FAP-overexpressing fibroblasts produce an extracellular matrix that enhances invasive velocity and directionality of pancreatic cancer cells.
about
Breast cancer cells alter the dynamics of stromal fibronectin-collagen interactionsCapturing relevant extracellular matrices for investigating cell migrationExtracellular matrix macromolecules: potential tools and targets in cancer gene therapyThe wound healing, chronic fibrosis, and cancer progression triadTumor stroma as targets for cancer therapyThe application of the fibroblast activation protein α-targeted immunotherapy strategyHematopoietic stem cell-derived adipocytes and fibroblasts in the tumor microenvironmentFibroblast activation protein α in tumor microenvironment: recent progression and implications (review)Controlled breast cancer microarrays for the deconvolution of cellular multilayering and density effects upon drug responsesFighting the force: Potential of homeobox genes for tumor microenvironment regulationRegulation of mesenchymal-to-epithelial transition by PARAXIS during somitogenesisA bioengineered heterotypic stroma-cancer microenvironment model to study pancreatic ductal adenocarcinoma.Miniaturized pre-clinical cancer models as research and diagnostic tools.Downregulation of FAP suppresses cell proliferation and metastasis through PTEN/PI3K/AKT and Ras-ERK signaling in oral squamous cell carcinoma.Stromal reengineering to treat pancreas cancerChemotherapy and tumor microenvironment of pancreatic cancer.Three-dimensional in vitro tumor models for cancer research and drug evaluation.Identification of inhibitory scFv antibodies targeting fibroblast activation protein utilizing phage display functional screens.Fibroblast activation protein (FAP) is essential for the migration of bone marrow mesenchymal stem cells through RhoA activationFibroblast activation protein overexpression and clinical implications in solid tumors: a meta-analysis.Characterization of dedifferentiating human mature adipocytes from the visceral and subcutaneous fat compartments: fibroblast-activation protein alpha and dipeptidyl peptidase 4 as major components of matrix remodeling.Fibroblast activation protein-α, a stromal cell surface protease, shapes key features of cancer associated fibroblasts through proteome and degradome alterationsFibroblast activation protein-α-expressing fibroblasts promote the progression of pancreatic ductal adenocarcinoma.Fibroblast-Derived Extracellular Matrices: An Alternative Cell Culture System That Increases Metastatic Cellular PropertiesA subset of myofibroblastic cancer-associated fibroblasts regulate collagen fiber elongation, which is prognostic in multiple cancers.Syndecan-1-Induced ECM Fiber Alignment Requires Integrin αvβ3 and Syndecan-1 Ectodomain and Heparan Sulfate ChainsCleavage of Type I Collagen by Fibroblast Activation Protein-α Enhances Class A Scavenger Receptor Mediated Macrophage Adhesion.Multiphysics and Multiscale Analysis for Chemotherapeutic Drug.Activatable near-infrared fluorescent probe for in vivo imaging of fibroblast activation protein-alpha.The mesenchymal tumor microenvironment: a drug-resistant niche.Revealing cytokine-induced changes in the extracellular matrix with secondary ion mass spectrometryA potent immunotoxin targeting fibroblast activation protein for treatment of breast cancer in miceOptical imaging of tumor microenvironmentModeling tumor microenvironments using custom-designed biomaterial scaffoldsTumor-secreted LOXL2 activates fibroblasts through FAK signaling.Podoplanin expression in cancer-associated fibroblasts enhances tumor progression of invasive ductal carcinoma of the pancreas.Matrix-regulated integrin αvβ5 maintains α5β1-dependent desmoplastic traits prognostic of neoplastic recurrence.Pancreatic cancer stroma: understanding biology leads to new therapeutic strategies.Fibroblast activation protein: A potential therapeutic target in cancer.Cancer-associated fibroblasts drive the progression of metastasis through both paracrine and mechanical pressure on cancer tissue.
P2860
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P2860
FAP-overexpressing fibroblasts produce an extracellular matrix that enhances invasive velocity and directionality of pancreatic cancer cells.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
FAP-overexpressing fibroblasts ...... ty of pancreatic cancer cells.
@ast
FAP-overexpressing fibroblasts ...... ty of pancreatic cancer cells.
@en
type
label
FAP-overexpressing fibroblasts ...... ty of pancreatic cancer cells.
@ast
FAP-overexpressing fibroblasts ...... ty of pancreatic cancer cells.
@en
prefLabel
FAP-overexpressing fibroblasts ...... ty of pancreatic cancer cells.
@ast
FAP-overexpressing fibroblasts ...... ty of pancreatic cancer cells.
@en
P2093
P2860
P356
P1433
P1476
FAP-overexpressing fibroblasts ...... ty of pancreatic cancer cells.
@en
P2093
Edna Cukierman
Hyung-Ok Lee
Janusz Franco-Barraza
Jonathan D Cheng
Matthildi Valianou
Stefanie R Mullins
P2860
P2888
P356
10.1186/1471-2407-11-245
P407
P577
2011-06-13T00:00:00Z
P5875
P6179
1028244391