Telomere dynamics in human cells reprogrammed to pluripotency.

http://www.wikidata.org/entity/Q30941414

about

From cellular to chemical approach for acute neural and alternative options for age-induced functional diseases Programming and Reprogramming Cellular Age in the Era of Induced Pluripotency Investigating cellular identity and manipulating cell fate using induced pluripotent stem cells Identification of MMP1 as a novel risk factor for intracranial aneurysms in ADPKD using iPSC models.Mitochondrial rejuvenation after induced pluripotency Mitochondrial-associated cell death mechanisms are reset to an embryonic-like state in aged donor-derived iPS cells harboring chromosomal aberrations Residual expression of the reprogramming factors prevents differentiation of iPSC generated from human fibroblasts and cord blood CD34+ progenitors Constitutive expression of pluripotency-associated genes in mesodermal progenitor cells (MPCs).Epigenetic mechanisms regulate MHC and antigen processing molecules in human embryonic and induced pluripotent stem cells The LARGE principle of cellular reprogramming: lost, acquired and retained gene expression in foreskin and amniotic fluid-derived human iPS cells Induced pluripotent stem cell models of lysosomal storage disorders.Epigenetic and phenotypic profile of fibroblasts derived from induced pluripotent stem cells.Caudalized human iPSC-derived neural progenitor cells produce neurons and glia but fail to restore function in an early chronic spinal cord injury model.Endothelial cells derived from human iPSCS increase capillary density and improve perfusion in a mouse model of peripheral arterial disease.Rejuvenating senescent and centenarian human cells by reprogramming through the pluripotent state.iPSCs as a major opportunity to understand and cure age-related diseases.Reprogramming of telomeric regions during the generation of human induced pluripotent stem cells and subsequent differentiation into fibroblast-like derivatives.Therapeutic opportunities: telomere maintenance in inducible pluripotent stem cells.The aging signature: a hallmark of induced pluripotent stem cells?Induced pluripotent stem cells in dermatology: potentials, advances, and limitations Transient delivery of modified mRNA encoding TERT rapidly extends telomeres in human cells.Telomere reprogramming and maintenance in porcine iPS cells.Direct evaluation of myocardial viability and stem cell engraftment demonstrates salvage of the injured myocardium.Generation of leukemia inhibitory factor and basic fibroblast growth factor-dependent induced pluripotent stem cells from canine adult somatic cells Telomere dynamics in induced pluripotent stem cells: Potentials for human disease modeling.Molecular insights into the heterogeneity of telomere reprogramming in induced pluripotent stem cells.PDGFRβ expression and function in fibroblasts derived from pluripotent cells is linked to DNA demethylation.Advances in genome-wide DNA methylation analysis.Aging and reprogramming: a two-way street.Functional vascular smooth muscle cells derived from human induced pluripotent stem cells via mesenchymal stem cell intermediates Defining the diversity of phenotypic respecification using multiple cell lines and reprogramming regimens The role of telomeres and telomerase reverse transcriptase isoforms in pluripotency induction and maintenance Near-to-perfect homeostasis: examples of universal aging rule which germline evades.Generation of Induced Pluripotent Stem (iPS) Cells by Nuclear Reprogramming.Systematic review of induced pluripotent stem cell technology as a potential clinical therapy for spinal cord injury.Stem cells and neurodegenerative diseases: where is it all going?Is aging a barrier to reprogramming? Lessons from induced pluripotent stem cells.Telomere length maintenance, shortening, and lengthening.Quality control: Genome maintenance in pluripotent stem cells.Telomerase as a "stemness" enzyme.

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P2860

Telomere dynamics in human cells reprogrammed to pluripotency.

http://www.wikidata.org/entity/Q30941414

description

2009 nî lūn-bûn

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2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

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2009年論文

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2009年论文

@wuu

name

Telomere dynamics in human cells reprogrammed to pluripotency.

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Telomere dynamics in human cells reprogrammed to pluripotency.

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type

label

Telomere dynamics in human cells reprogrammed to pluripotency.

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Telomere dynamics in human cells reprogrammed to pluripotency.

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prefLabel

Telomere dynamics in human cells reprogrammed to pluripotency.

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Telomere dynamics in human cells reprogrammed to pluripotency.

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JOURNAL.PONE.0008124

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Telomere dynamics in human cells reprogrammed to pluripotency

@en

P2093

Eun Ah Chang

http://www.w3.org/2001/XMLSchema#string

Jose B Cibelli

http://www.w3.org/2001/XMLSchema#string

Kai Wang

http://www.w3.org/2001/XMLSchema#string

Pablo J Ross

http://www.w3.org/2001/XMLSchema#string

Shashanka Murthy

http://www.w3.org/2001/XMLSchema#string

Steven T Suhr

http://www.w3.org/2001/XMLSchema#string

Zeki Beyhan

http://www.w3.org/2001/XMLSchema#string

P2860

Induced pluripotent stem cell lines derived from human somatic cells

Induction of pluripotent stem cells from adult human fibroblasts by defined factors

Reprogramming of human somatic cells to pluripotency with defined factors

Telomeres acquire embryonic stem cell characteristics in induced pluripotent stem cells

Telomeres shorten during ageing of human fibroblasts

A highly conserved repetitive DNA sequence, (TTAGGG)n, present at the telomeres of human chromosomes

Extension of cell life-span and telomere length in animals cloned from senescent somatic cells.

Telomere maintenance in telomerase-deficient mouse embryonic stem cells: characterization of an amplified telomeric DNA

Hypervariable ultra-long telomeres in mice.

Telomeres and telomerase: their mechanisms of action and the effects of altering their functions.

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10.1371/JOURNAL.PONE.0008124

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12

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4

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Ramon M Rodriguez

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2009-12-02T00:00:00Z

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40443667

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19956585

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2780312

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