Molecular markers in ductal carcinoma in situ of the breast. - Wikidata - awgldk - TriplyDB

Molecular markers in ductal carcinoma in situ of the breast.

Molecular markers in ductal carcinoma in situ of the breast.

http://www.wikidata.org/entity/Q31135997

about

S100A7 and the progression of breast cancer Expression of AMAP1, an ArfGAP, provides novel targets to inhibit breast cancer invasive activities Evidence that molecular changes in cells occur before morphological alterations during the progression of breast ductal carcinoma The Spemann organizer gene, Goosecoid, promotes tumor metastasis A neural survival factor is a candidate oncogene in breast cancer Genetic alteration and gene expression modulation during cancer progression S100A7 (psoriasin) expression is associated with aggressive features and alteration of Jab1 in ductal carcinoma in situ of the breast Transcriptomic changes in human breast cancer progression as determined by serial analysis of gene expression Changes in gene expression during the development of mammary tumors in MMTV-Wnt-1 transgenic mice.Identifying differential expression in multiple SAGE libraries: an overdispersed log-linear model approach Next-generation sequencing: a powerful tool for the discovery of molecular markers in breast ductal carcinoma in situ Ductal carcinoma in situ: terminology, classification, and natural history Statistical analysis and significance testing of serial analysis of gene expression data using a Poisson mixture model From mice to humans: identification of commonly deregulated genes in mammary cancer via comparative SAGE studies.Gene expression profiles of epithelial cells microscopically isolated from a breast-invasive ductal carcinoma and a nodal metastasis An integrated analysis of genes and pathways exhibiting metabolic differences between estrogen receptor positive breast cancer cells.S100A7-downregulation inhibits epidermal growth factor-induced signaling in breast cancer cells and blocks osteoclast formation.Identification and characterization of CCAAT/Enhancer Binding proteindelta (C/EBPdelta) target genes in G0 growth arrested mammary epithelial cells Regulation of transgenes in three-dimensional cultures of primary mouse mammary cells demonstrates oncogene dependence and identifies cells that survive deinduction.PI3K pathway activation in high-grade ductal carcinoma in situ--implications for progression to invasive breast carcinoma.Myc interacts with Max and Miz1 to repress C/EBPdelta promoter activity and gene expression.Frequent aberrant DNA methylation of ABCB1, FOXC1, PPP2R2B and PTEN in ductal carcinoma in situ and early invasive breast cancer.C/EBP{delta} targets cyclin D1 for proteasome-mediated degradation via induction of CDC27/APC3 expression CCAAT/enhancer binding protein delta (C/EBPdelta, CEBPD)-mediated nuclear import of FANCD2 by IPO4 augments cellular response to DNA damage.Heterogeneity Between Ducts of the Same Nuclear Grade Involved by Duct Carcinoma In Situ (DCIS) of the Breast CCAAT/Enhancer Binding Protein-delta (C/EBP-delta) regulates cell growth, migration and differentiation RNA-Seq of human breast ductal carcinoma in situ models reveals aldehyde dehydrogenase isoform 5A1 as a novel potential target.The expression of Psoriasin (S100A7) and CD24 is linked and related to the differentiation of mammary epithelial cells.Metastasis: new perspectives on an old problem The molecular pathology of breast cancer progression.Telomere shortening occurs in subsets of normal breast epithelium as well as in situ and invasive carcinoma.The role of nicotinamide adenine dinucleotide phosphate oxidase-derived reactive oxygen species in the acquisition of metastatic ability of tumor cells Expression of CD74 in high grade gliomas: a potential role in temozolomide resistance.Classification of ductal carcinoma in situ by gene expression profiling.Transcriptional changes associated with breast cancer occur as normal human mammary epithelial cells overcome senescence barriers and become immortalized.Identification of a Src tyrosine kinase/SIAH2 E3 ubiquitin ligase pathway that regulates C/EBPδ expression and contributes to transformation of breast tumor cells.Regulation of DCIS to invasive breast cancer progression by Singleminded-2s (SIM2s).Precancerous Lesions of the Breast Progression of Ductal Carcinoma in Situ from the Pathological Perspective.Intraductal proliferative lesions of the breast-terminology and biology matter: premalignant lesions or preinvasive cancer?

P2860

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P2860

Molecular markers in ductal carcinoma in situ of the breast.

http://www.wikidata.org/entity/Q31135997

description

2003 nî lūn-bûn

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2003 թուականի Մարտին հրատարակուած գիտական յօդուած

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2003 թվականի մարտին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

@zh-hk

2003年論文

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2003年論文

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2003年论文

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name

Molecular markers in ductal carcinoma in situ of the breast.

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Molecular markers in ductal carcinoma in situ of the breast.

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type

label

Molecular markers in ductal carcinoma in situ of the breast.

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Molecular markers in ductal carcinoma in situ of the breast.

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prefLabel

Molecular markers in ductal carcinoma in situ of the breast.

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Molecular markers in ductal carcinoma in situ of the breast.

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Molecular Cancer Research

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Molecular markers in ductal carcinoma in situ of the breast

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Amiel Cooper

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Andrea Richardson

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