The role of c-Jun and c-Fos expression in androgen-independent prostate cancer.
about
Prostate-associated gene 4 (PAGE4), an intrinsically disordered cancer/testis antigen, is a novel therapeutic target for prostate cancerTranscriptional activity of c-Jun is critical for the suppression of AR function.Cancer biomarker discovery: the entropic hallmark.The Stress-response protein prostate-associated gene 4, interacts with c-Jun and potentiates its transactivation.The microRNA-23b/27b/24-1 cluster is a disease progression marker and tumor suppressor in prostate cancer.Cop1 constitutively regulates c-Jun protein stability and functions as a tumor suppressor in mice.Interaction between c-jun and androgen receptor determines the outcome of taxane therapy in castration resistant prostate cancer.Loss of JUNB/AP-1 promotes invasive prostate cancerThe androgen receptor and signal-transduction pathways in hormone-refractory prostate cancer. Part 2: Androgen-receptor cofactors and bypass pathways.Phosphorylation-induced Conformational Ensemble Switching in an Intrinsically Disordered Cancer/Testis Antigen.Spotlight on the role of COP1 in tumorigenesis.The role of transcription factors in prostate cancer and potential for future RNA interference therapy.Transcriptional activation of PRMT5 by NF-Y is required for cell growth and negatively regulated by the PKC/c-Fos signaling in prostate cancer cells.Cancer/testis antigen PAGE4, a regulator of c-Jun transactivation, is phosphorylated by homeodomain-interacting protein kinase 1, a component of the stress-response pathway.Gossypol inhibits growth, invasiveness, and angiogenesis in human prostate cancer cells by modulating NF-κB/AP-1 dependent- and independent-signaling.Upregulation of MAPK pathway is associated with survival in castrate-resistant prostate cancer.JunB and PTEN in prostate cancer: 'loss is nothing else than change'.Correlations of the expressions of c-Jun and Egr-1 proteins with clinicopathological features and prognosis of patients with nasopharyngeal carcinoma.Tumor microenvironment promotes prostate cancer cell dissemination via the Akt/mTOR pathway.Observer variation in immunohistochemical analysis of protein expression, time for a change?
P2860
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P2860
The role of c-Jun and c-Fos expression in androgen-independent prostate cancer.
description
2004 nî lūn-bûn
@nan
2004 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
The role of c-Jun and c-Fos expression in androgen-independent prostate cancer.
@ast
The role of c-Jun and c-Fos expression in androgen-independent prostate cancer.
@en
type
label
The role of c-Jun and c-Fos expression in androgen-independent prostate cancer.
@ast
The role of c-Jun and c-Fos expression in androgen-independent prostate cancer.
@en
prefLabel
The role of c-Jun and c-Fos expression in androgen-independent prostate cancer.
@ast
The role of c-Jun and c-Fos expression in androgen-independent prostate cancer.
@en
P2093
P356
P1476
The role of c-Jun and c-Fos expression in androgen-independent prostate cancer
@en
P2093
John M S Bartlett
N Sarath Krishna
Rono Mukherjee
P304
P356
10.1002/PATH.1605
P577
2004-10-01T00:00:00Z