CD150 (SLAM) is a receptor for measles virus but is not involved in viral contact-mediated proliferation inhibition.
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Cellular localization of nectin-1 and glycoprotein D during herpes simplex virus infectionMeasles virus infects and suppresses proliferation of T lymphocytes from transgenic mice bearing human signaling lymphocytic activation moleculeMeasles virus infects both polarized epithelial and immune cells by using distinctive receptor-binding sites on its hemagglutininNonnucleoside inhibitor of measles virus RNA-dependent RNA polymerase complex activityUnity in diversity: shared mechanism of entry among paramyxovirusesIn situ tumor vaccination with adenovirus vectors encoding measles virus fusogenic membrane proteins and cytokines.DC-SIGN and CD150 have distinct roles in transmission of measles virus from dendritic cells to T-lymphocytes.Induction of membrane ceramides: a novel strategy to interfere with T lymphocyte cytoskeletal reorganisation in viral immunosuppression.Measles virus infection of alveolar macrophages and dendritic cells precedes spread to lymphatic organs in transgenic mice expressing human signaling lymphocytic activation molecule (SLAM, CD150)Measles virus selectively blind to signaling lymphocytic activation molecule (SLAM; CD150) is attenuated and induces strong adaptive immune responses in rhesus monkeys.Viral oncolysis - can insights from measles be transferred to canine distemper virus?Stringent requirement for the C protein of wild-type measles virus for growth both in vitro and in macaques.Canine distemper virus infection requires cholesterol in the viral envelopeTumor cell marker PVRL4 (nectin 4) is an epithelial cell receptor for measles virusMeasles virus-induced suppression of immune responses.Measles virus replication in lymphatic cells and organs of CD150 (SLAM) transgenic mice.The F gene of the Osaka-2 strain of measles virus derived from a case of subacute sclerosing panencephalitis is a major determinant of neurovirulence.Recombinant wild-type and edmonston strain measles viruses bearing heterologous H proteins: role of H protein in cell fusion and host cell specificityIdentification of a second major site for CD46 binding in the hemagglutinin protein from a laboratory strain of measles virus (MV): potential consequences for wild-type MV infection.Immature CD4+CD8+ thymocytes are preferentially infected by measles virus in human thymic organ culturesExtent of measles virus spread and immune suppression differentiates between wild-type and vaccine strains in the cotton rat model (Sigmodon hispidus).The morbillivirus receptor SLAM (CD150).Experimental adaptation of wild-type canine distemper virus (CDV) to the human entry receptor CD150.Neutral sphingomyelinase in physiological and measles virus induced T cell suppression.Given the opportunity, the Sendai virus RNA-dependent RNA polymerase could as well enter its template internally.Monkey CV1 cell line expressing the sheep-goat SLAM protein: a highly sensitive cell line for the isolation of peste des petits ruminants virus from pathological specimens.Efficient generation of vesicular stomatitis virus (VSV)-pseudotypes bearing morbilliviral glycoproteins and their use in quantifying virus neutralising antibodies.Measles virus-specific CD4 T-cell activity does not correlate with protection against lung infection or viral clearance.Mutations in the stalk of the measles virus hemagglutinin protein decrease fusion but do not interfere with virus-specific interaction with the homologous fusion protein.HIV-1 infection ex vivo accelerates measles virus infection by upregulating signaling lymphocytic activation molecule (SLAM) in CD4+ T cells.A human lung carcinoma cell line supports efficient measles virus growth and syncytium formation via a SLAM- and CD46-independent mechanism.Clathrin pit-mediated endocytosis of neutrophil elastase and cathepsin G by cancer cells.Immune regulation by SLAM family receptors and SAP-related adaptors.Efficient isolation of wild strains of canine distemper virus in Vero cells expressing canine SLAM (CD150) and their adaptability to marmoset B95a cells.Measles virus attachment proteins with impaired ability to bind CD46 interact more efficiently with the homologous fusion proteinFlexibility of the Head-Stalk Linker Domain of Paramyxovirus HN Glycoprotein Is Essential for Triggering Virus Fusion.Measles virus interacts with human SLAM receptor on dendritic cells to cause immunosuppression.Making it to the synapse: measles virus spread in and among neurons.Contribution of matrix, fusion, hemagglutinin, and large protein genes of the CAM-70 measles virus vaccine strain to efficient growth in chicken embryonic fibroblasts.Measles virus-induced immunosuppression: from effectors to mechanisms.
P2860
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P2860
CD150 (SLAM) is a receptor for measles virus but is not involved in viral contact-mediated proliferation inhibition.
description
2001 nî lūn-bûn
@nan
2001 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
CD150 (SLAM) is a receptor for ...... ated proliferation inhibition.
@ast
CD150 (SLAM) is a receptor for ...... ated proliferation inhibition.
@en
type
label
CD150 (SLAM) is a receptor for ...... ated proliferation inhibition.
@ast
CD150 (SLAM) is a receptor for ...... ated proliferation inhibition.
@en
prefLabel
CD150 (SLAM) is a receptor for ...... ated proliferation inhibition.
@ast
CD150 (SLAM) is a receptor for ...... ated proliferation inhibition.
@en
P2093
P2860
P1433
P1476
CD150 (SLAM) is a receptor for ...... ated proliferation inhibition.
@en
P2093
C Erlenhoefer
S Schneider-Schaulies
W J Wurzer
P2860
P304
P356
10.1128/JVI.75.10.4499-4505.2001
P577
2001-05-01T00:00:00Z