Rat carotid neointimal smooth muscle cells reexpress a developmentally regulated mRNA phenotype during repair of arterial injury.
about
Atrophin proteins interact with the Fat1 cadherin and regulate migration and orientation in vascular smooth muscle cellsExpression of the AT2 receptor developmentally programs extracellular signal-regulated kinase activity and influences fetal vascular growthExpression of apolipoprotein serum amyloid A mRNA in human atherosclerotic lesions and cultured vascular cells: implications for serum amyloid A functionType VIII collagen stimulates smooth muscle cell migration and matrix metalloproteinase synthesis after arterial injuryMacrophages express osteopontin during repair of myocardial necrosisGenetic causation of neointimal hyperplasia in hemodialysis vascular access dysfunctionUnique aspects of the developing lung circulation: structural development and regulation of vasomotor toneRestricted expression of homeobox genes distinguishes fetal from adult human smooth muscle cellsHepatocyte nuclear factor-3 homologue 1 (HFH-1) represses transcription of smooth muscle-specific genesPeroxide-inducible Ets-1 mediates platelet-derived growth factor receptor-alpha gene transcription in vascular smooth muscle cellsSp1 phosphorylation regulates apoptosis via extracellular FasL-Fas engagement.Targeted expression of SV40 large T-antigen to visceral smooth muscle induces proliferation of contractile smooth muscle cells and results in megacolonDeletion of yes-associated protein (YAP) specifically in cardiac and vascular smooth muscle cells reveals a crucial role for YAP in mouse cardiovascular development.Modulation of myocardin function by the ubiquitin E3 ligase UBR5.Osteopontin is elevated during neointima formation in rat arteries and is a novel component of human atherosclerotic plaques.H19, a developmentally regulated gene, is reexpressed in rat vascular smooth muscle cells after injury.Long-term biological response of injured rat carotid artery seeded with smooth muscle cells expressing retrovirally introduced human genes.Angiotensin II induces delayed mitogenesis and cellular proliferation in rat aortic smooth muscle cells. Correlation with the expression of specific endogenous growth factors and reversal by suramin.Extinction of autonomous growth potential in embryonic: adult vascular smooth muscle cell heterokaryons.Genetic engineering of vein grafts resistant to atherosclerosisCC chemokine receptor-1 activates intimal smooth muscle-like cells in graft arterial disease.Ontogeny of the opioid growth factor, [Met5]-enkephalin, preproenkephalin gene expression, and the zeta opioid receptor in the developing and adult aorta of rat.Perlecan regulates Oct-1 gene expression in vascular smooth muscle cells.Vascular smooth muscle diversity: insights from developmental biology.Neointima formation after acute vascular injury. Role of counteradhesive extracellular matrix proteinsRegulation of smooth muscle cell growth by endothelium-derived factors.In stent restenosis: bane of the stent era.A comparison of stent-induced stenosis in coronary and peripheral arteriesPostnatal regulation of fibroblast growth factor ligand and receptor gene expression in rat thoracic aortaBasic fibroblast growth factor has a differential effect on MyoD conversion of cultured aortic smooth muscle cells from newborn and adult rats.Smooth muscle cell expression of extracellular matrix genes after arterial injuryIntimal smooth muscle cells are a source but not a sensor of anti-inflammatory CYP450 derived oxylipins.Molecular cloning of a diverged homeobox gene that is rapidly down-regulated during the G0/G1 transition in vascular smooth muscle cells.Cell biology of human vascular smooth muscleH19, a marker of developmental transition, is reexpressed in human atherosclerotic plaques and is regulated by the insulin family of growth factors in cultured rabbit smooth muscle cells.The mitogen-activated protein kinase pathway can mediate growth inhibition and proliferation in smooth muscle cells. Dependence on the availability of downstream targetsKrüppel-like factor 4 (KLF4/GKLF) is a target of bone morphogenetic proteins and transforming growth factor beta 1 in the regulation of vascular smooth muscle cell phenotype.Serum induction of MEF2/RSRF expression in vascular myocytes is mediated at the level of translation.HOXB7 overexpression promotes differentiation of C3H10T1/2 cells to smooth muscle cells.A comparative molecular analysis of four rat smooth muscle cell lines.
P2860
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P2860
Rat carotid neointimal smooth muscle cells reexpress a developmentally regulated mRNA phenotype during repair of arterial injury.
description
1992 nî lūn-bûn
@nan
1992 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
name
Rat carotid neointimal smooth ...... ing repair of arterial injury.
@ast
Rat carotid neointimal smooth ...... ing repair of arterial injury.
@en
type
label
Rat carotid neointimal smooth ...... ing repair of arterial injury.
@ast
Rat carotid neointimal smooth ...... ing repair of arterial injury.
@en
prefLabel
Rat carotid neointimal smooth ...... ing repair of arterial injury.
@ast
Rat carotid neointimal smooth ...... ing repair of arterial injury.
@en
P2093
P356
P1433
P1476
Rat carotid neointimal smooth ...... ing repair of arterial injury.
@en
P2093
Giachelli CM
Majesky MW
Schwartz SM
P304
P356
10.1161/01.RES.71.4.759
P577
1992-10-01T00:00:00Z